STAT5 is critical to maintain effector CD8+ T cell responses. - Wikidata - awgldk - TriplyDB

STAT5 is critical to maintain effector CD8+ T cell responses.

STAT5 is critical to maintain effector CD8+ T cell responses.

http://www.wikidata.org/entity/Q34346687

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Regulating the immune system via IL-15 transpresentation The effects of TLR activation on T-cell development and differentiation Murine autoimmune cholangitis requires two hits: cytotoxic KLRG1(+) CD8 effector cells and defective T regulatory cells.Regulation of tissue-dependent differences in CD8+ T cell apoptosis during viral infection.Effects of 1,25-(OH)(2)D (3) on the expressions of vitamin D receptor, STAT5 and cytoskeletal rearrangement in human monocytes incubated with sera from type 2 diabetes patients and diabetic nephropathy patients with uremia.Cytokines and the inception of CD8 T cell responses.Intersection of mTOR and STAT signaling in immunity Regulatory T cells resist virus infection-induced apoptosis Efficacy of JAK/STAT pathway inhibition in murine xenograft models of early T-cell precursor (ETP) acute lymphoblastic leukemia.STAT5 is a potent negative regulator of TFH cell differentiation.Signal integration by Akt regulates CD8 T cell effector and memory differentiation.Generalized Liver- and Blood-Derived CD8+ T-Cell Impairment in Response to Cytokines in Chronic Hepatitis C Virus Infection Stable inhibitory activity of regulatory T cells requires the transcription factor Helios Differential expression of Ly6C and T-bet distinguish effector and memory Th1 CD4(+) cell properties during viral infection.NK Cell Proliferation Induced by IL-15 Transpresentation Is Negatively Regulated by Inhibitory Receptors.JAK and STAT signaling molecules in immunoregulation and immune-mediated disease.Disparate roles for STAT5 in primary and secondary CTL responses.IL-15 signaling promotes adoptive effector T-cell survival and memory formation in irradiation-induced lymphopenia IL-7 signaling and CD127 receptor regulation in the control of T cell homeostasis Protecting and rescuing the effectors: roles of differentiation and survival in the control of memory T cell development.Role of PI3K/Akt signaling in memory CD8 T cell differentiation.Cooperative therapeutic anti-tumor effect of IL-15 agonist ALT-803 and co-targeting soluble NKG2D ligand sMIC STAT3 promotes CD1d-mediated lipid antigen presentation by regulating a critical gene in glycosphingolipid biosynthesis.IL-21 and T Cell Differentiation: Consider the Context.Dying to protect: cell death and the control of T-cell homeostasis.TCF1+ hepatitis C virus-specific CD8+ T cells are maintained after cessation of chronic antigen stimulation.IL-4 and IL-15 promotion of virtual memory CD8+ T cells is determined by genetic background.When Aging Reaches CD4+ T-Cells: Phenotypic and Functional Changes.Peripheral human CD4+CD8+ T lymphocytes exhibit a memory phenotype and enhanced responses to IL-2, IL-7 and IL-15.Temporal Expression of Bim Limits the Development of Agonist-Selected Thymocytes and Skews Their TCRβ Repertoire.Mcl-1 antagonizes Bax/Bak to promote effector CD4(+) and CD8(+) T-cell responses.Longitudinal analysis of whole blood transcriptomes to explore molecular signatures associated with acute renal allograft rejection.Bim controls IL-15 availability and limits engagement of multiple BH3-only proteins.An interleukin-21-interleukin-10-STAT3 pathway is critical for functional maturation of memory CD8+ T cells The MicroRNA miR-191 Supports T Cell Survival Following Common γ Chain Signaling Bcl-2 allows effector and memory CD8+ T cells to tolerate higher expression of Bim.IL-15 preferentially enhances functional properties and antigen-specific responses of CD4+CD28(null) compared to CD4+CD28+ T cells.STAT5BN642H is a driver mutation for T cell neoplasia.Pivotal role of NAMPT in the switch of melanoma cells toward an invasive and drug-resistant phenotype.Stat5 gene dosage in T cells modulates CD8+T-cell homeostasis and attenuates contact hypersensitivity response in mice

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P2860

STAT5 is critical to maintain effector CD8+ T cell responses.

http://www.wikidata.org/entity/Q34346687

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

STAT5 is critical to maintain effector CD8+ T cell responses.

@ast

STAT5 is critical to maintain effector CD8+ T cell responses.

@en

STAT5 is critical to maintain effector CD8+ T cell responses.

@nl

type

label

STAT5 is critical to maintain effector CD8+ T cell responses.

@ast

STAT5 is critical to maintain effector CD8+ T cell responses.

@en

STAT5 is critical to maintain effector CD8+ T cell responses.

@nl

prefLabel

STAT5 is critical to maintain effector CD8+ T cell responses.

@ast

STAT5 is critical to maintain effector CD8+ T cell responses.

@en

STAT5 is critical to maintain effector CD8+ T cell responses.

@nl

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P356

JIMMUNOL.1000842

P1433

Journal of Immunology

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STAT5 is critical to maintain effector CD8+ T cell responses

@en

P2093

Allyson Sholl

http://www.w3.org/2001/XMLSchema#string

Fred D Finkelman

http://www.w3.org/2001/XMLSchema#string

Pulak Tripathi

http://www.w3.org/2001/XMLSchema#string

Sara Wojciechowski

http://www.w3.org/2001/XMLSchema#string

Sema Kurtulus

http://www.w3.org/2001/XMLSchema#string

Suzanne C Morris

http://www.w3.org/2001/XMLSchema#string

P2860

Stat5 is required for IL-2-induced cell cycle progression of peripheral T cells

The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

Antiviral CD4+ memory T cells are IL-15 dependent

NF-kappaB-mediated up-regulation of Bcl-x and Bfl-1/A1 is required for CD40 survival signaling in B lymphocytes

Impaired B and T cell antigen receptor signaling in p110delta PI 3-kinase mutant mice

Stat5b is essential for natural killer cell-mediated proliferation and cytolytic activity

Bim/Bcl-2 balance is critical for maintaining naive and memory T cell homeostasis

STAT5 requires the N-domain for suppression of miR15/16, induction of bcl-2, and survival signaling in myeloproliferative disease

Role of a STAT binding site in the regulation of the human perforin promoter.

Apoptosis and the homeostatic control of immune responses.

P304

2116-2124

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scholarly article

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10.4049/JIMMUNOL.1000842

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4

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185

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P50

H. Leighton Grimes

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2010-07-19T00:00:00Z

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45275325

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20644163

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2991082

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