Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy). - Wikidata - awgldk - TriplyDB

Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

http://www.wikidata.org/entity/Q34187223

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Overview of ventilation strategies for the early management of intubated preterm infants Affect of Early Life Oxygen Exposure on Proper Lung Development and Response to Respiratory Viral Infections Lung function following very preterm birth in the era of 'new' bronchopulmonary dysplasia Suppression of inflammatory cell trafficking and alveolar simplification by the heme oxygenase-1 product carbon monoxide Neonatal oxygen increases sensitivity to influenza A virus infection in adult mice by suppressing epithelial expression of Ear1 Lung volume, breathing pattern and ventilation inhomogeneity in preterm and term infants.Clara Cell Protein Expression in Mechanically Ventilated Term and Preterm Infants with Respiratory Distress Syndrome and at Risk of Bronchopulmonary Dysplasia: A Pilot Study.Chronic vitamin D deficiency induces lung fibrosis through activation of the renin-angiotensin system.Neonatal hyperoxia stimulates the expansion of alveolar epithelial type II cells Peroxisome proliferator-activated receptor-g agonist treatment increases septation and angiogenesis and decreases airway hyperresponsiveness in a model of experimental neonatal chronic lung disease.Reduced platelet-derived growth factor receptor expression is a primary feature of human bronchopulmonary dysplasia.Neonatal hyperoxia leads to persistent alterations in NK responses to influenza A virus infection.Neonatal hyperoxia causes pulmonary vascular disease and shortens life span in aging mice Continuing care of NICU graduates.Mesenchymal stromal cells from neonatal tracheal aspirates demonstrate a pattern of lung-specific gene expression.Long-term impact of bronchopulmonary dysplasia on pulmonary function.Effect of Hyperoxia on Retinoid Metabolism and Retinoid Receptor Expression in the Lungs of Newborn Mice.Lung development and the host response to influenza A virus are altered by different doses of neonatal oxygen in mice.Bone marrow-derived c-kit+ cells attenuate neonatal hyperoxia-induced lung injury.Neonatal hyperoxia enhances the inflammatory response in adult mice infected with influenza A virus.Chest computed tomography findings in bronchopulmonary dysplasia and correlation with lung function.Mechanisms of bronchopulmonary dysplasia.Absence of TNF-α enhances inflammatory response in the newborn lung undergoing mechanical ventilation.Catch-up alveolarization in ex-preterm children: evidence from (3)He magnetic resonance.Low birth weight and respiratory disease in adulthood: a population-based case-control study.Growth, lung function, and physical activity in schoolchildren who were very-low-birth-weight preterm infants.Neonatal oxygen adversely affects lung function in adult mice without altering surfactant composition or activity.Isolation of tracheal aspirate mesenchymal stromal cells predicts bronchopulmonary dysplasia.Review series: What goes around, comes around: childhood influences on later lung health? Long-term follow-up of infants with lung disease of prematurity.Tracheobronchomalacia diagnosed by tracheobronchography in ventilator-dependent infants.Adult respiratory outcomes of extreme preterm birth. A regional cohort study.Sensitivity of newly defined impulse oscillometry indices in preschool children.Imaging Bronchopulmonary Dysplasia-A Multimodality Update.A 6 year-old boy was referred from a district hospital with congestive cardiac failure.Sex-specific differences in neonatal hyperoxic lung injury Antenatal steroid treatment reduces childhood asthma risk in very low birth weight infants without bronchopulmonary dysplasia.Lung function outcome at school age in very low birth weight children.Respiratory symptoms in the first 7 years of life and birth weight at term: the PIAMA Birth Cohort.β-Naphthoflavone treatment attenuates neonatal hyperoxic lung injury in wild type and Cyp1a2-knockout mice.Neonatal hyperoxia depletes pulmonary vein cardiomyocytes in adult mice via mitochondrial oxidation.

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Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

http://www.wikidata.org/entity/Q34187223

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2001 թվականի ապրիլին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

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Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

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Long term sequelae of bronchopulmonary dysplasia

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Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

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Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

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Long term sequelae of bronchopulmonary dysplasia

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Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

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Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

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Long term sequelae of bronchopulmonary dysplasia

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P2860

Pulmonary disease following respirator therapy of hyaline-membrane disease. Bronchopulmonary dysplasia

Maternal asthma, premature birth, and the risk of respiratory morbidity in schoolchildren in Merseyside.

The new BPD: an arrest of lung development.

Abnormal pulmonary outcomes in premature infants: prediction from oxygen requirement in the neonatal period.

Is chronic lung disease in low birth weight infants preventable? A survey of eight centers.

Birth weight at term and lung function in adolescence: no evidence for a programmed effect

Respiratory health in a total very low birthweight cohort and their classroom controls

Growth and development of very low birthweight infants recovering from bronchopulmonary dysplasia

Relation of birth weight and childhood respiratory infection to adult lung function and death from chronic obstructive airways disease

Effects of prematurity and intrauterine growth on respiratory health and lung function in childhood.

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