HDAC3 is a critical negative regulator of long-term memory formation. - Wikidata - awgldk - TriplyDB

HDAC3 is a critical negative regulator of long-term memory formation.

HDAC3 is a critical negative regulator of long-term memory formation.

http://www.wikidata.org/entity/Q34158695

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Inhibition of histone deacetylases facilitates extinction and attenuates reinstatement of nicotine self-administration in rats An epigenetic blockade of cognitive functions in the neurodegenerating brain Where Environment Meets Cognition: A Focus on Two Developmental Intellectual Disability Disorders Making Sense of Epigenetics Dendritic Spines in Depression: What We Learned from Animal Models Sensory Cortical Plasticity Participates in the Epigenetic Regulation of Robust Memory Formation.Epigenetic Alterations in Alzheimer's Disease Sex steroid hormones matter for learning and memory: estrogenic regulation of hippocampal function in male and female rodents Epigenetics, oestradiol and hippocampal memory consolidation Small molecule inhibitors of zinc-dependent histone deacetylases Epigenetic mechanisms in experience-driven memory formation and behavior Role of nucleosome remodeling in neurodevelopmental and intellectual disability disorders Transcriptional co-repressors and memory storage Does PKM(zeta) maintain memory?A role for histone deacetylases in the cellular and behavioral mechanisms underlying learning and memory Epigenetics of Alzheimer's disease and frontotemporal dementia HDAC6 as a target for neurodegenerative diseases: what makes it different from the other HDACs?Epigenetic mechanisms in mood disorders: targeting neuroplasticity Overexpression of SIRT6 in the hippocampal CA1 impairs the formation of long-term contextual fear memory Age-related memory impairment is associated with disrupted multivariate epigenetic coordination in the hippocampus The promise and perils of HDAC inhibitors in neurodegeneration Inhibition of histone deacetylase 3 causes replication stress in cutaneous T cell lymphoma G9a/GLP histone lysine dimethyltransferase complex activity in the hippocampus and the entorhinal cortex is required for gene activation and silencing during memory consolidation Aberrant histone deacetylase2-mediated histone modifications and synaptic plasticity in the amygdala predisposes to anxiety and alcoholism.Genetic knock-down of HDAC3 does not modify disease-related phenotypes in a mouse model of Huntington's disease Histone Deacetylase Inhibition via RGFP966 Releases the Brakes on Sensory Cortical Plasticity and the Specificity of Memory Formation Reducing HDAC6 ameliorates cognitive deficits in a mouse model for Alzheimer's disease Class I HDAC inhibition blocks cocaine-induced plasticity by targeted changes in histone methylation.Preferential loss of dorsal-hippocampus synapses underlies memory impairments provoked by short, multimodal stress.Active, phosphorylated fingolimod inhibits histone deacetylases and facilitates fear extinction memory.Comparative modeling and benchmarking data sets for human histone deacetylases and sirtuin families.Methamphetamine-associated memory is regulated by a writer and an eraser of permissive histone methylation.Pharmacological enhancement of memory or cognition in normal subjects MRI uncovers disrupted hippocampal microstructure that underlies memory impairments after early-life adversity.The histone deacetylase HDAC3 is essential for Purkinje cell function, potentially complicating the use of HDAC inhibitors in SCA1 A selective inhibitor of histone deacetylase 3 prevents cognitive deficits and suppresses striatal CAG repeat expansions in Huntington's disease mice Reassessing the effects of histone deacetylase inhibitors on hippocampal memory and cognitive aging.CBP in the nucleus accumbens regulates cocaine-induced histone acetylation and is critical for cocaine-associated behaviors.A novel class of small molecule inhibitors of HDAC6.17β-Estradiol regulates histone alterations associated with memory consolidation and increases Bdnf promoter acetylation in middle-aged female mice.

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HDAC3 is a critical negative regulator of long-term memory formation.

http://www.wikidata.org/entity/Q34158695

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

HDAC3 is a critical negative regulator of long-term memory formation.

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HDAC3 is a critical negative regulator of long-term memory formation.

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HDAC3 is a critical negative regulator of long-term memory formation.

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type

label

HDAC3 is a critical negative regulator of long-term memory formation.

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HDAC3 is a critical negative regulator of long-term memory formation.

@en

HDAC3 is a critical negative regulator of long-term memory formation.

@nl

prefLabel

HDAC3 is a critical negative regulator of long-term memory formation.

@ast

HDAC3 is a critical negative regulator of long-term memory formation.

@en

HDAC3 is a critical negative regulator of long-term memory formation.

@nl

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JNEUROSCI.5052-10.2011

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Journal of Neuroscience

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HDAC3 is a critical negative regulator of long-term memory formation.

@en

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Dina P Matheos

http://www.w3.org/2001/XMLSchema#string

George A Rogge

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James R Rusche

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Marcelo A Wood

http://www.w3.org/2001/XMLSchema#string

Mitchell A Lazar

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Rebecca J Post

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Ruth M Barrett

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Shannon E Mullican

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Steven Jones

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Susan C McQuown

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P2860

A core SMRT corepressor complex containing HDAC3 and TBL1, a WD40-repeat protein linked to deafness

Regulation of histone deacetylase 4 and 5 and transcriptional activity by 14-3-3-dependent cellular localization

Enzymatic activity associated with class II HDACs is dependent on a multiprotein complex containing HDAC3 and SMRT/N-CoR

The nuclear receptor corepressor deacetylase activating domain is essential for repression by thyroid hormone receptor

Both corepressor proteins SMRT and N-CoR exist in large protein complexes containing HDAC3

HDAC2 negatively regulates memory formation and synaptic plasticity.

A new mathematical model for relative quantification in real-time RT-PCR

Chromatin modifications and their function

Histone deacetylases associated with the mSin3 corepressor mediate mad transcriptional repression

CBP histone acetyltransferase activity is a critical component of memory consolidation

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764-774

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scholarly article

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10.1523/JNEUROSCI.5052-10.2011

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2

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21228185

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3160172

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