M protein and hyaluronic acid capsule are essential for in vivo selection of covRS mutations characteristic of invasive serotype M1T1 group A Streptococcus.
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Interaction of Bacterial Exotoxins with Neutrophil Extracellular Traps: Impact for the Infected HostRole for streptococcal collagen-like protein 1 in M1T1 group A Streptococcus resistance to neutrophil extracellular trapsEvolutionary Constraints Shaping Streptococcus pyogenes-Host Interactions.Tracing the evolutionary history of the pandemic group A streptococcal M1T1 clone.ETosis: A Microbicidal Mechanism beyond Cell Death.LL-37 opsonizes and inhibits biofilm formation of Aggregatibacter actinomycetemcomitans at subbactericidal concentrations.Streptococcal collagen-like protein A and general stress protein 24 are immunomodulating virulence factors of group A Streptococcus.Novel role of the antimicrobial peptide LL-37 in the protection of neutrophil extracellular traps against degradation by bacterial nucleases.Disease manifestations and pathogenic mechanisms of Group A Streptococcus.A bacterial pathogen co-opts host plasmin to resist killing by cathelicidin antimicrobial peptidesMutual exclusivity of hyaluronan and hyaluronidase in invasive group A Streptococcus.Determining antibody-binding site of streptococcal pyrogenic exotoxin B to protect mice from group a streptococcus infectionUnique genomic arrangements in an invasive serotype M23 strain of Streptococcus pyogenes identify genes that induce hypervirulence.Transcription of the Streptococcus pyogenes hyaluronic acid capsule biosynthesis operon is regulated by previously unknown upstream elementsDifferential virulence gene expression of group A Streptococcus serotype M3 in response to co-culture with Moraxella catarrhalis.Characterization of the effect of the histidine kinase CovS on response regulator phosphorylation in group A Streptococcus.Antibiotic modulation of capsular exopolysaccharide and virulence in Acinetobacter baumanniiMutations in the control of virulence sensor gene from Streptococcus pyogenes after infection in mice lead to clonal bacterial variants with altered gene regulatory activity and virulence.Serotype M3 and M28 Group A Streptococci Have Distinct Capacities to Evade Neutrophil and TNF-α Responses and to Invade Soft Tissues.Zinc disrupts central carbon metabolism and capsule biosynthesis in Streptococcus pyogenes.A group A Streptococcus ADP-ribosyltransferase toxin stimulates a protective interleukin 1β-dependent macrophage immune responseA Neutralizing Monoclonal IgG1 Antibody of Platelet-Activating Factor Acetylhydrolase SsE Protects Mice against Lethal Subcutaneous Group A Streptococcus InfectionCationic antimicrobial peptide resistance mechanisms of streptococcal pathogens.CovRS-Regulated Transcriptome Analysis of a Hypervirulent M23 Strain of Group A Streptococcus pyogenes Provides New Insights into Virulence Determinants.Conquering Neutrophils.Contemporary Pharyngeal and Invasive emm1 and Invasive emm12 Group A Streptococcus Isolates Exhibit Similar In Vivo Selection for CovRS Mutants in Mice.The Mga Regulon but Not Deoxyribonuclease Sda1 of Invasive M1T1 Group A Streptococcus Contributes to In Vivo Selection of CovRS Mutations and Resistance to Innate Immune Killing Mechanisms.Neutrophil extracellular traps: double-edged swords of innate immunity.Immunization with a streptococcal multiple-epitope recombinant protein protects mice against invasive group A streptococcal infectionA conserved UDP-glucose dehydrogenase encoded outside the hasABC operon contributes to capsule biogenesis in group A StreptococcusStudy of streptococcal hemoprotein receptor (Shr) in iron acquisition and virulence of M1T1 group A streptococcus.Host and pathogen hyaluronan signal through human siglec-9 to suppress neutrophil activation.A natural inactivating mutation in the CovS component of the CovRS regulatory operon in a pattern D Streptococcal pyogenes strain influences virulence-associated genesBacterial Evasion of Host Antimicrobial Peptide DefensesAcquisition of the Sda1-encoding bacteriophage does not enhance virulence of the serotype M1 Streptococcus pyogenes strain SF370Bacterial strategies of resistance to antimicrobial peptides.IgG protease Mac/IdeS is not essential for phagocyte resistance or mouse virulence of M1T1 group A StreptococcusRole of hypoxia inducible factor-1 in keratinocyte inflammatory response and neutrophil recruitment.Complement-mediated opsonization of invasive group A Streptococcus pyogenes strain AP53 is regulated by the bacterial two-component cluster of virulence responder/sensor (CovRS) system.A novel C5a-derived immunobiotic peptide reduces Streptococcus agalactiae colonization through targeted bacterial killing.
P2860
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P2860
M protein and hyaluronic acid capsule are essential for in vivo selection of covRS mutations characteristic of invasive serotype M1T1 group A Streptococcus.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@ast
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@en
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@nl
type
label
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@ast
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@en
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@nl
prefLabel
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@ast
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@en
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@nl
P2093
P2860
P50
P356
P1433
P1476
M protein and hyaluronic acid ...... pe M1T1 group A Streptococcus.
@en
P2093
Andrew Hollands
Jason N Cole
Maren von Köckritz-Blickwede
P2860
P356
10.1128/MBIO.00191-10
P577
2010-08-31T00:00:00Z