The p53 transcription factor modulates microglia behavior through microRNA-dependent regulation of c-Maf. - Wikidata - awgldk - TriplyDB

The p53 transcription factor modulates microglia behavior through microRNA-dependent regulation of c-Maf.

The p53 transcription factor modulates microglia behavior through microRNA-dependent regulation of c-Maf.

http://www.wikidata.org/entity/Q34331564

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MicroRNAs: Key Regulators in the Central Nervous System and Their Implication in Neurological Diseases MicroRNAs Regulate Mitochondrial Function in Cerebral Ischemia-Reperfusion Injury Recombinant adeno-associated viral (rAAV) vectors mediate efficient gene transduction in cultured neonatal and adult microglia The p53 Transcriptional Network Influences Microglia Behavior and Neuroinflammation miR-155 Is Essential for Inflammation-Induced Hippocampal Neurogenic Dysfunction Ischemic Preconditioning in White Matter: Magnitude and Mechanism.miR-2861 acts as a tumor suppressor via targeting EGFR/AKT2/CCND1 pathway in cervical cancer induced by human papillomavirus virus 16 E6 MicroRNAs mediating CNS inflammation: Small regulators with powerful potential.Roles of microglia in brain development, tissue maintenance and repair.Transcriptional control of microglia phenotypes in health and disease.Ischemia/Reperfusion Induces Interferon-Stimulated Gene Expression in Microglia.microRNAs in Cerebrovascular Disease.The Emerging Role of Epigenetics in Cerebral Ischemia.Fate of microglia during HIV-1 infection: From activation to senescence?Neuroimmune Response in Ischemic Preconditioning.Control of the Inflammatory Macrophage Transcriptional Signature by miR-155.miR-146a-Traf6 regulatory axis controls autoimmunity and myelopoiesis, but is dispensable for hematopoietic stem cell homeostasis and tumor suppression.Detection and quantification of microRNA in cerebral microdialysate Bridging Autism Spectrum Disorders and Schizophrenia through inflammation and biomarkers - pre-clinical and clinical investigations.Exportin 1 (XPO1) inhibition leads to restoration of tumor suppressor miR-145 and consequent suppression of pancreatic cancer cell proliferation and migration.Correction to: Neuroimmune Response in Ischemic Preconditioning.Neuroprotective effects of pifithrin-α against traumatic brain injury in the striatum through suppression of neuroinflammation, oxidative stress, autophagy, and apoptosis.

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P2860

The p53 transcription factor modulates microglia behavior through microRNA-dependent regulation of c-Maf.

http://www.wikidata.org/entity/Q34331564

description

2013 nî lūn-bûn

@nan

2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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The p53 transcription factor m ...... dependent regulation of c-Maf.

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Journal of Immunology

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The p53 transcription factor m ...... dependent regulation of c-Maf.

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Aurelio Silvestroni

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Bryce Sopher

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Gwenn A Garden

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Jonathan Weinstein

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Nicole K Nesser

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Simon Ammanuel

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Stephanie Hopkins

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Thomas Möller

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Wei Su

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P2860

Induction of EMT by twist proteins as a collateral effect of tumor-promoting inactivation of premature senescence

Glial cells as intrinsic components of non-cell-autonomous neurodegenerative disease

The protooncogene c-Maf is an essential transcription factor for IL-10 gene expression in macrophages

p53 represses c-Myc through induction of the tumor suppressor miR-145

MicroRNA-145 suppresses cell invasion and metastasis by directly targeting mucin 1

A CREB-C/EBPbeta cascade induces M2 macrophage-specific gene expression and promotes muscle injury repair

Requirement of bic/microRNA-155 for normal immune function

MicroRNAs are universal regulators of differentiation, activation, and polarization of microglia and macrophages in normal and diseased CNS

Conserved seed pairing, often flanked by adenosines, indicates that thousands of human genes are microRNA targets

Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

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10.4049/JIMMUNOL.1301397

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