Virulence genes in the pathogenic yeast Candida albicans.
about
Control of the C. albicans cell wall damage response by transcriptional regulator Cas5.Cryptosporidium pathogenicity and virulenceSpt3 plays opposite roles in filamentous growth in Saccharomyces cerevisiae and Candida albicans and is required for C. albicans virulence.The biology of mating in Candida albicansA forkhead transcription factor is important for true hyphal as well as yeast morphogenesis in Candida albicans.CaMtw1, a member of the evolutionarily conserved Mis12 kinetochore protein family, is required for efficient inner kinetochore assembly in the pathogenic yeast Candida albicans.Chemically engineered extracts as an alternative source of bioactive natural product-like compounds.Early-expressed chemokines predict kidney immunopathology in experimental disseminated Candida albicans infectionsStrains and strategies for large-scale gene deletion studies of the diploid human fungal pathogen Candida albicansZebrafish as a model host for Candida albicans infection.Wild-type Drosophila melanogaster as an alternative model system for investigating the pathogenicity of Candida albicans.Transcriptional regulators Cph1p and Efg1p mediate activation of the Candida albicans virulence gene SAP5 during infectionThe CENP-A homolog CaCse4p in the pathogenic yeast Candida albicans is a centromere protein essential for chromosome transmission.Expressed sequence tag analysis of the human pathogen Paracoccidioides brasiliensis yeast phase: identification of putative homologues of Candida albicans virulence and pathogenicity genes.Rapid transcriptome characterization and parsing of sequences in a non-model host-pathogen interaction; pea-Sclerotinia sclerotiorum.Effects of depleting the essential central metabolic enzyme fructose-1,6-bisphosphate aldolase on the growth and viability of Candida albicans: implications for antifungal drug target discovery.Dynamic transcript profiling of Candida albicans infection in zebrafish: a pathogen-host interaction study.Roles of Candida albicans Dfg5p and Dcw1p cell surface proteins in growth and hypha formation.Candida albicans Cas5, a regulator of cell wall integrity, is required for virulence in murine and toll mutant fly models.Effect of farnesol on a mouse model of systemic candidiasis, determined by use of a DPP3 knockout mutant of Candida albicans.Calcineurin is essential for virulence in Candida albicans.Screening of Pharmacologically Active Small Molecule Compounds Identifies Antifungal Agents Against Candida BiofilmsCandida, still number one--what do we know and where are we going from there?Relationship between Candida albicans virulence during experimental hematogenously disseminated infection and endothelial cell damage in vitro.Candida albicans response regulator gene SSK1 regulates a subset of genes whose functions are associated with cell wall biosynthesis and adaptation to oxidative stress.The structure of the Candida albicans Ess1 prolyl isomerase reveals a well-ordered linker that restricts domain mobilityAnimal models: an important tool in mycology.The Candida albicans Pho4 Transcription Factor Mediates Susceptibility to Stress and Influences Fitness in a Mouse Commensalism Model.SKN7 of Candida albicans: mutant construction and phenotype analysisAntifungal activity of Cymbopogon winterianus jowitt ex bor against Candida albicansEctopic expression of URA3 can influence the virulence phenotypes and proteome of Candida albicans but can be overcome by targeted reintegration of URA3 at the RPS10 locus.Candida albicans transcription factor Rim101 mediates pathogenic interactions through cell wall functions.Trafficking of heme and porphyrins in metazoa.Massive induction of innate immune response to Candida albicans in the kidney in a murine intravenous challenge model.The role of the cell wall in fungal pathogenesis.The moonlighting protein Tsa1p is implicated in oxidative stress response and in cell wall biogenesis in Candida albicans.The Sho1 adaptor protein links oxidative stress to morphogenesis and cell wall biosynthesis in the fungal pathogen Candida albicans.Identification and characterization of a Candida albicans mating pheromone.Temporal events in the intravenous challenge model for experimental Candida albicans infections in female mice.Inactivation of transcription factor gene ACE2 in the fungal pathogen Candida glabrata results in hypervirulence.
P2860
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P2860
Virulence genes in the pathogenic yeast Candida albicans.
description
2001 nî lūn-bûn
@nan
2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Virulence genes in the pathogenic yeast Candida albicans.
@ast
Virulence genes in the pathogenic yeast Candida albicans.
@en
Virulence genes in the pathogenic yeast Candida albicans.
@nl
type
label
Virulence genes in the pathogenic yeast Candida albicans.
@ast
Virulence genes in the pathogenic yeast Candida albicans.
@en
Virulence genes in the pathogenic yeast Candida albicans.
@nl
prefLabel
Virulence genes in the pathogenic yeast Candida albicans.
@ast
Virulence genes in the pathogenic yeast Candida albicans.
@en
Virulence genes in the pathogenic yeast Candida albicans.
@nl
P2093
P1476
Virulence genes in the pathogenic yeast Candida albicans.
@en
P2093
Navarro-García F
P304
P356
10.1016/S0168-6445(00)00066-8
P577
2001-04-01T00:00:00Z