TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
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Structure of the extracellular domains of human and Xenopus Fn14: implications in the evolution of TWEAK and Fn14 interactionsThe TWEAK receptor Fn14 is a potential cell surface portal for targeted delivery of glioblastoma therapeuticsRoles of the canonical myomiRs miR-1, -133 and -206 in cell development and diseaseTWEAK and the progression of renal disease: clinical translationThe TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and diseaseSoluble Fn14 Is Detected and Elevated in Mouse and Human Kidney DiseaseTWEAK Regulates Muscle Functions in a Mouse Model of RNA Toxicity.TWEAK/Fn14, a pathway and novel therapeutic target in myotonic dystrophy.TNF-like weak inducer of apoptosis (TWEAK) promotes beta cell neogenesis from pancreatic ductal epithelium in adult mice.Regulation of fibroblast growth factor-inducible 14 (Fn14) expression levels via ligand-independent lysosomal degradationThe TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscleFibroblast growth factor inducible 14 as potential target in patients with alcoholic hepatitisTWEAK mediates inflammation in experimental atopic dermatitis and psoriasis.Inter- not intraindividual differences in sTWEAK levels predict functional deterioration and mortality in patients with dilated cardiomyopathy.DNA methyltransferase 3a and mitogen-activated protein kinase signaling regulate the expression of fibroblast growth factor-inducible 14 (Fn14) during denervation-induced skeletal muscle atrophy.Single muscle fiber gene expression with run taper.The relationship between asymptomatic organ damage, and serum soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) and Interleukin-17A (IL-17A) levels in non-diabetic hypertensive patientsPGC1α plays a critical role in TWEAK-induced cardiac dysfunction.TWEAK-independent Fn14 self-association and NF-κB activation is mediated by the C-terminal region of the Fn14 cytoplasmic domainDevelopment of human serine protease-based therapeutics targeting Fn14 and identification of Fn14 as a new target overexpressed in TNBC.Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.Structural basis and targeting of the interaction between fibroblast growth factor-inducible 14 and tumor necrosis factor-like weak inducer of apoptosisModerate exercise training attenuates the severity of experimental rodent colitis: the importance of crosstalk between adipose tissue and skeletal muscles.TWEAK-Fn14 pathway activation after exercise in human skeletal muscle: insights from two exercise modes and a time course investigation.Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.AR-regulated TWEAK-FN14 pathway promotes prostate cancer bone metastasisTWEAK/Fn14 Signaling Involvement in the Pathogenesis of Cutaneous Disease in the MRL/lpr Model of Spontaneous Lupus.Blocking TWEAK-Fn14 interaction inhibits hematopoietic stem cell transplantation-induced intestinal cell death and reduces GVHD.TWEAK and TRAF6 regulate skeletal muscle atrophy.Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Promotes Hepatic Stellate Cells Migration via Canonical NF-κB/MMP9 Pathway.Inhibition of the TWEAK/Fn14 pathway attenuates renal disease in nephrotoxic serum nephritisTWEAK/Fn14 pathway modulates properties of a human microvascular endothelial cell model of blood brain barrier.Soluble TNF-Like Weak Inducer of Apoptosis as a New Marker in Preeclampsia: A Pilot Clinical Study.Deletion of Fn14 receptor protects from right heart fibrosis and dysfunctionFN14 and GRP94 expression are prognostic/predictive biomarkers of brain metastasis outcome that open up new therapeutic strategies.Clinical targeting of the TNF and TNFR superfamiliesThe HER2- and heregulin β1 (HRG)-inducible TNFR superfamily member Fn14 promotes HRG-driven breast cancer cell migration, invasion, and MMP9 expression.Fn14-Fc suppresses germinal center formation and pathogenic B cells in a lupus mouse model via inhibition of the TWEAK/Fn14 Pathway.Neuropsychiatric disease in murine lupus is dependent on the TWEAK/Fn14 pathway.TWEAK/Fn14 Axis: A Promising Target for the Treatment of Cardiovascular Diseases.
P2860
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P2860
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
description
2011 nî lūn-bûn
@nan
2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
@ast
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
@en
type
label
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
@ast
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
@en
prefLabel
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
@ast
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
@en
P2093
P1476
TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.
@en
P2093
Jennifer S Michaelson
Linda C Burkly
Timothy S Zheng
P304
P356
10.1111/J.1600-065X.2011.01054.X
P577
2011-11-01T00:00:00Z