Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A - Wikidata - awgldk - TriplyDB

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

http://www.wikidata.org/entity/Q34379744

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Role of protein kinase A in regulating mitochondrial function and neuronal development: implications to neurodegenerative diseases Emerging modes of PINK1 signaling: another task for MARK2 Beyond mitophagy: cytosolic PINK1 as a messenger of mitochondrial health Exploring the nexus of Alzheimer's disease and related dementias with cancer and cancer therapies: A convening of the Alzheimer's Association & Alzheimer's Drug Discovery Foundation.Parkinson's disease-implicated kinases in the brain; insights into disease pathogenesis.Mitochondrial Morphology and Fundamental Parameters of the Mitochondrial Respiratory Chain Are Altered in Caenorhabditis elegans Strains Deficient in Mitochondrial Dynamics and Homeostasis Processes.Cytosolic PTEN-induced Putative Kinase 1 Is Stabilized by the NF-κB Pathway and Promotes Non-selective Mitophagy.Parkinson Disease and Malignant Disease: Minding Cancer's Own Business.Melanoma genome evolution across species.PINK1 expression increases during brain development and stem cell differentiation, and affects the development of GFAP-positive astrocytes.Cytosolic PINK1 promotes the targeting of ubiquitinated proteins to the aggresome-autophagy pathway during proteasomal stress.PINK1 disables the anti-fission machinery to segregate damaged mitochondria for mitophagy.Back to the tubule: microtubule dynamics in Parkinson's disease.Cytosolic PINK1 escapes from mitochondria to promote dendritic outgrowth.E6-Associated Protein Dependent Estrogen Receptor Regulation of Protein Kinase A Regulatory Subunit R2A Expression in Neuroblastoma.Monomethylarsonous acid, but not inorganic arsenic, is a mitochondria-specific toxicant in vascular smooth muscle cells.PKA Phosphorylation of NCLX Reverses Mitochondrial Calcium Overload and Depolarization, Promoting Survival of PINK1-Deficient Dopaminergic Neurons PINK1 in the limelight: multiple functions of an eclectic protein in human health and disease.Parkin and PINK1 functions in oxidative stress and neurodegeneration.Long-term oral kinetin does not protect against α-synuclein-induced neurodegeneration in rodent models of Parkinson's disease.PINK1 Primes Parkin-Mediated Ubiquitination of PARIS in Dopaminergic Neuronal Survival.Glycolysis selectively shapes the presynaptic action potential waveform.Pink1 attenuates propofol-induced apoptosis and oxidative stress in developing neurons.Mitochondria: A Common Target for Genetic Mutations and Environmental Toxicants in Parkinson's Disease.The ubiquitin E3 ligase CHIP promotes proteasomal degradation of the serine/threonine protein kinase PINK1 during staurosporine-induced cell death.Mitochondrial Calcium Dysregulation Contributes to Dendrite Degeneration Mediated by PD/LBD-Associated LRRK2 Mutants.PINK1 regulates mitochondrial trafficking in dendrites of cortical neurons through mitochondrial PKA.Mitochondrial O-GlcNAc Transferase (mOGT) Regulates Mitochondrial Structure, Function, and Survival in HeLa Cells.The mitochondrial transcription factor TFAM in neurodegeneration: emerging evidence and mechanisms.Novel Loci Associated With Attention-Deficit/Hyperactivity Disorder Are Revealed by Leveraging Polygenic Overlap With Educational Attainment.Protocols for assessing mitophagy in neuronal cell lines and primary neurons.Role of Mitochondrial Dysfunction in Degenerative Brain Diseases, an Overview Excitatory Dendritic Mitochondrial Calcium Toxicity: Implications for Parkinson's and Other Neurodegenerative Diseases

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P2860

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

http://www.wikidata.org/entity/Q34379744

description

2013 nî lūn-bûn

@nan

2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

@ast

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

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Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

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type

label

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

@ast

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

@en

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

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prefLabel

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

@ast

Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

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Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

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Journal of Neurochemistry

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Beyond the mitochondrion: cytosolic PINK1 remodels dendrites through protein kinase A

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Irene Pien

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Jason Callio

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Jianhui Zhu

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Kent Z Q Wang

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Raul Y Dagda

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Ruth Wang

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P2860

PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

Parkinson's disease-associated kinase PINK1 regulates Miro protein level and axonal transport of mitochondria

PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility

Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability

PINK1 protects against oxidative stress by phosphorylating mitochondrial chaperone TRAP1

PINK1 cleavage at position A103 by the mitochondrial protease PARL.

LRRK2 kinase regulates synaptic morphology through distinct substrates at the presynaptic and postsynaptic compartments of the Drosophila neuromuscular junction

A new cytosolic pathway from a Parkinson disease-associated kinase, BRPK/PINK1: activation of AKT via mTORC2

Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission

The kinase domain of mitochondrial PINK1 faces the cytoplasm

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6

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Charleen T. Chu

Tania Das Banerjee

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