Genetic deletion and pharmacological inhibition of Nogo-66 receptor impairs cognitive outcome after traumatic brain injury in mice.
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Genetic manipulation of cell death and neuroplasticity pathways in traumatic brain injuryThe Controlled Cortical Impact Model: Applications, Considerations for Researchers, and Future DirectionsNogo-A expression in the brain of mice with cerebral malariaInteraction between amyloid precursor protein and Nogo receptors regulates amyloid deposition.Nogo-A expression dynamically varies after spinal cord injury.Animal modelling of traumatic brain injury in preclinical drug development: where do we go from here?Chinese Medicine's Intervention Effect on Nogo-A/NgRHippocampal expression of myelin-associated inhibitors is induced with age-related cognitive decline and correlates with deficits of spatial learning and memory.Aging, synaptic dysfunction, and insulin-like growth factor (IGF)-1Lateralized response of dynorphin a peptide levels after traumatic brain injury.Indomethacin protects rats from neuronal damage induced by traumatic brain injury and suppresses hippocampal IL-1β release through the inhibition of Nogo-A expression.Chronic Histopathological and Behavioral Outcomes of Experimental Traumatic Brain Injury in Adult Male Animals.Inhibition of Nogo-66 receptor 1 enhances recovery of cognitive function after traumatic brain injury in mice.The Nogo-66 receptor family in the intact and diseased CNS.Functional assessment of long-term deficits in rodent models of traumatic brain injury.Traumatic brain injury using mouse models.Peptide Pharmacological Approaches to Treating Traumatic Brain Injury: a Case for Arginine-Rich Peptides.Posttraumatic mossy fiber sprouting is related to the degree of cortical damage in three mouse strainsEscalated handling of young C57BL/6 mice results in altered Morris water maze performance.Facilitated assessment of tissue loss following traumatic brain injuryNogo presence is inversely associated with shifts in cortical microglial morphology following experimental diffuse brain injury.Lacosamide improves outcome in a murine model of traumatic brain injury.
P2860
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P2860
Genetic deletion and pharmacological inhibition of Nogo-66 receptor impairs cognitive outcome after traumatic brain injury in mice.
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2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@ast
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@en
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@nl
type
label
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@ast
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@en
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@nl
prefLabel
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@ast
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@en
Genetic deletion and pharmacol ...... raumatic brain injury in mice.
@nl
P2093
P2860
P50
P356
P1476
Genetic deletion and pharmacol ...... traumatic brain injury in mice
@en
P2093
Daniel Lee
David A Gimbel
Kristine Jansson
Maria Björk
Ola Philipson
Tracy K McIntosh
P2860
P304
P356
10.1089/NEU.2009.1255
P50
P577
2010-07-01T00:00:00Z