Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
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Huntington's disease induced cardiac amyloidosis is reversed by modulating protein folding and oxidative stress pathways in the Drosophila heartReal-time imaging of Huntingtin aggregates diverting target search and gene transcriptionStructural transformation of bovine serum albumin induced by dimethyl sulfoxide and probed by fluorescence correlation spectroscopy and additional methods.Atomic structure of a toxic, oligomeric segment of SOD1 linked to amyotrophic lateral sclerosis (ALS).
P2860
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
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2012 nî lūn-bûn
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2012 թուականի Յունիսին հրատարակուած գիտական յօդուած
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2012 թվականի հունիսին հրատարակված գիտական հոդված
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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name
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@ast
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@en
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@nl
type
label
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@ast
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@en
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@nl
prefLabel
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@ast
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@en
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@nl
P2093
P2860
P1433
P1476
Huntingtin fragments and SOD1 mutants form soluble oligomers in the cell
@en
P2093
Evan Eisenberg
J Paul Taylor
Lois E Greene
Mark Norton
Xiaohong Zhao
Yang-Nim Park
P2860
P304
P356
10.1371/JOURNAL.PONE.0040329
P407
P577
2012-06-29T00:00:00Z