Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
about
Alternative RNA splicing: contribution to pain and potential therapeutic strategyA splice variant of the two-pore domain potassium channel TREK-1 with only one pore domain reduces the surface expression of full-length TREK-1 channelsPhosphatidylinositol 3-kinase facilitates microtubule-dependent membrane transport for neuronal growth cone guidanceModulation of K2P 2.1 and K2P 10.1 K(+) channel sensitivity to carvedilol by alternative mRNA translation initiation.Molecular regulations governing TREK and TRAAK channel functions.Optical control of endogenous proteins with a photoswitchable conditional subunit reveals a role for TREK1 in GABA(B) signaling.Alternatively Spliced Isoforms of KV10.1 Potassium Channels Modulate Channel Properties and Can Activate Cyclin-dependent Kinase in Xenopus Oocytes.Variants of stretch-activated two-pore potassium channel TREK-1 associated with preterm labor in humans.Alternatively Spliced Human TREK-1 Variants Alter TREK-1 Channel Function and Localization.Mixing and matching TREK/TRAAK subunits generate heterodimeric K2P channels with unique properties.Aristolochic acid, a plant extract used in the treatment of pain and linked to Balkan endemic nephropathy, is a regulator of K2P channels.TREK-1 currents in smooth muscle cells from pregnant human myometrium.A role for two-pore potassium (K2P) channels in endometrial epithelial function.Two-pore domain potassium channels: potential therapeutic targets for the treatment of pain.The family of K2P channels: salient structural and functional properties.The role of protein-protein interactions in the intracellular traffic of the potassium channels TASK-1 and TASK-3.Much more than a leak: structure and function of K₂p-channels.Identification and characterization of alternative splice variants of the mouse Trek2/Kcnk10 gene.VLCAD enzyme activity determinations in newborns identified by screening: a valuable tool for risk assessment.Alternative splicing determines mRNA translation initiation and function of human K(2P)10.1 K+ channels.Identification of the muscarinic pathway underlying cessation of sleep-related burst activity in rat thalamocortical relay neurons.GI-530159, a novel, selective, mechanosensitive two-pore-domain potassium (K2P ) channel opener, reduces rat dorsal root ganglion neuron excitability.Novel nuclear hENT2 isoforms regulate cell cycle progression via controlling nucleoside transport and nuclear reservoir.The Thermosensitive Potassium Channel TREK-1 Contributes to Coolness-Evoked Responses of Grueneberg Ganglion Neurons
P2860
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P2860
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@ast
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@en
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@nl
type
label
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@ast
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@en
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@nl
prefLabel
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@ast
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@en
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain.
@nl
P2860
P50
P356
P1476
Dominant negative effects of a non-conducting TREK1 splice variant expressed in brain
@en
P2093
Kathryn A Rees
P2860
P304
29295-29304
P356
10.1074/JBC.M110.108423
P407
P577
2010-07-06T00:00:00Z