The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
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KCa3.1 Channel Modulators as Potential Therapeutic Compounds for Glioblastoma.Oxidative stress promotes myocardial fibrosis by upregulating KCa3.1 channel expression in AGT-REN double transgenic hypertensive mice.Protective role of ACE2-Ang-(1-7)-Mas in myocardial fibrosis by downregulating KCa3.1 channel via ERK1/2 pathway.
P2860
The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
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The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
@en
The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
@nl
type
label
The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
@en
The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
@nl
prefLabel
The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
@en
The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
@nl
P2093
P2860
P1433
P1476
The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.
@en
P2093
Dang-Xia Zhou
Hui-Fang Wang
Li-Mei Zhao
Li-Ping Wang
Xiao-Zhen Ma
Xiu-Ling Deng
P2860
P2888
P304
P356
10.1007/S00424-015-1694-4
P577
2015-02-27T00:00:00Z