Reciprocal interaction between macrophages and T cells stimulates IFN-γ and MCP-1 production in Ang II-induced cardiac inflammation and fibrosis
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Association study of MCP-1 promoter polymorphisms with the susceptibility and progression of sepsisActivation of NAD(P)H oxidase by tryptophan-derived 3-hydroxykynurenine accelerates endothelial apoptosis and dysfunction in vivo.Neutralization of TSLP inhibits airway remodeling in a murine model of allergic asthma induced by chronic exposure to house dust mite.Knockout of CD8 delays reendothelialization and accelerates neointima formation in injured arteries of mouse via TNF-α inhibiting the endothelial cells migration.Granulocyte-macrophage colony stimulatory factor enhances the pro-inflammatory response of interferon-γ-treated macrophages to Pseudomonas aeruginosa infection.Cardiac Fibrosis: The Fibroblast AwakensExpression of Transglutaminase in Foreskin of Children with Balanitis Xerotica ObliteransInhibition of platelet activation by clopidogrel prevents hypertension-induced cardiac inflammation and fibrosis.Could interferon-gamma be a therapeutic target for treating heart failure?Crosstalk between fibroblasts and inflammatory cells.Immunopathogenesis of Myocarditis: The Interplay Between Cardiac Fibroblast Cells, Dendritic Cells, Macrophages and CD4+ T Cells.CXCR4 signaling in macrophages contributes to periodontal mechanical hypersensitivity in Porphyromonas gingivalis-induced periodontitis in mice.The Processes and Mechanisms of Cardiac and Pulmonary Fibrosis.Role of Axl in early kidney inflammation and progression of salt-dependent hypertension.Th1 effector T cells selectively orchestrate cardiac fibrosis in nonischemic heart failure.Recruitment of macrophages from the spleen contributes to myocardial fibrosis and hypertension induced by angiotensin II.Properties and Immune Function of Cardiac Fibroblasts.Protective Roles of Interferon-γ in Cardiac Hypertrophy Induced by Sustained Pressure Overload.The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.Inhibition of Toll-like receptor 2 reduces cardiac fibrosis by attenuating macrophage-mediated inflammation.Early plasma monocyte chemoattractant protein 1 predicts the development of sepsis in trauma patients: A prospective observational study.
P2860
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P2860
Reciprocal interaction between macrophages and T cells stimulates IFN-γ and MCP-1 production in Ang II-induced cardiac inflammation and fibrosis
description
2012 nî lūn-bûn
@nan
2012 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Reciprocal interaction between ...... diac inflammation and fibrosis
@ast
Reciprocal interaction between ...... diac inflammation and fibrosis
@en
Reciprocal interaction between ...... diac inflammation and fibrosis
@nl
type
label
Reciprocal interaction between ...... diac inflammation and fibrosis
@ast
Reciprocal interaction between ...... diac inflammation and fibrosis
@en
Reciprocal interaction between ...... diac inflammation and fibrosis
@nl
prefLabel
Reciprocal interaction between ...... diac inflammation and fibrosis
@ast
Reciprocal interaction between ...... diac inflammation and fibrosis
@en
Reciprocal interaction between ...... diac inflammation and fibrosis
@nl
P2093
P2860
P1433
P1476
Reciprocal interaction between ...... diac inflammation and fibrosis
@en
P2093
Hong-jia Zhang
Ji-zhong Cheng
Li-xin Jia
Ya-lei Han
Yong-fen Qi
P2860
P304
P356
10.1371/JOURNAL.PONE.0035506
P407
P577
2012-05-02T00:00:00Z