Neonatal oxygen adversely affects lung function in adult mice without altering surfactant composition or activity. - Wikidata - awgldk - TriplyDB

Neonatal oxygen adversely affects lung function in adult mice without altering surfactant composition or activity.

Neonatal oxygen adversely affects lung function in adult mice without altering surfactant composition or activity.

http://www.wikidata.org/entity/Q37406405

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Understanding the Impact of Infection, Inflammation, and Their Persistence in the Pathogenesis of Bronchopulmonary Dysplasia Affect of Early Life Oxygen Exposure on Proper Lung Development and Response to Respiratory Viral Infections Impact of Dietary Tomato Juice on Changes in Pulmonary Oxidative Stress, Inflammation and Structure Induced by Neonatal Hyperoxia in Mice (Mus musculus)Neonatal oxygen increases sensitivity to influenza A virus infection in adult mice by suppressing epithelial expression of Ear1 Expression level and subcellular localization of heme oxygenase-1 modulates its cytoprotective properties in response to lung injury: a mouse model Periostin downregulation is an early marker of inhibited neonatal murine lung alveolar septation Postnatal inflammation in the pathogenesis of bronchopulmonary dysplasia Neonatal hyperoxia stimulates the expansion of alveolar epithelial type II cells Hyperoxia-derived lung damage in preterm infants.Long term consequences of oxygen therapy in the neonatal period.Severity of neonatal hyperoxia determines structural and functional changes in developing mouse airway.A potential role of the JNK pathway in hyperoxia-induced cell death, myofibroblast transdifferentiation and TGF-β1-mediated injury in the developing murine lung.Thioredoxin-1 redox signaling regulates cell survival in response to hyperoxia.Animal models of bronchopulmonary dysplasia. The term mouse models Perinatal oxygen in the developing lung.Neonatal hyperoxia causes pulmonary vascular disease and shortens life span in aging mice Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus.Neonatal hyperoxia contributes additively to cigarette smoke-induced chronic obstructive pulmonary disease changes in adult mice.Aberrant signaling pathways of the lung mesenchyme and their contributions to the pathogenesis of bronchopulmonary dysplasia Airway Hyperreactivity Is Delayed after Mild Neonatal Hyperoxic Exposure.Early exposure to hyperoxia or hypoxia adversely impacts cardiopulmonary development Effects of antenatal lipopolysaccharide and postnatal hyperoxia on airway reactivity and remodeling in a neonatal mouse model.rIL-10 enhances IL-10 signalling proteins in foetal alveolar type II cells exposed to hyperoxia Lung development and the host response to influenza A virus are altered by different doses of neonatal oxygen in mice.Prenatal inflammation exacerbates hyperoxia-induced functional and structural changes in adult mice.Memory CD8+ T cells are sufficient to alleviate impaired host resistance to influenza A virus infection caused by neonatal oxygen supplementation.Detoxification of Mitochondrial Oxidants and Apoptotic Signaling Are Facilitated by Thioredoxin-2 and Peroxiredoxin-3 during Hyperoxic Injury.Mechanisms of injury to the preterm lung and airway: implications for long-term pulmonary outcome.The role of hyperoxia in the pathogenesis of experimental BPD.Small molecular modulation of macrophage migration inhibitory factor in the hyperoxia-induced mouse model of bronchopulmonary dysplasia.Exposure to neonatal cigarette smoke causes durable lung changes but does not potentiate cigarette smoke-induced chronic obstructive pulmonary disease in adult mice.The Oxygen Environment at Birth Specifies the Population of Alveolar Epithelial Stem Cells in the Adult Lung.Hyperoxia and interferon-γ-induced injury in developing lungs occur via cyclooxygenase-2 and the endoplasmic reticulum stress-dependent pathway.Neonatal hyperoxic exposure persistently alters lung secretoglobins and annexin A1 Neonatal hyperoxia alters the host response to influenza A virus infection in adult mice through multiple pathways.Neonatal oxygen exposure alters airway hyper-responsiveness but not the response to allergen challenge in adult mice.Assessment of inhibited alveolar-capillary membrane structural development and function in bronchopulmonary dysplasia.Growth factors, stem cells and bronchopulmonary dysplasia.Aberrant cGMP signaling persists during recovery in mice with oxygen-induced pulmonary hypertension.Heme oxygenase-1 regulates postnatal lung repair after hyperoxia: role of β-catenin/hnRNPK signaling.

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Neonatal oxygen adversely affects lung function in adult mice without altering surfactant composition or activity.

http://www.wikidata.org/entity/Q37406405

description

article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on 17 July 2009

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vedecký článok

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Neonatal oxygen adversely affe ...... ctant composition or activity.

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Neonatal oxygen adversely affe ...... ctant composition or activity.

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Neonatal oxygen adversely affe ...... ctant composition or activity.

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Neonatal oxygen adversely affe ...... ctant composition or activity.

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AJPLUNG.00023.2009

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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Neonatal oxygen adversely affe ...... ctant composition or activity.

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David A Dean

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Michael A O'Reilly

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Min Yee

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Patricia R Chess

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Robert Gelein

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Robert H Notter

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Rui Zhou

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Sharon A McGrath-Morrow

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Zhengdong Wang

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P2860

A RAPID METHOD OF TOTAL LIPID EXTRACTION AND PURIFICATION

Vascular endothelial growth factor gene therapy increases survival, promotes lung angiogenesis, and prevents alveolar damage in hyperoxia-induced lung injury: evidence that angiogenesis participates in alveolarization

Vascular endothelial growth factor co-ordinates proper development of lung epithelium and vasculature

Oxygen toxicity in the newborn. The effect of prolonged 100 per cent O2 exposure on the lungs of newborn mice.

Oxygen toxicity in the newborn lung: reversal of inhibition of DNA synthesis in the mouse.

The mammalian respiratory system and critical windows of exposure for children's health.

Pulmonary inflammation disrupts surfactant function during Pneumocystis carinii pneumonia.

Long term sequelae of bronchopulmonary dysplasia (chronic lung disease of infancy).

Populations at risk for developing respiratory syncytial virus and risk factors for respiratory syncytial virus severity: infants with predisposing conditions.

Exposure to hyperoxia decreases the expression of vascular endothelial growth factor and its receptors in adult rat lungs.

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10.1152/AJPLUNG.00023.2009

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4

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2009-07-17T00:00:00Z

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19617311

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2770788

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