Cancer stem cells from epithelial ovarian cancer patients privilege oxidative phosphorylation, and resist glucose deprivation. - Wikidata - awgldk - TriplyDB

Cancer stem cells from epithelial ovarian cancer patients privilege oxidative phosphorylation, and resist glucose deprivation.

Cancer stem cells from epithelial ovarian cancer patients privilege oxidative phosphorylation, and resist glucose deprivation.

http://www.wikidata.org/entity/Q34100946

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Cancer stem cell metabolism Antioxidant Mechanisms and ROS-Related MicroRNAs in Cancer Stem Cells Oxidative stress, epigenetics, and cancer stem cells in arsenic carcinogenesis and prevention Migration and invasion of drug-resistant lung adenocarcinoma cells are dependent on mitochondrial activity Glucose deprivation elicits phenotypic plasticity via ZEB1-mediated expression of NNMT.Mitochondria as therapeutic targets for cancer stem cells.Cancer Stem Cells in Small Cell Lung Cancer Cell Line H446: Higher Dependency on Oxidative Phosphorylation and Mitochondrial Substrate-Level Phosphorylation than Non-Stem Cancer Cells Mitochondrial-Targeted Decyl-Triphenylphosphonium Enhances 2-Deoxy-D-Glucose Mediated Oxidative Stress and Clonogenic Killing of Multiple Myeloma Cells High mitochondrial mass identifies a sub-population of stem-like cancer cells that are chemo-resistant.Oxidative metabolism drives inflammation-induced platinum resistance in human ovarian cancer.A hypoxic signature marks tumors formed by disseminated tumor cells in the BALB-neuT mammary cancer model.TRAP1 downregulation in human ovarian cancer enhances invasion and epithelial-mesenchymal transition.Induction of metastasis, cancer stem cell phenotype, and oncogenic metabolism in cancer cells by ionizing radiation.Resistance to glucose starvation as metabolic trait of platinum-resistant human epithelial ovarian cancer cells.TRIM28 multi-domain protein regulates cancer stem cell population in breast tumor development.Heterogeneity in Cancer Metabolism: New Concepts in an Old Field.Identification of a cancer stem cell-specific function for the histone deacetylases, HDAC1 and HDAC7, in breast and ovarian cancer.The metabolic landscape of cancer stem cells.Autophagy inhibition reduces chemoresistance and tumorigenic potential of human ovarian cancer stem cells.Loss of fructose-1,6-bisphosphatase induces glycolysis and promotes apoptosis resistance of cancer stem-like cells: an important role in hexavalent chromium-induced carcinogenesis.Hallmarks of cancer stem cell metabolism.Mitochondrial biogenesis is required for the anchorage-independent survival and propagation of stem-like cancer cells Mitochondrial dynamics as regulators of cancer biology.Tumor-associated myeloid cells as guiding forces of cancer cell stemness.Bioenergetic Adaptations in Chemoresistant Ovarian Cancer Cells.K-core decomposition of a protein domain co-occurrence network reveals lower cancer mutation rates for interior cores Licochalcone A specifically induces cell death in glioma stem cells via mitochondrial dysfunction.Therapeutic potential of the metabolic modulator phenformin in targeting the stem cell compartment in melanoma.Peroxiredoxin 3 maintains the survival of endometrial cancer stem cells by regulating oxidative stress.Therapeutic potential of the metabolic modulator Metformin on osteosarcoma cancer stem-like cells.PGC1α: Friend or Foe in Cancer?Metabolic regulation of glioma stem-like cells in the tumor micro-environment.Detachment from the primary site and suspension in ascites as the initial step in metabolic reprogramming and metastasis to the omentum in ovarian cancer.Combination of a thioxodihydroquinazolinone with cisplatin eliminates ovarian cancer stem cell-like cells (CSC-LCs) and shows preclinical potential.Transcriptional Regulation of Stem Cell and Cancer Stem Cell Metabolism.Resveratrol inhibits ovarian tumor growth in an in vivo mouse model.CD73 Regulates Stemness and Epithelial-Mesenchymal Transition in Ovarian Cancer-Initiating Cells.Regulation of Tumor Progression by Programmed Necrosis.Elucidating the Metabolic Plasticity of Cancer: Mitochondrial Reprogramming and Hybrid Metabolic States.Cancer stem cells (CSCs): metabolic strategies for their identification and eradication.

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P2860

Cancer stem cells from epithelial ovarian cancer patients privilege oxidative phosphorylation, and resist glucose deprivation.

http://www.wikidata.org/entity/Q34100946

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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ONCOTARGET.2010

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Cancer stem cells from epithel ...... nd resist glucose deprivation.

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Alberto Amadori

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Andrea Rasola

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Anna Pastò

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Chiara Bellio

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Chiara Frasson

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Giorgia Nardo

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Giorgia Pilotto

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Giulia Guzzo

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Maria Ornella Nicoletto

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Mariangela Manicone

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P2860

Identification and characterization of ovarian cancer-initiating cells from primary human tumors

Aldehyde dehydrogenase 1 is a marker for normal and malignant human colonic stem cells (SC) and tracks SC overpopulation during colon tumorigenesis

Cancer metabolism: key players in metabolic reprogramming

Pyruvate kinase M2 is a phosphotyrosine-binding protein

Declining NAD(+) induces a pseudohypoxic state disrupting nuclear-mitochondrial communication during aging

Molecular phenotyping of human ovarian cancer stem cells unravels the mechanisms for repair and chemoresistance

The M2 splice isoform of pyruvate kinase is important for cancer metabolism and tumour growth

Otto Warburg's contributions to current concepts of cancer metabolism

Metabolic reprogramming: a cancer hallmark even warburg did not anticipate

Brain tumor initiating cells adapt to restricted nutrition through preferential glucose uptake.

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10.18632/ONCOTARGET.2010

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12

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Stefano Indraccolo

Vincenzo Ciminale

Elisabetta Zulato

Micol Silic-Benussi

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2014-06-01T00:00:00Z

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