Deacetylation of the herpes simplex virus type 1 latency-associated transcript (LAT) enhancer and a decrease in LAT abundance precede an increase in ICP0 transcriptional permissiveness at early times postexplant.
about
Herpes simplex virus type 1 latency and reactivation: an updateA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyRole of chromatin during herpesvirus infections.De novo synthesis of VP16 coordinates the exit from HSV latency in vivo.Host microRNA regulation of human cytomegalovirus immediate early protein translation promotes viral latency.Transcriptional coactivator HCF-1 couples the histone chaperone Asf1b to HSV-1 DNA replication componentsControl of alpha-herpesvirus IE gene expression by HCF-1 coupled chromatin modification activities.Epigenetic regulation of latent HSV-1 gene expression.Changes to euchromatin on LAT and ICP4 following reactivation are more prevalent in an efficiently reactivating strain of HSV-1.Disruption of HDAC/CoREST/REST repressor by dnREST reduces genome silencing and increases virulence of herpes simplex virusGenome-wide histone acetylation profiling of Herpesvirus saimiri in human T cells upon induction with a histone deacetylase inhibitor.Tissue-specific splicing of the herpes simplex virus type 1 latency-associated transcript (LAT) intron in LAT transgenic miceThe checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexHSV-1 gene expression from reactivated ganglia is disordered and concurrent with suppression of latency-associated transcript and miRNAsDynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies.Sodium butyrate: a chemical inducer of in vivo reactivation of herpes simplex virus type 1 in the ocular mouse model.The herpes simplex virus type 1 BgKL variant, unlike the BgOL variant, shows a higher association with orolabial infection than with infections at other sites, supporting the variant-dispersion-replacement hypothesis.In vivo changes in the patterns of chromatin structure associated with the latent herpes simplex virus type 1 genome in mouse trigeminal ganglia can be detected at early times after butyrate treatment.CTCF occupation of the herpes simplex virus 1 genome is disrupted at early times postreactivation in a transcription-dependent manner.A novel selective LSD1/KDM1A inhibitor epigenetically blocks herpes simplex virus lytic replication and reactivation from latency.Targeting the JMJD2 histone demethylases to epigenetically control herpesvirus infection and reactivation from latencyAssociation of the cellular coactivator HCF-1 with the Golgi apparatus in sensory neurons.Establishment of murine cytomegalovirus latency in vivo is associated with changes in histone modifications and recruitment of transcriptional repressors to the major immediate-early promoter.The dynamics of HCF-1 modulation of herpes simplex virus chromatin during initiation of infection.Chromatin control of herpes simplex virus lytic and latent infection.Role of polycomb proteins in regulating HSV-1 latency.Broadening the repertoire of functional herpes simplex virus type 1-specific CD8+ T cells reduces viral reactivation from latency in sensory gangliaOcular HSV-1 latency, reactivation and recurrent disease.Recruitment of the transcriptional coactivator HCF-1 to viral immediate-early promoters during initiation of reactivation from latency of herpes simplex virus type 1.Inhibition of the histone demethylase LSD1 blocks alpha-herpesvirus lytic replication and reactivation from latency.Epigenetic repression of herpes simplex virus infection by the nucleosome remodeler CHD3Role of viral chromatin structure in the regulation of herpes simplex virus 1 gene expression and replication.How to control an infectious bead string: nucleosome-based regulation and targeting of herpesvirus chromatin.Histone modifications in herpesvirus infections.Chromatin dynamics during lytic infection with herpes simplex virus 1.Host-virus interactions: from the perspectives of epigenetics.Histone modifications associated with herpes simplex virus type 1 genomes during quiescence and following ICP0-mediated de-repression.Roles of Non-coding RNAs During Herpesvirus Infection.Early nucleosome deposition on, and replication of, HSV DNA requires cell factor PCNA.
P2860
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P2860
Deacetylation of the herpes simplex virus type 1 latency-associated transcript (LAT) enhancer and a decrease in LAT abundance precede an increase in ICP0 transcriptional permissiveness at early times postexplant.
description
2006 nî lūn-bûn
@nan
2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Deacetylation of the herpes si ...... ss at early times postexplant.
@ast
Deacetylation of the herpes si ...... ss at early times postexplant.
@en
Deacetylation of the herpes simplex virus type 1 latency-associated transcript
@nl
type
label
Deacetylation of the herpes si ...... ss at early times postexplant.
@ast
Deacetylation of the herpes si ...... ss at early times postexplant.
@en
Deacetylation of the herpes simplex virus type 1 latency-associated transcript
@nl
prefLabel
Deacetylation of the herpes si ...... ss at early times postexplant.
@ast
Deacetylation of the herpes si ...... ss at early times postexplant.
@en
Deacetylation of the herpes simplex virus type 1 latency-associated transcript
@nl
P2093
P2860
P1433
P1476
Deacetylation of the herpes si ...... ss at early times postexplant.
@en
P2093
Antonio L Amelio
David C Bloom
Jerome E O'neil
Nicole J Kubat
Nicole V Giordani
P2860
P304
P356
10.1128/JVI.80.4.2063-2068.2006
P407
P577
2006-02-01T00:00:00Z