Mechanisms establishing TLR4-responsive activation states of inflammatory response genes. - Wikidata - awgldk - TriplyDB

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

http://www.wikidata.org/entity/Q34102791

about

Epigenetic Control of Macrophage Polarisation and Soluble Mediator Gene Expression during Inflammation Molecular control of activation and priming in macrophages Preparing the first responders: building the inflammatory transcriptome from the ground up Anti-inflammatory therapy in chronic disease: challenges and opportunities Towards an understanding of cell-specific functions of signal-dependent transcription factors 2009 pandemic H1N1 influenza virus elicits similar clinical course but differential host transcriptional response in mouse, macaque, and swine infection models Genomics pipelines and data integration: challenges and opportunities in the research setting.Innate immune activity conditions the effect of regulatory variants upon monocyte gene expression NCoR repression of LXRs restricts macrophage biosynthesis of insulin-sensitizing omega 3 fatty acids.Glucocorticoid receptor coordinates transcription factor-dominated regulatory network in macrophages.H3S28 phosphorylation is a hallmark of the transcriptional response to cellular stress.Control of VEGF-A transcriptional programs by pausing and genomic compartmentalization Distinct mechanisms for induction and tolerance regulate the immediate early genes encoding interleukin 1β and tumor necrosis factor α.BTB-ZF transcriptional regulator PLZF modifies chromatin to restrain inflammatory signaling programs.A dual cis-regulatory code links IRF8 to constitutive and inducible gene expression in macrophages.Dual RNA sequencing reveals the expression of unique transcriptomic signatures in lipopolysaccharide-induced BV-2 microglial cells.Myosin VI regulates gene pairing and transcriptional pause release in T cells.Functional Impact of RNA editing and ADARs on regulation of gene expression: perspectives from deep sequencing studies.Bidirectional Transcription Arises from Two Distinct Hubs of Transcription Factor Binding and Active Chromatin Epigenetic Modulation of Microglial Inflammatory Gene Loci in Helminth-Induced Immune Suppression: Implications for Immune Regulation in Neurocysticercosis Transcript dynamics of proinflammatory genes revealed by sequence analysis of subcellular RNA fractions Role of transcriptional coregulator GRIP1 in the anti-inflammatory actions of glucocorticoids.Nonlinear dependencies of biochemical reactions for context-specific signaling dynamics.Splicing kinetics and transcript release from the chromatin compartment limit the rate of Lipid A-induced gene expression Modulation of immune function by polyphenols: possible contribution of epigenetic factors Tissue damage drives co-localization of NF-κB, Smad3, and Nrf2 to direct Rev-erb sensitive wound repair in mouse macrophages A systems analysis identifies a feedforward inflammatory circuit leading to lethal influenza infection Glucocorticoid receptor represses proinflammatory genes at distinct steps of the transcription cycle Recent progress using systems biology approaches to better understand molecular mechanisms of immunity.Hormone-regulated transcriptomes: lessons learned from estrogen signaling pathways in breast cancer cells.Time Series miRNA-mRNA integrated analysis reveals critical miRNAs and targets in macrophage polarization Switching of the relative dominance between feedback mechanisms in lipopolysaccharide-induced NF-κB signaling Regulation of microglia activation and deactivation by nuclear receptors.Epigenetic regulation of macrophage polarization and function.Deciphering cis-regulatory control in inflammatory cells Epigenomic control of the innate immune response.The role of chromatin dynamics in immune cell development.Ready, pause, go: regulation of RNA polymerase II pausing and release by cellular signaling pathways Signaling via the NFκB system.S100-alarmin-induced innate immune programming protects newborn infants from sepsis.

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Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

http://www.wikidata.org/entity/Q34102791

description

2011 nî lūn-bûn

@nan

2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

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Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

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Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

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type

label

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

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Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

@en

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

@nl

prefLabel

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

@ast

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

@en

Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.

@nl

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JOURNAL.PGEN.1002401

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Mechanisms establishing TLR4-responsive activation states of inflammatory response genes

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Alexander Hoffmann

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Andrea Crotti

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Christine S Cheng

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Colleen Ludka

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Donna Reichart

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Jean Lozach

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Josh Stender

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Laure Escoubet-Lozach

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Michael G Rosenfeld

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Nathan J Spann

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A large fraction of extragenic RNA pol II transcription sites overlap enhancers

Translating the Histone Code

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Selective anchoring of TFIID to nucleosomes by trimethylation of histone H3 lysine 4

Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities

Genome-wide maps of chromatin state in pluripotent and lineage-committed cells

Genome-wide analysis in vivo of translation with nucleotide resolution using ribosome profiling

Parallel SUMOylation-dependent pathways mediate gene- and signal-specific transrepression by LXRs and PPARgamma

Innate immune recognition

The transcriptional coactivators p300 and CBP are histone acetyltransferases

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e1002401

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scholarly article

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10.1371/JOURNAL.PGEN.1002401

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12

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7

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Minna U Kaikkonen

Ivan Garcia-Bassets

Serena Ghisletti

Christopher K Glass

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2011-12-08T00:00:00Z

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51884654

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22174696

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Toll-like receptor

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3234212

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