Cell death and plasticity after experimental spinal cord injury.
about
Axonal degeneration as a self-destructive defense mechanism against neurotropic virus infectionAquaporins in spinal cord injury: the janus face of aquaporin 4Neurotrophic factors, cellular bridges and gene therapy for spinal cord injury.Transplanted oligodendrocytes and motoneuron progenitors generated from human embryonic stem cells promote locomotor recovery after spinal cord transection.Post-traumatic inflammation following spinal cord injury.Sprouting, regeneration and circuit formation in the injured spinal cord: factors and activity.Intermittent noxious stimulation following spinal cord contusion injury impairs locomotor recovery and reduces spinal brain-derived neurotrophic factor-tropomyosin-receptor kinase signaling in adult rats.Aspiration of a cervical spinal contusion injury in preparation for delayed peripheral nerve grafting does not impair forelimb behavior or axon regenerationEffects of valproic acid, a histone deacetylase inhibitor, on improvement of locomotor function in rat spinal cord injury based on epigenetic science.Endogenous neural progenitor cells as therapeutic target after spinal cord injury.Microglial activation in rat experimental spinal cord injury model.Stem Cells and Labeling for Spinal Cord Injury.Concise review: human pluripotent stem cells in the treatment of spinal cord injury.Detrimental effects of antiapoptotic treatments in spinal cord injury.Calpain inhibitor inhibits p35-p25-Cdk5 activation, decreases tau hyperphosphorylation, and improves neurological function after spinal cord hemisection in rats.Spontaneous and augmented growth of axons in the primate spinal cord: effects of local injury and nerve growth factor-secreting cell grafts.Neuroprotective effect of the proanthocyanidin-rich fraction in experimental model of spinal cord injury.Soluble epoxide hydrolase inhibition provides multi-target therapeutic effects in rats after spinal cord injury.Stem Cells Therapy for Spinal Cord Injury.TWEAK-Fn14 Influences Neurogenesis Status via Modulating NF-κB in Mice with Spinal Cord Injury.Effect of Apocynin, an inhibitor of NADPH oxidase, in the inflammatory process induced by an experimental model of spinal cord injury.The protocadherin alpha cluster is required for axon extension and myelination in the developing central nervous system.
P2860
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P2860
Cell death and plasticity after experimental spinal cord injury.
description
2000 nî lūn-bûn
@nan
2000 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2000年の論文
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2000年学术文章
@wuu
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
name
Cell death and plasticity after experimental spinal cord injury.
@ast
Cell death and plasticity after experimental spinal cord injury.
@en
Cell death and plasticity after experimental spinal cord injury.
@nl
type
label
Cell death and plasticity after experimental spinal cord injury.
@ast
Cell death and plasticity after experimental spinal cord injury.
@en
Cell death and plasticity after experimental spinal cord injury.
@nl
prefLabel
Cell death and plasticity after experimental spinal cord injury.
@ast
Cell death and plasticity after experimental spinal cord injury.
@en
Cell death and plasticity after experimental spinal cord injury.
@nl
P2093
P1476
Cell death and plasticity after experimental spinal cord injury.
@en
P2093
P356
10.1016/S0079-6123(00)28003-5
P577
2000-01-01T00:00:00Z