Reperfusion injury induces apoptosis in rabbit cardiomyocytes. - Wikidata - awgldk - TriplyDB

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

http://www.wikidata.org/entity/Q34190792

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Apoptosis in myocardial ischaemia and infarction Apoptosis repressor with caspase recruitment domain protects against cell death by interfering with Bax activation The apoptosis inhibitor ARC undergoes ubiquitin-proteasomal-mediated degradation in response to death stimuli: identification of a degradation-resistant mutant Reflecting on 80 years of excellence Netrin-1 prevents ischemia/reperfusion-induced myocardial infarction via a DCC/ERK1/2/eNOS s1177/NO/DCC feed-forward mechanism Cellular mechanisms of cardioprotection by calorie restriction: state of the science and future perspectives MEKK1 suppresses oxidative stress-induced apoptosis of embryonic stem cell-derived cardiac myocytes Effect of the degree of ischaemic injury and reoxygenation time on the type of myocardial cell death in man: role of caspases.Molecular and cellular mechanisms of cardiotoxicity.Targeting the Innate Immune Response to Improve Cardiac Graft Recovery after Heart Transplantation: Implications for the Donation after Cardiac Death Acute Stress Decreases but Chronic Stress Increases Myocardial Sensitivity to Ischemic Injury in Rodents Mechanisms of oxidative stress and myocardial protection during open-heart surgery Reactive oxygen species-activated nanomaterials as theranostic agents Small molecules, big effects: the role of microRNAs in regulation of cardiomyocyte death Functional O-GlcNAc modifications: implications in molecular regulation and pathophysiology GLP-1 and cardioprotection: from bench to bedside Local oxidative stress expansion through endothelial cells--a key role for gap junction intercellular communication Lidocaine Enhances Contractile Function of Ischemic Myocardial Regions in Mouse Model of Sustained Myocardial Ischemia Past and present course of cardioprotection against ischemia-reperfusion injury Phosphoinositide 3-kinase accelerates necrotic cell death during hypoxia Cytoprotection by the modulation of mitochondrial electron transport chain: the emerging role of mitochondrial STAT3 Apoptosis repressor with a CARD domain (ARC) restrains Bax-mediated pathogenesis in dystrophic skeletal muscle Bax translocates to mitochondria of heart cells during simulated ischaemia: involvement of AMP-activated and p38 mitogen-activated protein kinases Involvement of GADD153 and cardiac ankyrin repeat protein in cardiac ischemia-reperfusion injury Lipopolysaccharide improves cardiomyocyte survival and function after serum deprivation cGMP-dependent protein kinase type I inhibits TAB1-p38 mitogen-activated protein kinase apoptosis signaling in cardiac myocytes Imaging Myocardial Ischemia and Reperfusion Injury via Cy5.5-Annexin V.Liposomes in diagnosis and treatment of cardiovascular disorders.Reduction of ischemia and reperfusion-induced myocardial damage by cytochrome P450 inhibitors.Apoptosis in dilated cardiomyopathy.Mechanical tissue resuscitation (MTR): a nonpharmacological approach to treatment of acute myocardial infarction.Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes.Signal transduction of nitric oxide donor-induced protection in hydrogen peroxide-mediated apoptosis in H9C2 cardiomyoblasts.Modulation of ceramide content and lack of apoptosis in the chronically hypoxic neonatal rat heart.Simvastatin induces apoptosis of cultured rat cardiomyocytes.A novel method to determine specificity and sensitivity of the TUNEL reaction in the quantitation of apoptosis.Near infrared light protects cardiomyocytes from hypoxia and reoxygenation injury by a nitric oxide dependent mechanism.Effects of calcium channel blockers on cellular apoptosis: implications for carcinogenic potential.Protective effect of picroside II on myocardial ischemia reperfusion injury in rats Cellular stress responses: cell survival and cell death

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P2860

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

http://www.wikidata.org/entity/Q34190792

description

1994 nî lūn-bûn

@nan

1994 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1994 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

1994年の論文

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1994年学术文章

@wuu

1994年学术文章

@zh-cn

1994年学术文章

@zh-hans

1994年学术文章

@zh-my

1994年学术文章

@zh-sg

1994年學術文章

@yue

name

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@ast

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@en

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@nl

type

label

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@ast

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@en

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@nl

prefLabel

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@ast

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@en

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@nl

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Journal of Clinical Investigation

P1476

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

@en

P2093

B M Babior

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K O Burleson

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R A Gottlieb

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R A Kloner

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R L Engler

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P2860

Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

Myocardial reperfusion: a double-edged sword?

Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation

Deleterious effects of oxygen radicals in ischemia/reperfusion. Resolved and unresolved issues

Cell death: the significance of apoptosis

Regulation of intercellular adhesion molecule-1 (ICAM-1) in ischemic and reperfused canine myocardium.

Dose-dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli.

Adherence of neutrophils to canine cardiac myocytes in vitro is dependent on intercellular adhesion molecule-1.

Macrophage phagocytosis of aging neutrophils in inflammation. Programmed cell death in the neutrophil leads to its recognition by macrophages

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7929838

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reperfusion injury

apoptotic process

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295322

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