Role of platelets in smooth muscle cell proliferation and migration after vascular injury in rat carotid artery.
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The clopidogrel after surgery for coronary artery disease (CASCADE) randomized controlled trial: clopidogrel and aspirin versus aspirin alone after coronary bypass surgery [NCT00228423].Immune-mediated vascular injury and dysfunction in transplant arteriosclerosisPlatelet CD62 expression and PDGFAB secretion in patients undergoing PTCA and treatment with abciximabAngiotensin-converting enzyme inhibitor versus angiotensin II, AT1 receptor antagonist. Effects on smooth muscle cell migration and proliferation after balloon catheter injurySlingshot isoform-specific regulation of cofilin-mediated vascular smooth muscle cell migration and neointima formationH19, a developmentally regulated gene, is reexpressed in rat vascular smooth muscle cells after injury.Long-term biological response of injured rat carotid artery seeded with smooth muscle cells expressing retrovirally introduced human genes.Intimal hyperplasia after vascular injury is inhibited by antisense cdk 2 kinase oligonucleotides.Production of transforming growth factor beta 1 during repair of arterial injury.Inflammation as a key event in the development of neointima following vascular balloon injury.Periadventitial atRA citrate-based polyester membranes reduce neointimal hyperplasia and restenosis after carotid injury in rats.Proliferation of smooth muscle cells after vascular injury is inhibited by an antibody against basic fibroblast growth factorExpression of Q227L-Galpha(s) inhibits intimal vessel wall hyperplasia after balloon injury.Inhibition of thrombosis and intimal thickening by in situ photopolymerization of thin hydrogel barriersPlatelet-derived growth factor promotes smooth muscle migration and intimal thickening in a rat model of balloon angioplasty.Nitric oxide affects UbcH10 levels differently in type 1 and type 2 diabetic rats.Regulation of insulin-like growth factor-I and its receptor in rat aorta after balloon denudation. Evidence for local bioactivity.Neointima formation after acute vascular injury. Role of counteradhesive extracellular matrix proteinsArteriosclerosis. Impairment of cellular interactions in the arterial wall.Intracellular signaling pathways required for rat vascular smooth muscle cell migration. Interactions between basic fibroblast growth factor and platelet-derived growth factorAdenovirus-mediated over-expression of the cyclin/cyclin-dependent kinase inhibitor, p21 inhibits vascular smooth muscle cell proliferation and neointima formation in the rat carotid artery model of balloon angioplasty.Estradiol attenuates directed migration of vascular smooth muscle cells in vitroChimera analysis supports a predominant role of PDGFRbeta in promoting smooth-muscle cell chemotaxis after arterial injury.Expression of smooth muscle cell phenotype by rat mesangial cells in immune complex nephritis. Alpha-smooth muscle actin is a marker of mesangial cell proliferation.Intimal smooth muscle cell proliferation after balloon catheter injury. The role of basic fibroblast growth factor.Localization of Fos and Jun proteins in rat aortic smooth muscle cells after vascular injury.Potentiation of the hyporeactivity induced by in vivo endothelial injury in the rat carotid artery by chronic treatment with fish oil.MFG-E8 activates proliferation of vascular smooth muscle cells via integrin signaling.Continued platelet interaction with de-endothelialized aortae associated with slower re-endothelialization and more extensive intimal hyperplasia in spontaneously diabetic BB Wistar rats.Intratumoural administration of dendritic cells: hostile environment and help by gene therapy.PDGF ligand and receptor gene expression during repair of arterial injury.Preclinical restenosis models: challenges and successes.Nitric oxide and nanotechnology: a novel approach to inhibit neointimal hyperplasia.Gene therapy via inducible nitric oxide synthase: a tool for the treatment of a diverse range of pathological conditions.CTP synthase 1, a smooth muscle-sensitive therapeutic target for effective vascular repairFrequency and severity of cyclic flow alternations and platelet aggregation predict the severity of neointimal proliferation following experimental coronary stenosis and endothelial injury.Identification of a 38-kDa heparin-binding glycoprotein (gp38k) in differentiating vascular smooth muscle cells as a member of a group of proteins associated with tissue remodeling.Enhanced biocompatibility of CD47-functionalized vascular stents.Molecular biology and gene transfer in atherosclerosis in the stenting era.Growth factors and arterial smooth muscle cell proliferation.
P2860
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P2860
Role of platelets in smooth muscle cell proliferation and migration after vascular injury in rat carotid artery.
description
1989 nî lūn-bûn
@nan
1989 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Role of platelets in smooth mu ...... injury in rat carotid artery.
@ast
Role of platelets in smooth mu ...... injury in rat carotid artery.
@en
Role of platelets in smooth mu ...... injury in rat carotid artery.
@nl
type
label
Role of platelets in smooth mu ...... injury in rat carotid artery.
@ast
Role of platelets in smooth mu ...... injury in rat carotid artery.
@en
Role of platelets in smooth mu ...... injury in rat carotid artery.
@nl
prefLabel
Role of platelets in smooth mu ...... injury in rat carotid artery.
@ast
Role of platelets in smooth mu ...... injury in rat carotid artery.
@en
Role of platelets in smooth mu ...... injury in rat carotid artery.
@nl
P2093
P2860
P356
P1476
Role of platelets in smooth mu ...... injury in rat carotid artery.
@en
P2093
A W Clowes
J Fingerle
M W Majesky
P2860
P304
P356
10.1073/PNAS.86.21.8412
P407
P577
1989-11-01T00:00:00Z