Endogenously released ATP mediates shear stress-induced Ca2+ influx into pulmonary artery endothelial cells. - Wikidata - awgldk - TriplyDB

Endogenously released ATP mediates shear stress-induced Ca2+ influx into pulmonary artery endothelial cells.

Endogenously released ATP mediates shear stress-induced Ca2+ influx into pulmonary artery endothelial cells.

http://www.wikidata.org/entity/Q34192793

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Purinergic signaling in inflammatory renal disease MYADM controls endothelial barrier function through ERM-dependent regulation of ICAM-1 expression Acetylcholine released by endothelial cells facilitates flow-mediated dilatation Connexin 40 and ATP-dependent intercellular calcium wave in renal glomerular endothelial cells Transepithelial pressure pulses induce nucleotide release in polarized MDCK cells.High oxygen tension constricts epineurial arterioles of the rat sciatic nerve via reactive oxygen species.Lysophosphatidic acid induces shear stress-dependent Ca2+ influx in mouse aortic endothelial cells in situ.The roles of P2Y2 purinergic receptors in osteoblasts and mechanotransduction Ca2+-mediated ascorbate release from coronary artery endothelial cells.P2X3 receptors and peripheral pain mechanisms.Exercise hyperemia and vasoconstrictor responses in humans with cystic fibrosis P2Y₂ and Gq/G₁₁ control blood pressure by mediating endothelial mechanotransduction.Update on vascular endothelial Ca(2+) signalling: A tale of ion channels, pumps and transporters Genetic Background Strongly Influences the Bone Phenotype of P2X7 Receptor Knockout Mice Endothelium-Derived Hyperpolarization and Coronary Vasodilation: Diverse and Integrated Roles of Epoxyeicosatrienoic Acids, Hydrogen Peroxide, and Gap Junctions.Autocrine ATP release coupled to extracellular pyrophosphate accumulation in vascular smooth muscle cells.PI3K, Rho, and ROCK play a key role in hypoxia-induced ATP release and ATP-stimulated angiogenic responses in pulmonary artery vasa vasorum endothelial cells P2Y2 receptor modulates shear stress-induced cell alignment and actin stress fibers in human umbilical vein endothelial cells.3D time-varying simulations of Ca2+ dynamics in arterial coupled cells: A massively parallel implementation.Mechanotransduction of shear in the endothelium: basic studies and clinical implications.Flow-induced ATP release in patient-specific arterial geometries--a comparative study of computational models.Loss of apical monocilia on collecting duct principal cells impairs ATP secretion across the apical cell surface and ATP-dependent and flow-induced calcium signals.Purinergic receptors in tubulointerstitial inflammatory cells: a pathophysiological mechanism of salt-sensitive hypertension.Ion Channels in Endothelial Responses to Fluid Shear Stress.Regulation of cardiac Ca2+ and ion channels by shear mechanotransduction.Possible participation of chloride ion channels in ATP release from cancer cells in suspension.Secretion of adenylate kinase 1 is required for extracellular ATP synthesis in C2C12 myotubes.Mathematical model for shear stress dependent NO and adenine nucleotide production from endothelial cells Shear stress induces a longitudinal Ca(2+) wave via autocrine activation of P2Y1 purinergic signalling in rat atrial myocytes.Mechanotransduction through growth-factor shedding into the extracellular space Conducted dilatation to ATP and K+ in rat skeletal muscle arterioles Purinergic-mediated Ca2+ influx in Dictyostelium discoideum.Modulation of ATP/ADP concentration at the endothelial cell surface by flow: effect of cell topography.Shear stress modulates endothelial KLF2 through activation of P2X4.Shear stress-induced ATP-mediated endothelial constitutive nitric oxide synthase expression in human lymphatic endothelial cells.Gravity loading induces adenosine triphosphate release and phosphorylation of extracellular signal-regulated kinases in human periodontal ligament cells.Simultaneous imaging of [Ca2+]i and intracellular NO production in freshly isolated uterine artery endothelial cells: effects of ovarian cycle and pregnancy.ATP inhibits pump activity of lymph vessels via adenosine A1 receptor-mediated involvement of NO- and ATP-sensitive K+ channels.Computer model of nucleotide transport in a realistic porcine aortic trifurcation.Purinergic receptors in human placenta: evidence for functionally active P2X4, P2X7, P2Y2, and P2Y6.

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P2860

Endogenously released ATP mediates shear stress-induced Ca2+ influx into pulmonary artery endothelial cells.

http://www.wikidata.org/entity/Q34192793

description

2003 nî lūn-bûn

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2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2003 թվականի ապրիլին հրատարակված գիտական հոդված

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年學術文章

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Endogenously released ATP medi ...... nary artery endothelial cells.

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Endogenously released ATP medi ...... nary artery endothelial cells.

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Endogenously released ATP medi ...... nary artery endothelial cells.

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Endogenously released ATP medi ...... nary artery endothelial cells.

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Endogenously released ATP medi ...... nary artery endothelial cells.

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Endogenously released ATP medi ...... nary artery endothelial cells.

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Endogenously released ATP medi ...... nary artery endothelial cells.

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AJPHEART.01155.2002

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American Journal of Physiology - Heart and Circulatory Physiology

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Akira Kamiya

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Hideki Nakatsuka

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Joji Ando

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Kimiko Yamamoto

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Norihiko Ohura

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Takaaki Sokabe

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P2860

Angiostatin binds ATP synthase on the surface of human endothelial cells

P2X(4) receptors mediate ATP-induced calcium influx in human vascular endothelial cells

Fluid shear stress activates Ca(2+) influx into human endothelial cells via P2X4 purinoceptors

Endothelial cell surface F1-F0 ATP synthase is active in ATP synthesis and is inhibited by angiostatin

Extracellular ATP formation on vascular endothelial cells is mediated by ecto-nucleotide kinase activities via phosphotransfer reactions.

Endothelial Ca2+ waves preferentially originate at specific loci in caveolin-rich cell edges.

Cystic fibrosis transmembrane regulator-independent release of ATP. Its implications for the regulation of P2Y2 receptors in airway epithelia.

Increased flow-induced ATP release from isolated vascular endothelial cells but not smooth muscle cells.

Flow modulation of agonist (ATP)-response (Ca2+) coupling in vascular endothelial cells.

Flow-induced changes in Ca2+ signaling of vascular endothelial cells: effect of shear stress and ATP.

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