The stimulation of glycolysis by hypoxia in activated monocytes is mediated by AMP-activated protein kinase and inducible 6-phosphofructo-2-kinase.
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6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase: head-to-head with a bifunctional enzyme that controls glycolysis14-3-3s regulate fructose-2,6-bisphosphate levels by binding to PKB-phosphorylated cardiac fructose-2,6-bisphosphate kinase/phosphataseHypothalamic AMPK as a Regulator of Energy HomeostasisRegulation and function of AMPK in physiology and diseasesAMP-activated protein kinase: a target for drugs both ancient and modernAMP-activated protein kinase: an energy sensor that regulates all aspects of cell functionMechanisms of regulation of PFKFB expression in pancreatic and gastric cancer cellsMolecular basis of the fructose-2,6-bisphosphatase reaction of PFKFB3: Transition state and the C-terminal functionRegulation of ubiquitous 6-phosphofructo-2-kinase by the ubiquitin-proteasome proteolytic pathway during myogenic C2C12 cell differentiationProgestins activate 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase 3 (PFKFB3) in breast cancer cellsCrystal structure of the hypoxia-inducible form of 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase (PFKFB3): a possible new target for cancer therapy6-Phosphofructo-2-kinase (pfkfb3) gene promoter contains hypoxia-inducible factor-1 binding sites necessary for transactivation in response to hypoxiaTargeting 6-phosphofructo-2-kinase (PFKFB3) as a therapeutic strategy against cancerAMP-activated kinase AMPK is expressed in boar spermatozoa and regulates motilityHypoxia induces expression and activation of AMPK in rat dental pulp cellsActivation of AMPK alpha- and gamma-isoform complexes in the intact ischemic rat heartAMPK: a nutrient and energy sensor that maintains energy homeostasisTGF-β activation by bone marrow-derived thrombospondin-1 causes Schistosoma- and hypoxia-induced pulmonary hypertension.AMPK as a metabolic tumor suppressor: control of metabolism and cell growth.Activation of AMP-activated protein kinase alpha1 alleviates endothelial cell apoptosis by increasing the expression of anti-apoptotic proteins Bcl-2 and survivin.Acute modulation of sugar transport in brain capillary endothelial cell cultures during activation of the metabolic stress pathway.NDRG2 overexpression enhances glucose deprivation-mediated apoptosis in breast cancer cells via inhibition of the LKB1-AMPK pathway.The vestigial enzyme D-dopachrome tautomerase protects the heart against ischemic injury.Mitochondrial fragmentation leads to intracellular acidification in Caenorhabditis elegans and mammalian cells.Anticancer targets in the glycolytic metabolism of tumors: a comprehensive reviewThioredoxin-interacting protein (Txnip) gene expression: sensing oxidative phosphorylation status and glycolytic rate.High-fat and obesogenic diets: current and future strategies to fight obesity and diabetes.PFKFB3 activation in cancer cells by the p38/MK2 pathway in response to stress stimuli.Hypoxia induces transcription of 6-phosphofructo-2-kinase/fructose-2,6-biphosphatase-4 gene via hypoxia-inducible factor-1alpha activation.Cyclin D1 Restrains Oncogene-Induced Autophagy by Regulating the AMPK-LKB1 Signaling Axis.LKB1 and AMPK and the cancer-metabolism link - ten years afterActivation of AMP-Activated Protein Kinase Attenuates Tumor Necrosis Factor-α-Induced Lipolysis via Protection of Perilipin in 3T3-L1 Adipocytes.5'-AMP-activated protein kinase (AMPK) is induced by low-oxygen and glucose deprivation conditions found in solid-tumor microenvironments.Roles of 5'-AMP-activated protein kinase (AMPK) in mammalian glucose homoeostasisSmall molecule adenosine 5'-monophosphate activated protein kinase (AMPK) modulators and human diseasesA novel model of common Toll-like receptor 4- and injury-induced transcriptional themes in human leukocytes.Carbohydrate metabolism is perturbed in peroxisome-deficient hepatocytes due to mitochondrial dysfunction, AMP-activated protein kinase (AMPK) activation, and peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) suppression.Carbon source metabolism and its regulation in cancer cells.Regulation of NAD(P)H oxidases by AMPK in cardiovascular systemsAMPK Protects Leukemia-Initiating Cells in Myeloid Leukemias from Metabolic Stress in the Bone Marrow
P2860
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P2860
The stimulation of glycolysis by hypoxia in activated monocytes is mediated by AMP-activated protein kinase and inducible 6-phosphofructo-2-kinase.
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@ast
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@en
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@nl
type
label
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@ast
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@en
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@nl
prefLabel
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@ast
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@en
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@nl
P2093
P2860
P356
P1476
The stimulation of glycolysis ...... ible 6-phosphofructo-2-kinase.
@en
P2093
Anne-Sophie Marsin
Caroline Bouzin
Luc Bertrand
P2860
P304
30778-30783
P356
10.1074/JBC.M205213200
P407
P577
2002-06-13T00:00:00Z