Neutrophil accumulation in the lung in alpha 1-antitrypsin deficiency. Spontaneous release of leukotriene B4 by alveolar macrophages - Wikidata - awgldk - TriplyDB

Neutrophil accumulation in the lung in alpha 1-antitrypsin deficiency. Spontaneous release of leukotriene B4 by alveolar macrophages

Neutrophil accumulation in the lung in alpha 1-antitrypsin deficiency. Spontaneous release of leukotriene B4 by alveolar macrophages

http://www.wikidata.org/entity/Q34198111

about

Rationale and design of the Genomic Research in Alpha-1 Antitrypsin Deficiency and Sarcoidosis. Alpha-1 protocol Circulating monocytes from healthy individuals and COPD patients Leukocyte adhesion and recruitment, and alpha-1-antitrypsin deficiency: a report from ATS 2001, May 18-23, San Francisco Inhaled hypertonic saline for cystic fibrosis: Reviewing the potential evidence for modulation of neutrophil signalling and function Role of leukotriene A4 hydrolase aminopeptidase in the pathogenesis of emphysema.Effect of the leukotriene A4 hydrolase aminopeptidase augmentor 4-methoxydiphenylmethane in a pre-clinical model of pulmonary emphysema Role of LTB₄ in the pathogenesis of elastase-induced murine pulmonary emphysema Sputum myeloperoxidase in chronic obstructive pulmonary disease.Genetics and pulmonary medicine. 9. Molecular genetics of chronic obstructive pulmonary disease Neutrophils and protease/antiprotease imbalance.Anti-PcrV antibody in cystic fibrosis: a novel approach targeting Pseudomonas aeruginosa airway infection.Expression of human alpha1-antitrypsin in mice and dogs following AAV6 vector-mediated gene transfer to the lungs.Alpha-1-antitrypsin deficiency: what next?Factors influencing airway inflammation in chronic obstructive pulmonary disease.A review of augmentation therapy for alpha-1 antitrypsin deficiency.α1-Antitrypsin activates protein phosphatase 2A to counter lung inflammatory responses.Why has it been so difficult to prove the efficacy of alpha-1-antitrypsin replacement therapy? Insights from the study of disease pathogenesis Assessment of airway neutrophils by sputum colour: correlation with airways inflammation.Sputum chemotactic activity in chronic obstructive pulmonary disease: effect of alpha(1)-antitrypsin deficiency and the role of leukotriene B(4) and interleukin 8 Increased leukotriene B4 and 8-isoprostane in exhaled breath condensate of patients with exacerbations of COPD.Exhaled leukotrienes and prostaglandins in COPD.Inhibition of leukotriene B4-induced CD11B/CD18 (Mac-1) expression by BIIL 284, a new long acting LTB4 receptor antagonist, in patients with rheumatoid arthritis New therapies for chronic obstructive pulmonary disease Polymers of Z alpha1-antitrypsin co-localize with neutrophils in emphysematous alveoli and are chemotactic in vivo Alpha1-antitrypsin deficiency. 4: Molecular pathophysiology Elastolytic proteases: inflammation resolution and dysregulation in chronic infective lung disease.Neutrophilic inflammation and IL-8 levels in induced sputum of alpha-1-antitrypsin PiMZ subjects Proteinases and oxidants as targets in the treatment of chronic obstructive pulmonary disease.Interferon gamma induction of pulmonary emphysema in the adult murine lung COPD exacerbations . 2: aetiology Enhancing Autophagy with Drugs or Lung-directed Gene Therapy Reverses the Pathological Effects of Respiratory Epithelial Cell Proteinopathy.Collection of exhaled breath and exhaled breath condensate in veterinary medicine. A review.Roles for proteinases in the pathogenesis of chronic obstructive pulmonary disease.Matrix Metalloproteinase Expression in the Rat Myometrium During Pregnancy, Term Labor, and Postpartum Signal transduction pathways linking the activation of alveolar macrophages with the recruitment of neutrophils to lungs in chronic obstructive pulmonary disease.Augmentation therapy in alpha-1 antitrypsin deficiency: advances and controversies.Emerging drugs for alpha-1-antitrypsin deficiency.Asthma and COPD in alpha-1 antitrypsin deficiency. Evidence for the Dutch hypothesis.Lung disease associated with alpha1-antitrypsin deficiency.Alpha-1 antitrypsin: a potent anti-inflammatory and potential novel therapeutic agent.

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Neutrophil accumulation in the lung in alpha 1-antitrypsin deficiency. Spontaneous release of leukotriene B4 by alveolar macrophages

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1991 nî lūn-bûn

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Crystal RG

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Fells G

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Gadek J

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Hubbard RC

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P2860

Morphologic-physiologic correlates of the severity of fibrosis and degree of cellularity in idiopathic pulmonary fibrosis

Decreased leukotriene B4 synthesis in smokers' alveolar macrophages in vitro.

Secretory products of macrophages.

Recombinant interferon-gamma primes alveolar macrophages cultured in vitro for the release of leukotriene B4 in response to IgG stimulation

Pathogenesis of the adult respiratory distress syndrome. Evidence of oxidant activity in bronchoalveolar lavage fluid.

Relative contribution of leukotriene B4 to the neutrophil chemotactic activity produced by the resident human alveolar macrophage.

Oxidants spontaneously released by alveolar macrophages of cigarette smokers can inactivate the active site of alpha 1-antitrypsin, rendering it ineffective as an inhibitor of neutrophil elastase

Z-type alpha 1-antitrypsin is less competent than M1-type alpha 1-antitrypsin as an inhibitor of neutrophil elastase.

Mechanism for the inflammatory response in primate lungs. Demonstration and partial characterization of an alveolar macrophage-derived chemotactic factor with preferential activity for polymorphonuclear leukocytes.

Role of alveolar macrophages in asbestosis: modulation of neutrophil migration to the lung after acute asbestos exposure

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10.1172/JCI115391

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