Inhibition of AP-1 binding and transcription by gold and selenium involving conserved cysteine residues in Jun and Fos.
about
Human thioredoxin reductase from HeLa cells: selective alkylation of selenocysteine in the protein inhibits enzyme activity and reduction with NADPH influences affinity to heparinGold compounds as therapeutic agents for human diseasesSelenite inhibits the c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) through a thiol redox mechanismGold is a T cell polyclonal activator in BN and LEW rats but favors IL-4 expression only in autoimmune prone BN ratsRedox regulation of DNA repair: implications for human health and cancer therapeutic development.Inhibition of transcription factors by anti-inflammatory and anti-rheumatic drugs: can variability in response be overcome?APE1/Ref-1 role in redox signaling: translational applications of targeting the redox function of the DNA repair/redox protein APE1/Ref-1.Effects of high doses of selenium, as sodium selenite, in septic shock: a placebo-controlled, randomized, double-blind, phase II study.Inhibition of NF-kappaB DNA binding and nitric oxide induction in human T cells and lung adenocarcinoma cells by selenite treatmentAuranofin, as an anti-rheumatic gold compound, suppresses LPS-induced homodimerization of TLR4.Zinc finger proteins as templates for metal ion exchange and ligand reactivity. Chemical and biological consequences.Thiol alkylation inhibits the mitogenic effects of platelet-derived growth factor and renders it proapoptotic via activation of STATs and p53 and induction of expression of caspase1 and p21(waf1/cip1).Antagonistic effects of selenium and lipid peroxides on growth control in early hepatocellular carcinoma.Involvement of superoxide dismutases in the response of Escherichia coli to selenium oxides.Protection of mice from allergen-induced asthma by selenite: prevention of eosinophil infiltration by inhibition of NF-kappa B activation.Induction of cellular antioxidative stress genes through heterodimeric transcription factor Nrf2/small Maf by antirheumatic gold(I) compounds.The use of biologics in the treatment of rheumatoid arthritis (RA)--the good news and the bad news.Induction of mitochondrial permeability transition by auranofin, a gold(I)-phosphine derivativeRedox regulation of GA-binding protein-alpha DNA binding activity.Catalytic selenols couple the redox cycles of metallothionein and glutathione.Can skin cancer in humans be prevented by alleviation of oxidative stress?Identification of genes affecting selenite toxicity and resistance in Saccharomyces cerevisiae.Gold-Based Therapeutic Agents: A New PerspectiveNutrition, epigenetics and ageing
P2860
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P2860
Inhibition of AP-1 binding and transcription by gold and selenium involving conserved cysteine residues in Jun and Fos.
description
1995 nî lūn-bûn
@nan
1995 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@ast
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@en
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@nl
type
label
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@ast
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@en
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@nl
prefLabel
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@ast
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@en
Inhibition of AP-1 binding and ...... teine residues in Jun and Fos.
@nl
P2093
P2860
P356
P1476
Inhibition of AP-1 binding and ...... steine residues in Jun and Fos
@en
P2093
M L Handel
R L Sutherland
P2860
P304
P356
10.1073/PNAS.92.10.4497
P407
P50
P577
1995-05-01T00:00:00Z