Ischemic Preconditioning in White Matter: Magnitude and Mechanism. - Wikidata - awgldk - TriplyDB

Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

http://www.wikidata.org/entity/Q36319834

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Ischemia/Reperfusion Induces Interferon-Stimulated Gene Expression in Microglia.Microglial Interferon Signaling and White Matter.Neuroimmune Response in Ischemic Preconditioning.White matter damage after traumatic brain injury: A role for damage associated molecular patterns.Type-1 interferons contribute to the neuroinflammatory response and disease progression of the MPTP mouse model of Parkinson's disease.Dimethyl fumarate improves white matter function following severe hypoperfusion: Involvement of microglia/macrophages and inflammatory mediators.Loss of IRF2BP2 in Microglia Increases Inflammation and Functional Deficits after Focal Ischemic Brain Injury.NogoA Neutralization Promotes Axonal Restoration After White Matter Injury In Subcortical Stroke.Stroke: Basic and Clinical.Microglia activation mediated by toll-like receptor-4 impairs brain white matter tracts in rats.Correction to: Neuroimmune Response in Ischemic Preconditioning.

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Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

http://www.wikidata.org/entity/Q36319834

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

@zh

2015年學術文章

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name

Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

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Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

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Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

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Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

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Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

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Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

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JNEUROSCI.2544-15.2015

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Journal of Neuroscience

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Ischemic Preconditioning in White Matter: Magnitude and Mechanism.

@en

P2093

Jamie R Colman

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Thu Le

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P2860

Functional role of type I and type II interferons in antiviral defense

Models that matter: white matter stroke models

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

Type I interferon is selectively required by dendritic cells for immune rejection of tumors

Flip and flop: a cell-specific functional switch in glutamate-operated channels of the CNS

A family of AMPA-selective glutamate receptors

Physiology of microglia

Recommendations for standards regarding preclinical neuroprotective and restorative drug development.

Toll-like receptor 4 contributes to retinal ischemia/reperfusion injury.

The p53 transcription factor modulates microglia behavior through microRNA-dependent regulation of c-Maf.

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15599-15611

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scholarly article

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10.1523/JNEUROSCI.2544-15.2015

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47

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35

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Margaret A Hamner

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2015-11-01T00:00:00Z

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26609155

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4659824

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