Triggering the succinate receptor GPR91 enhances pressure overload-induced right ventricular hypertrophy. - Wikidata - awgldk - TriplyDB

Triggering the succinate receptor GPR91 enhances pressure overload-induced right ventricular hypertrophy.

Triggering the succinate receptor GPR91 enhances pressure overload-induced right ventricular hypertrophy.

http://www.wikidata.org/entity/Q34371092

about

The Roles of Mitochondrial Damage-Associated Molecular Patterns in Diseases Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation.Oncometabolite succinate promotes angiogenesis by upregulating VEGF expression through GPR91-mediated STAT3 and ERK activation.Metabolism in cardiomyopathy: every substrate matters.GPR91 deficiency exacerbates allergic contact dermatitis while reducing arthritic disease in mice.Glutamine metabolism drives succinate accumulation in plasma and the lung during hemorrhagic shock.Mitochondrial Damage-Associated Molecular Patterns: From Inflammatory Signaling to Human Diseases.

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P2860

Triggering the succinate receptor GPR91 enhances pressure overload-induced right ventricular hypertrophy.

http://www.wikidata.org/entity/Q34371092

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Triggering the succinate recep ...... right ventricular hypertrophy.

@ast

Triggering the succinate recep ...... right ventricular hypertrophy.

@en

Triggering the succinate recep ...... right ventricular hypertrophy.

@nl

type

label

Triggering the succinate recep ...... right ventricular hypertrophy.

@ast

Triggering the succinate recep ...... right ventricular hypertrophy.

@en

Triggering the succinate recep ...... right ventricular hypertrophy.

@nl

prefLabel

Triggering the succinate recep ...... right ventricular hypertrophy.

@ast

Triggering the succinate recep ...... right ventricular hypertrophy.

@en

Triggering the succinate recep ...... right ventricular hypertrophy.

@nl

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International Journal of Clinical and Experimental Pathology

P1476

Triggering the succinate recep ...... right ventricular hypertrophy

@en

P2093

Di Yu

http://www.w3.org/2001/XMLSchema#string

Huan-Huan Fan

http://www.w3.org/2001/XMLSchema#string

Kai Zhou

http://www.w3.org/2001/XMLSchema#string

Lei Yang

http://www.w3.org/2001/XMLSchema#string

Liang Hu

http://www.w3.org/2001/XMLSchema#string

Wei-Yan Zhang

http://www.w3.org/2001/XMLSchema#string

Xu-Ming Mo

http://www.w3.org/2001/XMLSchema#string

Yu Feng

http://www.w3.org/2001/XMLSchema#string

P2860

Treatment goals of pulmonary hypertension

Right ventricular failure and pathobiology in patients with congenital heart disease - implications for long-term follow-up

Citric acid cycle intermediates as ligands for orphan G-protein-coupled receptors

Sitagliptin reduces cardiac apoptosis, hypertrophy and fibrosis primarily by insulin-dependent mechanisms in experimental type-II diabetes. Potential roles of GLP-1 isoforms

Cytosolic dynamics of annexin A6 trigger feedback regulation of hypertrophy via atrial natriuretic peptide in cardiomyocytes

Keynote Lecture-Transmitral hypertrophic obstructive cardiomyopathy (HOCM) repair.

Succinate is an inflammatory signal that induces IL-1β through HIF-1α.

Succinate: a metabolic signal in inflammation.

Biventricular remodeling in murine models of right ventricular pressure overload

Molecular signature of a right heart failure program in chronic severe pulmonary hypertension.

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scholarly article

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