Sodium channels and their genes: dynamic expression in the normal nervous system, dysregulation in disease states(1).
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New insights into neuron-glia communicationFibroblast growth factor homologous factor 1B binds to the C terminus of the tetrodotoxin-resistant sodium channel rNav1.9a (NaN)Ion channels and schizophrenia: a gene set-based analytic approach to GWAS data for biological hypothesis testing.Gating-pore currents demonstrate selective and specific modulation of individual sodium channel voltage-sensors by biological toxins.Association between multiple sclerosis and epilepsy: large population-based record-linkage studiesChanges in physiological properties of rat ganglion cells during retinal degenerationNerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice.The therapeutic potential of Na+ and Ca2+ channel blockers in pain management.Breakthrough pain in malignant and non-malignant diseases: a review of prevalence, characteristics and mechanisms.Nociceptor Sensitization Depends on Age and Pain Chronicity(1,2,3).Orofacial pain in cancer: part I--mechanisms.Peripheral drive in Aα/β-fiber neurons is altered in a rat model of osteoarthritis: changes in following frequency and recovery from inactivationAlzheimer's secretases regulate voltage-gated sodium channels.THE ROLE OF INTRACELLULAR SODIUM (Na) IN THE REGULATION OF CALCIUM (Ca)-MEDIATED SIGNALING AND TOXICITY.The axon initial segment in nervous system disease and injuryComparison of the Effects of Zonisamide, Ethosuximide and Pregabalin in the Chronic Constriction Injury Induced Neuropathic Pain in Rats.Effect of systemic and intracortical administration of phenytoin in two genetic models of absence epilepsy.Role of hippocampal sodium channel Nav1.6 in kindling epileptogenesis.Emergence of sustained spontaneous hyperactivity and temporary preservation of OFF responses in ganglion cells of the retinal degeneration (rd1) mouse.Optimized expression and purification of NavAb provide the structural insight into the voltage dependence.Dexmedetomidine and clonidine inhibit the function of Na(v)1.7 independent of α(2)-adrenoceptor in adrenal chromaffin cells.Hyperpolarized shifts in the voltage dependence of fast inactivation of Nav1.4 and Nav1.5 in a rat model of critical illness myopathy.Sodium influx during action potential in innervated and denervated rat skeletal muscles.Endothelin-1 Decreases Excitability of the Dorsal Root Ganglion Neurons via ETB Receptor.
P2860
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P2860
Sodium channels and their genes: dynamic expression in the normal nervous system, dysregulation in disease states(1).
description
2000 nî lūn-bûn
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2000 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի դեկտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
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2000年论文
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name
Sodium channels and their gene ...... gulation in disease states(1).
@ast
Sodium channels and their gene ...... gulation in disease states(1).
@en
Sodium channels and their gene ...... gulation in disease states(1).
@nl
type
label
Sodium channels and their gene ...... gulation in disease states(1).
@ast
Sodium channels and their gene ...... gulation in disease states(1).
@en
Sodium channels and their gene ...... gulation in disease states(1).
@nl
prefLabel
Sodium channels and their gene ...... gulation in disease states(1).
@ast
Sodium channels and their gene ...... gulation in disease states(1).
@en
Sodium channels and their gene ...... gulation in disease states(1).
@nl
P2093
P1433
P1476
Sodium channels and their gene ...... gulation in disease states(1).
@en
P2093
P356
10.1016/S0006-8993(00)02774-8
P407
P577
2000-12-01T00:00:00Z