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Neuroinflammation in the normal aging hippocampusIL-1RA blocks E. coli-induced suppression of Arc and long-term memory in aged F344xBN F1 ratsSynaptic correlates of increased cognitive vulnerability with aging: peripheral immune challenge and aging interact to disrupt theta-burst late-phase long-term potentiation in hippocampal area CA1Memory for context is impaired by injecting anisomycin into dorsal hippocampus following context exploration.Aging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.The role of hepatic and splenic macrophages in E. coli-induced memory impairments in aged ratsHigh-fat diet consumption disrupts memory and primes elevations in hippocampal IL-1β, an effect that can be prevented with dietary reversal or IL-1 receptor antagonism.Aging and a peripheral immune challenge interact to reduce mature brain-derived neurotrophic factor and activation of TrkB, PLCgamma1, and ERK in hippocampal synaptoneurosomes.Greater glucocorticoid receptor activation in hippocampus of aged rats sensitizes microglia.Little exercise, big effects: reversing aging and infection-induced memory deficits, and underlying processesMicroglia inflammatory responses are controlled by an intrinsic circadian clock.Aging and infection reduce expression of specific brain-derived neurotrophic factor mRNAs in hippocampus.Intracisternal interleukin-1 receptor antagonist prevents postoperative cognitive decline and neuroinflammatory response in aged rats.Prostaglandins are necessary and sufficient to induce contextual fear learning impairments after interleukin-1 beta injections into the dorsal hippocampus.Time course of hippocampal IL-1 beta and memory consolidation impairments in aging rats following peripheral infectionThe Alarmin HMGB1 Mediates Age-Induced Neuroinflammatory PrimingGlucocorticoids Mediate Short-Term High-Fat Diet Induction of Neuroinflammatory Priming, the NLRP3 Inflammasome, and the Danger Signal HMGB1Characterization of the sickness response in young and aging rats following E. coli infection.High-fat diet and aging interact to produce neuroinflammation and impair hippocampal- and amygdalar-dependent memory.Prior laparotomy or corticosterone potentiates lipopolysaccharide-induced fever and sickness behaviors.Stable, long-term, spatial memory in young and aged rats achieved with a one day Morris water maze training protocol.Divergent effects of brain interleukin-1ß in mediating fever, lethargy, anorexia and conditioned fear memory.Neonatal bacterial infection alters fever to live and simulated infections in adulthood.Reductions in frontocortical cytokine levels are associated with long-lasting alterations in reward valuation after methamphetamine.IL-1RA injected intra-cisterna magna confers extended prophylaxis against lipopolysaccharide-induced neuroinflammatory and sickness responses.Memory for context is impaired by a post context exposure injection of interleukin-1 beta into dorsal hippocampus.Spinal gap junctions: potential involvement in pain facilitation.The role of the dorsal hippocampus in the acquisition and retrieval of context memory representations.Brain-derived neurotrophic factor mRNA downregulation produced by social isolation is blocked by intrahippocampal interleukin-1 receptor antagonist.Snake venom phospholipase A2s (Asp49 and Lys49) induce mechanical allodynia upon peri-sciatic administration: involvement of spinal cord glia, proinflammatory cytokines and nitric oxide.Diminished circadian rhythms in hippocampal microglia may contribute to age-related neuroinflammatory sensitization.IL-1 receptor type I gene expression in the amygdala of inflammatory susceptible Lewis and inflammatory resistant Fischer rats.Peripheral infection and aging interact to impair hippocampal memory consolidation.Early-life infection leads to altered BDNF and IL-1beta mRNA expression in rat hippocampus following learning in adulthood.BDNF mRNA expression in rat hippocampus following contextual learning is blocked by intrahippocampal IL-1beta administration.Aging and an Immune Challenge Interact to Produce Prolonged, but Not Permanent, Reductions in Hippocampal L-LTP and mBDNF in a Rodent Model with Features of Delirium.Food for thought: how nutrition impacts cognition and emotionThe role of microglia in neurogenesis: exercise and aging as cofactorsVoluntary exercise as an anti-neuroinflammatory therapeuticIdentification of a novel inflammation-protective locus in the Fischer rat
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P50
description
hulumtuese
@sq
researcher
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wetenschapper
@nl
հետազոտող
@hy
name
Ruth M. Barrientos
@ast
Ruth M. Barrientos
@en
Ruth M. Barrientos
@es
Ruth M. Barrientos
@nl
Ruth M. Barrientos
@sl
type
label
Ruth M. Barrientos
@ast
Ruth M. Barrientos
@en
Ruth M. Barrientos
@es
Ruth M. Barrientos
@nl
Ruth M. Barrientos
@sl
prefLabel
Ruth M. Barrientos
@ast
Ruth M. Barrientos
@en
Ruth M. Barrientos
@es
Ruth M. Barrientos
@nl
Ruth M. Barrientos
@sl
P1053
E-3244-2013
P106
P1153
6601938964
P21
P31
P3829
P496
0000-0001-7224-4109