Defining a new paradigm for human arrhythmia syndromes: phenotypic manifestations of gene mutations in ion channel- and transporter-associated proteins.
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The role of late I Na in development of cardiac arrhythmiasCortactin is required for N-cadherin regulation of Kv1.5 channel functionSimulation of the undiseased human cardiac ventricular action potential: model formulation and experimental validationPopeye domain containing proteins are essential for stress-mediated modulation of cardiac pacemaking in miceCardiac Mechano-Gated Ion Channels and Arrhythmias.Caveolin-1 Sensitivity of Excitatory Amino Acid Transporters EAAT1, EAAT2, EAAT3, and EAAT4.Loss of type 9 adenylyl cyclase triggers reduced phosphorylation of Hsp20 and diastolic dysfunctionEffects of proarrhythmic drugs on relaxation time and beating pattern in rat engineered heart tissue.Dysfunction in the βII spectrin-dependent cytoskeleton underlies human arrhythmia.Cardiac myocytes and local signaling in nano-domains.Different subcellular populations of L-type Ca2+ channels exhibit unique regulation and functional roles in cardiomyocytesThe genetic basis for inherited forms of sinoatrial dysfunction and atrioventricular node dysfunctionNovel rare variants in congenital cardiac arrhythmia genes are frequent in drug-induced torsades de pointes.Increased late sodium current contributes to long QT-related arrhythmia susceptibility in female mice.Gene mutations in cardiac arrhythmias: a review of recent evidence in ion channelopathies.Systems biology and cardiac arrhythmiasGenetics can contribute to the prognosis of Brugada syndrome: a pilot model for risk stratification.Genetics of sudden death: focus on inherited channelopathies.Implementing genomics and pharmacogenomics in the clinic: The National Human Genome Research Institute's genomic medicine portfolio.A Novel Mechanism for Human Cardiac Ankyrin-B Syndrome due to Reciprocal Chromosomal Translocation.Nav channel complex heterogeneity: new targets for the treatment of arrhythmia?Refining repolarization reserve.Cytoskeleton regulation of ion channels.Defining the disconnect between in vitro models and human arrhythmogenic disease: context matters.Ankyrin-B structurally defines terminal microdomains of peripheral somatosensory axons.The role of βII spectrin in cardiac health and disease.Sex and regional differences in rabbit right ventricular L-type calcium current levels and mathematical modelling of arrhythmia vulnerability.Risk factors for symptoms in long QT syndrome patients in a single pediatric center.
P2860
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P2860
Defining a new paradigm for human arrhythmia syndromes: phenotypic manifestations of gene mutations in ion channel- and transporter-associated proteins.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Defining a new paradigm for hu ...... ansporter-associated proteins.
@ast
Defining a new paradigm for hu ...... ansporter-associated proteins.
@en
Defining a new paradigm for hu ...... ansporter-associated proteins.
@nl
type
label
Defining a new paradigm for hu ...... ansporter-associated proteins.
@ast
Defining a new paradigm for hu ...... ansporter-associated proteins.
@en
Defining a new paradigm for hu ...... ansporter-associated proteins.
@nl
prefLabel
Defining a new paradigm for hu ...... ansporter-associated proteins.
@ast
Defining a new paradigm for hu ...... ansporter-associated proteins.
@en
Defining a new paradigm for hu ...... ansporter-associated proteins.
@nl
P2860
P1433
P1476
Defining a new paradigm for hu ...... ansporter-associated proteins.
@en
P2093
Michael J Ackerman
Peter J Mohler
P2860
P304
P356
10.1161/CIRCRESAHA.110.224592
P577
2010-08-01T00:00:00Z