Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo. - Wikidata - awgldk - TriplyDB

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

http://www.wikidata.org/entity/Q34117698

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Microglial Priming and Alzheimer's Disease: A Possible Role for (Early) Immune Challenges and Epigenetics?The Pro-inflammatory Effects of Glucocorticoids in the Brain Neuroimmune mechanisms of stress: sex differences, developmental plasticity, and implications for pharmacotherapy of stress-related disease Aging-related changes in neuroimmune-endocrine function: implications for hippocampal-dependent cognition Impact of age-related neuroglial cell responses on hippocampal deterioration The yin and yang of microglia Neuroinflammation in the normal aging hippocampus Estrogens regulate neuroinflammatory genes via estrogen receptors α and β in the frontal cortex of middle-aged female rats A multispecies approach for understanding neuroimmune mechanisms of stress.Neuroinflammatory challenges compromise neuronal function in the aging brain: Postoperative cognitive delirium and Alzheimer's disease.TGFβ produced by IL-10 redirected astrocytes attenuates microglial activation Differentially Severe Cognitive Effects of Compromised Cerebral Blood Flow in Aged Mice: Association with Myelin Degradation and Microglia Activation.Memory impairments in healthy aging: Role of aging-induced microglial sensitization.The role of hepatic and splenic macrophages in E. coli-induced memory impairments in aged rats Greater glucocorticoid receptor activation in hippocampus of aged rats sensitizes microglia.Higher mortality and impaired elimination of bacteria in aged mice after intracerebral infection with E. coli are associated with an age-related decline of microglia and macrophage functions.Little exercise, big effects: reversing aging and infection-induced memory deficits, and underlying processes Microglia inflammatory responses are controlled by an intrinsic circadian clock.A lifespan approach to neuroinflammatory and cognitive disorders: a critical role for glia.Age related changes in microglial phenotype vary between CNS regions: grey versus white matter differences.Prior exposure to repeated morphine potentiates mechanical allodynia induced by peripheral inflammation and neuropathy.Intracisternal interleukin-1 receptor antagonist prevents postoperative cognitive decline and neuroinflammatory response in aged rats.Sulforaphane induces Nrf2 target genes and attenuates inflammatory gene expression in microglia from brain of young adult and aged mice Influence of hypothalamic IL-6/gp130 receptor signaling on the HPA axis response to chronic stress.Aging leads to prolonged duration of inflammation-induced depression-like behavior caused by Bacillus Calmette-Guérin.The Alarmin HMGB1 Mediates Age-Induced Neuroinflammatory Priming Dysregulated neuronal-microglial cross-talk during aging, stress and inflammation.Nanoparticles and colloids as contributing factors in neurodegenerative disease.Feeding the beast: can microglia in the senescent brain be regulated by diet?MicroRNAs: Roles in Regulating Neuroinflammation.High-fat diet and aging interact to produce neuroinflammation and impair hippocampal- and amygdalar-dependent memory.Microglia Priming with Aging and Stress.How Does Exercise Reduce the Rate of Age-Associated Cognitive Decline? A Review of Potential Mechanisms.Microglia activation: one of the checkpoints in the CNS inflammation caused by Angiostrongylus cantonensis infection in rodent model.Dietary Luteolin Reduces Proinflammatory Microglia in the Brain of Senescent Mice Glial and neuroinflammatory targets for treating substance use disorders.Sex differences in the phagocytic and migratory activity of microglia and their impairment by palmitic acid.Diminished circadian rhythms in hippocampal microglia may contribute to age-related neuroinflammatory sensitization.Depiction of microglial activation in aging and dementia: Positron emission tomography with [11C]DPA713 versus [11C]( R)PK11195.Stereological Analysis of Microglia in Aged Male and Female Fischer 344 Rats in Socially Relevant Brain Regions.

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P2860

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

http://www.wikidata.org/entity/Q34117698

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@ast

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@en

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@nl

type

label

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@ast

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@en

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@nl

prefLabel

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@ast

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@en

Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

@nl

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J.JNEUROIM.2010.05.022

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Journal of Neuroimmunology

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Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.

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Matthew G Frank

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Steven F Maier

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P2860

Gapped BLAST and PSI-BLAST: a new generation of protein database search programs

Exploring the full spectrum of macrophage activation

mRNA up-regulation of MHC II and pivotal pro-inflammatory genes in normal brain aging

Microglia: a sensor for pathological events in the CNS

Neuroinflammation and disruption in working memory in aged mice after acute stimulation of the peripheral innate immune system.

Aging sensitizes mice to behavioral deficits induced by central HIV-1 gp120

Minocycline attenuates lipopolysaccharide (LPS)-induced neuroinflammation, sickness behavior, and anhedonia.

Aging, microglial cell priming, and the discordant central inflammatory response to signals from the peripheral immune system

Time course of hippocampal IL-1 beta and memory consolidation impairments in aging rats following peripheral infection

Exaggerated sickness behavior and brain proinflammatory cytokine expression in aged mice in response to intracerebroventricular lipopolysaccharide.

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10.1016/J.JNEUROIM.2010.05.022

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1-2

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226

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Ruth M. Barrientos

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2010-05-26T00:00:00Z

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