Cardiac restricted overexpression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarction - Wikidata - awgldk - TriplyDB

Cardiac restricted overexpression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarction

Cardiac restricted overexpression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarction

http://www.wikidata.org/entity/Q34144778

about

Targeting matrix metalloproteinases in heart disease: lessons from endogenous inhibitors Translating Koch's postulates to identify matrix metalloproteinase roles in postmyocardial infarction remodeling: cardiac metalloproteinase actions (CarMA) postulates Membrane-associated matrix proteolysis and heart failure.Heterogeneity in MT1-MMP activity with ischemia-reperfusion and previous myocardial infarction: relation to regional myocardial function.Diabetes influences cardiac extracellular matrix remodelling after myocardial infarction and subsequent development of cardiac dysfunction.Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodeling Targeted regional injection of biocomposite microspheres alters post-myocardial infarction remodeling and matrix proteolytic pathways.Diversity of myocardial interstitial proteolytic pathways: gene deletion reveals unexpected consequences Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature.Pressure overload-dependent membrane type 1-matrix metalloproteinase induction: relationship to LV remodeling and fibrosis.MT1-MMP-dependent remodeling of cardiac extracellular matrix structure and function following myocardial infarction The history of matrix metalloproteinases: milestones, myths, and misperceptions.Cellular mechanisms of tissue fibrosis. 2. Contributory pathways leading to myocardial fibrosis: moving beyond collagen expression.Differential membrane type 1 matrix metalloproteinase substrate processing with ischemia-reperfusion: relationship to interstitial microRNA dynamics and myocardial function.Using the laws of thermodynamics to understand how matrix metalloproteinases coordinate the myocardial response to injury Aging and the cardiac collagen matrix: Novel mediators of fibrotic remodelling Myocardial Infarction Superimposed on Aging: MMP-9 Deletion Promotes M2 Macrophage Polarization.Cyclosporin A in left ventricular remodeling after myocardial infarction.Mechanistic relationship between membrane type-1 matrix metalloproteinase and the myocardial response to pressure overload.Regulation of myocardial matrix metalloproteinase expression and activity by cardiac fibroblasts.Involvement of monocytes/macrophages as key factors in the development and progression of cardiovascular diseases.Matrix metalloproteinases are involved in cardiovascular diseases.Cardiac Physiology of Aging: Extracellular Considerations.Development of Radiolabeled Membrane Type-1 Matrix Metalloproteinase Activatable Cell Penetrating Peptide Imaging Probes.Downregulation of RACK1 is associated with cardiomyocyte apoptosis after myocardial ischemia/reperfusion injury in adult rats.Alcohol modulation of cardiac matrix metalloproteinases (MMPs) and tissue inhibitors of MMPs favors collagen accumulation.PGE2 reduces MMP-14 and increases plasminogen activator inhibitor-1 in cardiac fibroblasts.Collagenolytic Activity Is Associated with Scar Resolution in Zebrafish Hearts after Cryoinjury.Matrix metalloproteinase-14 is a biomarker of angiogenic activity in proliferative diabetic retinopathy.

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P2860

Cardiac restricted overexpression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarction

http://www.wikidata.org/entity/Q34144778

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Cardiac restricted overexpress ...... ollowing myocardial infarction

@ast

Cardiac restricted overexpress ...... ollowing myocardial infarction

@en

Cardiac restricted overexpress ...... ollowing myocardial infarction

@nl

type

label

Cardiac restricted overexpress ...... ollowing myocardial infarction

@ast

Cardiac restricted overexpress ...... ollowing myocardial infarction

@en

Cardiac restricted overexpress ...... ollowing myocardial infarction

@nl

prefLabel

Cardiac restricted overexpress ...... ollowing myocardial infarction

@ast

Cardiac restricted overexpress ...... ollowing myocardial infarction

@en

Cardiac restricted overexpress ...... ollowing myocardial infarction

@nl

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JBC.M110.158196

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Journal of Biological Chemistry

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Cardiac restricted overexpress ...... ollowing myocardial infarction

@en

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Albert J Sinusas

http://www.w3.org/2001/XMLSchema#string

Christine N Koval

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Francis G Spinale

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Juozas A Zavadzkas

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Robert E Stroud

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Rupak Mukherjee

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Shenikqua Bouges

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P2860

Mechanism of cell surface activation of 72-kDa type IV collagenase. Isolation of the activated form of the membrane metalloprotease

Dysregulation of TGF-beta activation contributes to pathogenesis in Marfan syndrome

Membrane protease proteomics: Isotope-coded affinity tag MS identification of undescribed MT1-matrix metalloproteinase substrates

Isolation and characterization of the mouse cardiac myosin heavy chain genes.

Latency, activation, and binding proteins of TGF-beta.

Expression of matrix metalloproteinases during rat skin wound healing: evidence that membrane type-1 matrix metalloproteinase is a stromal activator of pro-gelatinase A

Relevance of matrix metalloproteinases and their inhibitors after myocardial infarction: a temporal and spatial window.

The importance of estimating the therapeutic index in the development of matrix metalloproteinase inhibitors.

MT1-MMP is the critical determinant of matrix degradation and invasion by ovarian cancer cells.

The role of TGF-beta signaling in myocardial infarction and cardiac remodeling

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10.1074/JBC.M110.158196

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39

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285

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Wawrzyniec L Dobrucki

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2010-07-19T00:00:00Z

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20643648

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