Cross-talk between PI3K and estrogen in the mouse thyroid predisposes to the development of follicular carcinomas with a higher incidence in females.
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Proto-oncogene PBF/PTTG1IP regulates thyroid cell growth and represses radioiodide treatmentMaternal High Estradiol Exposure is Associated with Elevated Thyroxine and Pax8 in Mouse Offspring.Estrogens and stem cells in thyroid cancer.Follicular thyroid cancers demonstrate dual activation of PKA and mTOR as modeled by thyroid-specific deletion of Prkar1a and Pten in mice.Anaplastic Thyroid Carcinoma: Current Treatments and Potential New Therapeutic Options with Emphasis on TfR1/CD71.Obatoclax overcomes resistance to cell death in aggressive thyroid carcinomas by countering Bcl2a1 and Mcl1 overexpression.Embryonic epithelial Pten deletion through Nkx2.1-cre leads to thyroid tumorigenesis in a strain-dependent manner.Inhibition of AMPK and Krebs cycle gene expression drives metabolic remodeling of Pten-deficient preneoplastic thyroid cells.Altered thyroid hormone profile in offspring after exposure to high estradiol environment during the first trimester of pregnancy: a cross-sectional studyModulation of sodium iodide symporter in thyroid cancerIn Vivo Role of INPP4B in Tumor and Metastasis Suppression through Regulation of PI3K-AKT Signaling at Endosomes.Mouse models of follicular and papillary thyroid cancer progression.Mouse models of thyroid cancer: A 2015 update.Roles of genetic variants in the PI3K and RAS/RAF pathways in susceptibility to endometrial cancer and clinical outcomes.MCM5 as a target of BET inhibitors in thyroid cancer cellsObesity and thyroid cancer: is leptin the (only) link?Activation of tumor cell proliferation by thyroid hormone in a mouse model of follicular thyroid carcinoma.Metformin blocks progression of obesity-activated thyroid cancer in a mouse model.Estrogen and thyroid cancer is a stem affair: A preliminary study.Mitochondrial regulation of cell cycle and proliferation.Modeling anaplastic thyroid carcinoma in the mouse.Expression of estrogen and progesterone receptors across human malignancies: new therapeutic opportunities.Modulatory role of 17β-estradiol in the tumor microenvironment of thyroid cancer.Thyrocyte-specific inactivation of p53 and Pten results in anaplastic thyroid carcinomas faithfully recapitulating human tumorsSGK1 Is a Critical Component of an AKT-Independent Pathway Essential for PI3K-Mediated Tumor Development and Maintenance.Thyroid-specific PPARγ deletion is benign in the mouse.Wnt Signaling in Thyroid Homeostasis and Carcinogenesis.
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P2860
Cross-talk between PI3K and estrogen in the mouse thyroid predisposes to the development of follicular carcinomas with a higher incidence in females.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Cross-talk between PI3K and es ...... a higher incidence in females.
@ast
Cross-talk between PI3K and es ...... a higher incidence in females.
@en
Cross-talk between PI3K and es ...... a higher incidence in females.
@nl
type
label
Cross-talk between PI3K and es ...... a higher incidence in females.
@ast
Cross-talk between PI3K and es ...... a higher incidence in females.
@en
Cross-talk between PI3K and es ...... a higher incidence in females.
@nl
prefLabel
Cross-talk between PI3K and es ...... a higher incidence in females.
@ast
Cross-talk between PI3K and es ...... a higher incidence in females.
@en
Cross-talk between PI3K and es ...... a higher incidence in females.
@nl
P2093
P2860
P356
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P1476
Cross-talk between PI3K and es ...... a higher incidence in females
@en
P2093
S Refetoff
V G Antico-Arciuch
P2860
P2888
P304
P356
10.1038/ONC.2010.308
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P577
2010-08-02T00:00:00Z
P5875
P6179
1013134172