Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature.
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Antifibrotic therapies to control cardiac fibrosisAnimal models of heart failure with preserved ejection fractionGender differences in non-ischemic myocardial remodeling: are they due to estrogen modulation of cardiac mast cells and/or membrane type 1 matrix metalloproteinaseMembrane-associated matrix proteolysis and heart failure.Substance P acting via the neurokinin-1 receptor regulates adverse myocardial remodeling in a rat model of hypertension.Role of cardiac magnetic resonance in the evaluation of dilated cardiomyopathy: diagnostic contribution and prognostic significance.Cardiac-restricted overexpression or deletion of tissue inhibitor of matrix metalloproteinase-4: differential effects on left ventricular structure and function following pressure overload-induced hypertrophy.Non-human primate and rat cardiac fibroblasts show similar extracellular matrix-related and cellular adhesion gene responses to substance P.Increased stiffness is the major early abnormality in a pig model of severe aortic stenosis and predisposes to congestive heart failure in the absence of systolic dysfunction.Heart failure with preserved ejection fraction: chronic low-intensity interval exercise training preserves myocardial O2 balance and diastolic function.Cellular mechanisms of tissue fibrosis. 2. Contributory pathways leading to myocardial fibrosis: moving beyond collagen expression.Mitigation of myocardial fibrosis by molecular cardiac surgery-mediated gene overexpression.Late Gadolinium Enhancement Amount As an Independent Risk Factor for the Incidence of Adverse Cardiovascular Events in Patients with Stage C or D Heart FailureMechanotransduction and fibrosis.Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution.Saxagliptin and Tadalafil Differentially Alter Cyclic Guanosine Monophosphate (cGMP) Signaling and Left Ventricular Function in Aortic-Banded Mini-Swine.Porcine model of progressive cardiac hypertrophy and fibrosis with secondary postcapillary pulmonary hypertension.Effect of QiShenYiQi Pill on Myocardial Collagen Metabolism in Rats with Partial Abdominal Aortic Coarctation.Multiple common co-morbidities produce left ventricular diastolic dysfunction associated with coronary microvascular dysfunction, oxidative stress and myocardial stiffening.Imaging technologies for cardiac fiber and heart failure: a review.
P2860
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P2860
Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Progressive induction of left ...... and a unique matrix signature.
@ast
Progressive induction of left ...... and a unique matrix signature.
@en
type
label
Progressive induction of left ...... and a unique matrix signature.
@ast
Progressive induction of left ...... and a unique matrix signature.
@en
prefLabel
Progressive induction of left ...... and a unique matrix signature.
@ast
Progressive induction of left ...... and a unique matrix signature.
@en
P2093
P2860
P1476
Progressive induction of left ...... and a unique matrix signature.
@en
P2093
Francis G Spinale
J Marshall Oelsen
Jennifer A Dixon
John S Ikonomidis
Michael R Zile
Robert E Stroud
Rupak Mukherjee
Shaina R Eckhouse
William M Yarbrough
William T Rivers
P2860
P304
P356
10.1016/J.JTCVS.2011.09.032
P407
P577
2011-11-04T00:00:00Z