Diabetic kidney disease in FVB/NJ Akita mice: temporal pattern of kidney injury and urinary nephrin excretion. - Wikidata - awgldk - TriplyDB

Diabetic kidney disease in FVB/NJ Akita mice: temporal pattern of kidney injury and urinary nephrin excretion.

Diabetic kidney disease in FVB/NJ Akita mice: temporal pattern of kidney injury and urinary nephrin excretion.

http://www.wikidata.org/entity/Q34229974

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Differential expression of dicer, miRNAs, and inflammatory markers in diabetic Ins2+/- Akita hearts Potential Role of Serum and Urinary Biomarkers in Diagnosis and Prognosis of Diabetic Nephropathy.Hyperfiltration is associated with the development of microalbuminuria in patients with sickle cell anemia Genetic modulation of diabetic nephropathy among mouse strains with Ins2 Akita mutation.Nephrin - a biomarker of early glomerular injury.Early-onset diabetic E1-DN mice develop albuminuria and glomerular injury typical of diabetic nephropathy.Gq signaling causes glomerular injury by activating TRPC6.Glomerulopathy in the KK.Cg-A(y) /J mouse reflects the pathology of diabetic nephropathy.Tensin2-deficient mice on FVB/N background develop severe glomerular disease.Inhibiting Amadori-modified albumin formation improves biomarkers of podocyte damage in diabetic rats.A compendium of urinary biomarkers indicative of glomerular podocytopathy.Insulin treatment attenuates renal ADAM17 and ACE2 shedding in diabetic Akita mice.Augmenting podocyte injury promotes advanced diabetic kidney disease in Akita mice.Podocyte-specific knockout of cyclooxygenase 2 exacerbates diabetic kidney disease.Original Research: Potential of urinary nephrin as a biomarker reflecting podocyte dysfunction in various kidney disease models.Expression of SHP-1 induced by hyperglycemia prevents insulin actions in podocytes.Modelling diabetic nephropathy in mice.Heterogeneous nuclear ribonucleoproteins F and K mediate insulin inhibition of renal angiotensinogen gene expression and prevention of hypertension and kidney injury in diabetic mice.Angiotensin-converting enzyme 2 amplification limited to the circulation does not protect mice from development of diabetic nephropathy.Renal protection by atorvastatin in a murine model of sickle cell nephropathy.A novel mouse model of podocyte depletion.

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P2860

Diabetic kidney disease in FVB/NJ Akita mice: temporal pattern of kidney injury and urinary nephrin excretion.

http://www.wikidata.org/entity/Q34229974

description

2012 nî lūn-bûn

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2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2012 թվականի ապրիլին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Diabetic kidney disease in FVB ...... and urinary nephrin excretion.

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Jae-Hyung Chang

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Lan Mao

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Liming Wang

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Natalie Mattocks

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Patrick J Flannery

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Phillip Ruiz

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Robert F Spurney

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Samy Hadjadj

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Seung-Yeol Paik

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Susan B Gurley

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P2860

Positionally cloned gene for a novel glomerular protein--nephrin--is mutated in congenital nephrotic syndrome

Proteinuria and perinatal lethality in mice lacking NEPH1, a novel protein with homology to NEPHRIN

Nephrin is specifically located at the slit diaphragm of glomerular podocytes

Beneficial effects of the Rho kinase inhibitor Y27632 in murine puromycin aminonucleoside nephrosis

Influence of genetic background on albuminuria and kidney injury in Ins2(+/C96Y) (Akita) mice.

Mouse models of diabetic nephropathy.

Cellular and functional defects in a mouse model of heart failure.

FVB mouse genotype confers susceptibility to OVE26 diabetic albuminuria

Podocyte biology and response to injury.

Calcineurin (CN) activation promotes apoptosis of glomerular podocytes both in vitro and in vivo

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Jean-Michel Goujon

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2012-04-04T00:00:00Z

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223978034

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22496773

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3319540

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