XPB and XPD helicases in TFIIH orchestrate DNA duplex opening and damage verification to coordinate repair with transcription and cell cycle via CAK kinase.
about
DNA damage and repair in plants - from models to cropsDouble strand binding-single strand incision mechanism for human flap endonuclease: implications for the superfamilyRoles of DNA helicases in the maintenance of genome integrityUnpairing and gating: sequence-independent substrate recognition by FEN superfamily nucleasesEmerging critical roles of Fe-S clusters in DNA replication and repair.Eukaryotic Class II Cyclobutane Pyrimidine Dimer Photolyase Structure Reveals Basis for Improved Ultraviolet Tolerance in PlantsAtl1 Regulates Choice between Global Genome and Transcription-Coupled Repair of O6-AlkylguaninesA structure-specific nucleic acid-binding domain conserved among DNA repair proteinsFunction of Conserved Topological Regions within the Saccharomyces cerevisiae Basal Transcription Factor TFIIHDouble-stranded DNA translocase activity of transcription factor TFIIH and the mechanism of RNA polymerase II open complex formation.Envisioning the dynamics and flexibility of Mre11-Rad50-Nbs1 complex to decipher its roles in DNA replication and repairNucleotide excision repair efficiencies of bulky carcinogen-DNA adducts are governed by a balance between stabilizing and destabilizing interactionsImplementation and performance of SIBYLS: a dual endstation small-angle X-ray scattering and macromolecular crystallography beamline at the Advanced Light Source.Architecture of the Human and Yeast General Transcription and DNA Repair Factor TFIIHG quadruplexes are genomewide targets of transcriptional helicases XPB and XPD.DNA Repair Pathway Alterations in Bladder Cancer.Structure, assembly and dynamics of macromolecular complexes by single particle cryo-electron microscopy.Nanobiomotors of archaeal DNA repair machineries: current research status and application potentialXRCC4 and XLF form long helical protein filaments suitable for DNA end protection and alignment to facilitate DNA double strand break repair.Nucleotide excision repair/transcription gene defects in the fetus and impaired TFIIH-mediated function in transcription in placenta leading to preeclampsia.XPA: A key scaffold for human nucleotide excision repair.Impact of age-associated cyclopurine lesions on DNA repair helicases.Blinded by the UV light: how the focus on transcription-coupled NER has distracted from understanding the mechanisms of Cockayne syndrome neurologic diseaseActive transcriptomic and proteomic reprogramming in the C. elegans nucleotide excision repair mutant xpa-1.TFIIH-dependent MMP-1 overexpression in trichothiodystrophy leads to extracellular matrix alterations in patient skin.Escherichia coli radD (yejH) gene: a novel function involved in radiation resistance and double-strand break repair.New mutation in the mouse Xpd/Ercc2 gene leads to recessive cataracts.ATP-stimulated, DNA-mediated redox signaling by XPD, a DNA repair and transcription helicase.DNA charge transport as a first step in coordinating the detection of lesions by repair proteinsTripartite DNA Lesion Recognition and Verification by XPC, TFIIH, and XPA in Nucleotide Excision Repair.DNA helicases involved in DNA repair and their roles in cancerMechanism of release and fate of excised oligonucleotides during nucleotide excision repairMolecular mechanisms of xeroderma pigmentosum (XP) proteins.The Cellular Response to Oxidatively Induced DNA Damage and Polymorphism of Some DNA Repair Genes Associated with Clinicopathological Features of Bladder Cancer.Transcriptional profiling reveals elevated Sox2 in DNA polymerase ß null mouse embryonic fibroblasts.ARCH domain of XPD, an anchoring platform for CAK that conditions TFIIH DNA repair and transcription activitiesValosin-containing Protein (VCP)/p97 Segregase Mediates Proteolytic Processing of Cockayne Syndrome Group B (CSB) in Damaged Chromatin.Mechanism of DNA loading by the DNA repair helicase XPD.Abnormal XPD-induced nuclear receptor transactivation in DNA repair disorders: trichothiodystrophy and xeroderma pigmentosum.Somatic ERCC2 mutations are associated with a distinct genomic signature in urothelial tumors.
P2860
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P2860
XPB and XPD helicases in TFIIH orchestrate DNA duplex opening and damage verification to coordinate repair with transcription and cell cycle via CAK kinase.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@ast
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@en
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@nl
type
label
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@ast
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@en
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@nl
prefLabel
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@ast
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@en
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase.
@nl
P2860
P1433
P1476
XPB and XPD helicases in TFIIH ...... and cell cycle via CAK kinase
@en
P2093
Jill O Fuss
P2860
P304
P356
10.1016/J.DNAREP.2011.04.028
P577
2011-05-14T00:00:00Z