The relationship between asymptomatic organ damage, and serum soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) and Interleukin-17A (IL-17A) levels in non-diabetic hypertensive patients - Wikidata - awgldk - TriplyDB

The relationship between asymptomatic organ damage, and serum soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) and Interleukin-17A (IL-17A) levels in non-diabetic hypertensive patients

The relationship between asymptomatic organ damage, and serum soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) and Interleukin-17A (IL-17A) levels in non-diabetic hypertensive patients

http://www.wikidata.org/entity/Q34309016

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Role of T lymphocytes in hypertension.The role of soluble tumor necrosis factor like weak inducer of apoptosis and interleukin-17A in the etiopathogenesis of celiac disease: A cross-sectional study.The ratio of monocyte frequency to HDL cholesterol level as a predictor of asymptomatic organ damage in patients with primary hypertension.Are sTWEAK and IL-17A Levels in Inflammatory Bowel Disease Associated with Disease Activity and Etiopathogenesis?Relationship between oxidative stress parameters and asymptomatic organ damage in hypertensive patients without diabetes mellitus.

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P2860

The relationship between asymptomatic organ damage, and serum soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) and Interleukin-17A (IL-17A) levels in non-diabetic hypertensive patients

http://www.wikidata.org/entity/Q34309016

description

2014 nî lūn-bûn

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2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

The relationship between asymp ...... diabetic hypertensive patients

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The relationship between asymp ...... diabetic hypertensive patients

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The relationship between asymp ...... diabetic hypertensive patients

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The relationship between asymp ...... diabetic hypertensive patients

@en

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A Nurdan Barça

http://www.w3.org/2001/XMLSchema#string

Canan Topcuoglu

http://www.w3.org/2001/XMLSchema#string

Fatih Dede

http://www.w3.org/2001/XMLSchema#string

Fatma Akyel

http://www.w3.org/2001/XMLSchema#string

Ihsan Ates

http://www.w3.org/2001/XMLSchema#string

Nihal Ozkayar

http://www.w3.org/2001/XMLSchema#string

Serdar Akyel

http://www.w3.org/2001/XMLSchema#string

P2860

TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

Interleukin 17 in vascular inflammation.

TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.

Interleukin 17 promotes angiotensin II-induced hypertension and vascular dysfunction.

ABC of hypertension: The pathophysiology of hypertension

Full-length, membrane-anchored TWEAK can function as a juxtacrine signaling molecule and activate the NF-kappaB pathway

2013 ESH/ESC guidelines for the management of arterial hypertension: the Task Force for the Management of Arterial Hypertension of the European Society of Hypertension (ESH) and of the European Society of Cardiology (ESC).

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

Inflammation, endothelial dysfunction, and the risk of high blood pressure: epidemiologic and biological evidence.

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15

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