Growth activation alone is not sufficient to cause metastatic thyroid cancer in a mouse model of follicular thyroid carcinoma - Wikidata - awgldk - TriplyDB

Growth activation alone is not sufficient to cause metastatic thyroid cancer in a mouse model of follicular thyroid carcinoma

Growth activation alone is not sufficient to cause metastatic thyroid cancer in a mouse model of follicular thyroid carcinoma

http://www.wikidata.org/entity/Q33775577

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Genomic profiling of genes contributing to metastasis in a mouse model of thyroid follicular carcinoma.Cross-talk between PI3K and estrogen in the mouse thyroid predisposes to the development of follicular carcinomas with a higher incidence in females.Molecular pathogenesis and mechanisms of thyroid cancer Synergistic signaling of KRAS and thyroid hormone receptor β mutants promotes undifferentiated thyroid cancer through MYC up-regulation.Thyroid hormone receptor beta (THRB) is a major target gene for microRNAs deregulated in papillary thyroid carcinoma (PTC)Oncogenic Actions of the Nuclear Receptor Corepressor (NCOR1) in a Mouse Model of Thyroid Cancer.Global expression profiling reveals gain-of-function oncogenic activity of a mutated thyroid hormone receptor in thyroid carcinogenesis.Extranuclear signaling of mutated thyroid hormone receptors in promoting metastatic spread in thyroid carcinogenesis Akt1 deficiency delays tumor progression, vascular invasion, and distant metastasis in a murine model of thyroid cancer Oncogenic mutations of thyroid hormone receptor β.SKI-606, an Src inhibitor, reduces tumor growth, invasion, and distant metastasis in a mouse model of thyroid cancer Modeling follicular thyroid cancer for future therapies Mouse models of follicular and papillary thyroid cancer progression.Thyroid hormone receptors and cancer.MicroRNAs in thyroid cancer.Thyroid hormone receptor β suppresses SV40-mediated tumorigenesis via novel nongenomic actions Role of TSH in the spontaneous development of asymmetrical thyroid carcinoma in mice with a targeted mutation in a single allele of the thyroid hormone-β receptor Diet-induced obesity increases tumor growth and promotes anaplastic change in thyroid cancer in a mouse model Activation of tumor cell proliferation by thyroid hormone in a mouse model of follicular thyroid carcinoma.Complex temporal changes in TGFβ oncogenic signaling drive thyroid carcinogenesis in a mouse model Obatoclax kills anaplastic thyroid cancer cells by inducing lysosome neutralization and necrosis Thyrotropin signaling confers more aggressive features with higher genomic instability on BRAF(V600E)-induced thyroid tumors in a mouse model Elevated HMGN4 expression potentiates thyroid tumorigenesis.Loss of tyrosine phosphorylation at Y406 abrogates the tumor suppressor functions of the thyroid hormone receptor β.Thyroid Stimulating Hormone Receptor RAP1GAP inhibits cytoskeletal remodeling and motility in thyroid cancer cells.Thyrocyte-specific inactivation of p53 and Pten results in anaplastic thyroid carcinomas faithfully recapitulating human tumors In vitro perturbations of targets in cancer hallmark processes predict rodent chemical carcinogenesis.Higher TSH level is a risk factor for differentiated thyroid cancer.BRAF-Oncogene-Induced Senescence and the Role of Thyroid-Stimulating Hormone Signaling in the Progression of Papillary Thyroid Carcinoma.Downregulated expression of TSHR is associated with distant metastasis in thyroid cancer.Interplay between TRα1 and Wnt signaling: A dangerous liaison

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P2860

Growth activation alone is not sufficient to cause metastatic thyroid cancer in a mouse model of follicular thyroid carcinoma

http://www.wikidata.org/entity/Q33775577

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Growth activation alone is not ...... f follicular thyroid carcinoma

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Growth activation alone is not ...... f follicular thyroid carcinoma

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type

label

Growth activation alone is not ...... f follicular thyroid carcinoma

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Growth activation alone is not ...... f follicular thyroid carcinoma

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Growth activation alone is not ...... f follicular thyroid carcinoma

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Growth activation alone is not ...... f follicular thyroid carcinoma

@en

P2093

Changxue Lu

http://www.w3.org/2001/XMLSchema#string

Hao Ying

http://www.w3.org/2001/XMLSchema#string

Li Zhao

http://www.w3.org/2001/XMLSchema#string

Mark C Willingham

http://www.w3.org/2001/XMLSchema#string

Sheue-Yann Cheng

http://www.w3.org/2001/XMLSchema#string

P2860

Evidence for in vivo phosphorylation of the Grb2 SH2-domain binding site on focal adhesion kinase by Src-family protein-tyrosine kinases

Characterization of seven novel mutations of the c-erbA beta gene in unrelated kindreds with generalized thyroid hormone resistance. Evidence for two "hot spot" regions of the ligand binding domain

Antitumor activity and pharmacology of a selective focal adhesion kinase inhibitor, PF-562,271

Focal adhesion kinase: in command and control of cell motility

p38 MAP-kinases pathway regulation, function and role in human diseases

An active Src kinase-beta-actin association is linked to actin dynamics at the periphery of colon cancer cells.

TSH signalling and cancer.

An unliganded thyroid hormone beta receptor activates the cyclin D1/cyclin-dependent kinase/retinoblastoma/E2F pathway and induces pituitary tumorigenesis

Endogenous thyrotropin and triiodothyronine concentrations in individuals with thyroid cancer

Higher serum thyroid stimulating hormone level in thyroid nodule patients is associated with greater risks of differentiated thyroid cancer and advanced tumor stage.

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1929-1939

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scholarly article

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10.1210/EN.2009-1017

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4

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151

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2010-02-04T00:00:00Z

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20133453

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2851190

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