Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys - Wikidata - awgldk - TriplyDB

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

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Scientific Statement on the Diagnostic Criteria, Epidemiology, Pathophysiology, and Molecular Genetics of Polycystic Ovary Syndrome.Developmental programming: exposure to testosterone excess disrupts steroidal and metabolic environment in pregnant sheep In an Ovine Model of Polycystic Ovary Syndrome (PCOS) Prenatal Androgens Suppress Female Fetal Renal Gluconeogenesis Prenatal Testosterone Exposure Leads to Gonadal Hormone-Dependent Hyperinsulinemia and Gonadal Hormone-Independent Glucose Intolerance in Adult Male Rat Offspring Developmental programming of polycystic ovary syndrome (PCOS): prenatal androgens establish pancreatic islet α/β cell ratio and subsequent insulin secretion Role of Sex Steroids in β Cell Function, Growth, and Survival.Clustering of PCOS-like traits in naturally hyperandrogenic female rhesus monkeys.Translational Insight Into Polycystic Ovary Syndrome (PCOS) From Female Monkeys with PCOS-like Traits.The Pathogenesis of Polycystic Ovary Syndrome (PCOS): The Hypothesis of PCOS as Functional Ovarian Hyperandrogenism Revisited.Apical Membrane Alterations in Non-intestinal Organs in Microvillus Inclusion Disease.Sex Differences in Androgen Regulation of Metabolism in Nonhuman Primates.The role of fructose‑1,6‑bisphosphatase 1 in abnormal development of ovarian follicles caused by high testosterone concentration.Genetic Variants Associated with Hyperandrogenemia in PCOS Pathophysiology.

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Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

http://www.wikidata.org/entity/Q34159978

description

2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

@ast

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

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Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

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type

label

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

@ast

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

@en

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

@nl

prefLabel

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

@ast

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

@en

Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

@nl

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JOURNAL.PONE.0106527

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Abnormal infant islet morphology precedes insulin resistance in PCOS-like monkeys

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Alice F Tarantal

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Daniel A Dumesic

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David H Abbott

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Lindsey E Nicol

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Timothy D O'Brien

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Tristan Grogan

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P2860

Epigenetic mechanism underlying the development of polycystic ovary syndrome (PCOS)-like phenotypes in prenatally androgenized rhesus monkeys

Nonhuman primate models of polycystic ovary syndrome

Criteria for Defining Polycystic Ovary Syndrome as a Predominantly Hyperandrogenic Syndrome: An Androgen Excess Society Guideline

Prenatal androgen exposure programs metabolic dysfunction in female mice.

Adaptive changes in pancreatic beta cell fractional area and beta cell turnover in human pregnancy.

The in utero programming effect of increased maternal androgens and a direct fetal intervention on liver and metabolic function in adult sheep.

Fetal, infant, adolescent and adult phenotypes of polycystic ovary syndrome in prenatally androgenized female rhesus monkeys.

Developmental programming: gestational testosterone treatment alters fetal ovarian gene expression.

Pancreas: a sex steroid-dependent tissue.

Experimentally induced gestational androgen excess disrupts glucoregulation in rhesus monkey dams and their female offspring.

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