Porphyromonas gingivalis lipopolysaccharide: an unusual pattern recognition receptor ligand for the innate host defense system.
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Hydrogen Sulfide, Oxidative Stress and Periodontal Diseases: A Concise ReviewPeriodontal disease immunology: 'double indemnity' in protecting the hostDeleted in malignant brain tumors-1 protein (DMBT1): a pattern recognition receptor with multiple binding sitesExpression of a Porphyromonas gingivalis lipid A palmitylacyltransferase in Escherichia coli yields a chimeric lipid A with altered ability to stimulate interleukin-8 secretion.Acyl chain specificity of the acyltransferases LpxA and LpxD and substrate availability contribute to lipid A fatty acid heterogeneity in Porphyromonas gingivalis.Manipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis.Mouse paneth cell secretory responses to cell surface glycolipids of virulent and attenuated pathogenic bacteria.Porphyromonas gingivalis lipopolysaccharide is both agonist and antagonist for p38 mitogen-activated protein kinase activation.Porphyromonas gingivalis peptidoglycans induce excessive activation of the innate immune system in silkworm larvae.Porphyromonas gingivalis exacerbates ligature-induced, RANKL-dependent alveolar bone resorption via differential regulation of Toll-like receptor 2 (TLR2) and TLR4.The mucosal immune system: from specialized immune defense to inflammation and allergy.Inhibitory effects of Ginkgo biloba extract on inflammatory mediator production by Porphyromonas gingivalis lipopolysaccharide in murine macrophages via Nrf-2 mediated heme oxygenase-1 signaling pathways.Tetra- and penta-acylated lipid A structures of Porphyromonas gingivalis LPS differentially activate TLR4-mediated NF-κB signal transduction cascade and immuno-inflammatory response in human gingival fibroblastsRole of periodontal pathogenic bacteria in RANKL-mediated bone destruction in periodontal disease.Receptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.MAPK usage in periodontal disease progression.Modulation of the innate immune response within the periodontium.Phosphate groups of lipid A are essential for Salmonella enterica serovar Typhimurium virulence and affect innate and adaptive immunityLipopolysaccharides-Induced Suppression of Innate-Like B Cell Apoptosis Is Enhanced by CpG Oligodeoxynucleotide and Requires Toll-Like Receptors 2 and 4Effect of genetic variability on the inflammatory response to periodontal infection.Host responses in maintaining periodontal health and determining periodontal disease.Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.Dendritic cells at the oral mucosal interface.Toll-like receptors and their role in periodontal health and disease.Novel host response therapeutic approaches to treat periodontal diseases.Human variability in innate immunity.Salivary agglutinin/glycoprotein-340/DMBT1: a single molecule with variable composition and with different functions in infection, inflammation and cancer.Serine lipids of Porphyromonas gingivalis are human and mouse Toll-like receptor 2 ligandsRapid myeloid cell transcriptional and proteomic responses to periodontopathogenic Porphyromonas gingivalisPolybacterial challenge effects on cytokine/chemokine production by macrophages and dendritic cells.Periodontitis as a risk factor of atherosclerosis.Porphyromonas gingivalis Fimbria-Induced Expression of Inflammatory Cytokines and Cyclooxygenase-2 in Mouse Macrophages and Its Inhibition by the Bioactive Compounds Fibronectin and Melatonin.Porphyromonas gingivalis: major periodontopathic pathogen overview.Alteration of gene expression profiles of peripheral mononuclear blood cells by tobacco smoke: implications for periodontal diseases.Diverse functions of defensins and other antimicrobial peptides in periodontal tissues.The role of natural killer cells in periodontitis.Salmeterol, a Long-Acting β2-Adrenergic Receptor Agonist, Inhibits Macrophage Activation by Lipopolysaccharide From Porphyromonas gingivalis.Anti-inflammatory effect of 635 nm irradiations on in vitro direct/indirect irradiation model.Phosphoglycerol dihydroceramide, a distinctive ceramide produced by Porphyromonas gingivalis, promotes RANKL-induced osteoclastogenesis by acting on non-muscle myosin II-A (Myh9), an osteoclast cell fusion regulatory factor.Plasminogen activator inhibitor type 1 expression induced by lipopolysaccharide of Porphyromonas gingivalis in human gingival fibroblast.
P2860
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P2860
Porphyromonas gingivalis lipopolysaccharide: an unusual pattern recognition receptor ligand for the innate host defense system.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Porphyromonas gingivalis lipop ...... he innate host defense system.
@ast
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
Porphyromonas gingivalis lipop ...... he innate host defense system.
@nl
type
label
Porphyromonas gingivalis lipop ...... he innate host defense system.
@ast
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
Porphyromonas gingivalis lipop ...... he innate host defense system.
@nl
prefLabel
Porphyromonas gingivalis lipop ...... he innate host defense system.
@ast
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
Porphyromonas gingivalis lipop ...... he innate host defense system.
@nl
P2860
P1476
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
P2093
Bainbridge BW
Darveau RP
P2860
P304
P356
10.1080/000163501750266710
P577
2001-06-01T00:00:00Z