Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states. - Wikidata - awgldk - TriplyDB

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

http://www.wikidata.org/entity/Q34383914

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Internal Tandem Duplication in FLT3 Attenuates Proliferation and Regulates Resistance to the FLT3 Inhibitor AC220 by Modulating p21Cdkn1a and Pbx1 in Hematopoietic Cells DNA damage during S-phase mediates the proliferation-quiescence decision in the subsequent G1 via p21 expression.The finite state projection approach to analyze dynamics of heterogeneous populations.Cell cycle proliferation decisions: the impact of single cell analyses.Hypoxia induces an undifferentiated phenotype of oral keratinocytes in vitro Are Quasi-Steady-State Approximated Models Suitable for Quantifying Intrinsic Noise Accurately?AG490 and PF431396 Sensitive Tyrosine Kinase Control the Population Heterogeneity of Basal STAT1 Activity in Ube1l Deficient Cells FNC efficiently inhibits mantle cell lymphoma growth.Variation is function: Are single cell differences functionally important?: Testing the hypothesis that single cell variation is required for aggregate function Principles of dynamical modularity in biological regulatory networks.A Dynamical Framework for the All-or-None G1/S Transition.Unraveling Growth Factor Signaling and Cell Cycle Progression in Individual Fibroblasts Dynamics of CDKN1A in Single Cells Defined by an Endogenous Fluorescent Tagging Toolkit.Roles of CDKN1A gene polymorphisms (rs1801270 and rs1059234) in the development of cervical neoplasia.Exit from quiescence displays a memory of cell growth and division.Inhibition of WIP1 phosphatase sensitizes breast cancer cells to genotoxic stress and to MDM2 antagonist nutlin-3.Around and beyond 53BP1 Nuclear Bodies.Inherited DNA lesions determine G1 duration in the next cell cycle.Leveraging and coping with uncertainty in the response of individual cells to therapy.Mathematical modelling of reversible transition between quiescence and proliferation.Mechanism of metastasis suppression by luteolin in breast cancer.Control of the Restriction Point by Rb and p21

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P2860

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

http://www.wikidata.org/entity/Q34383914

description

2014 nî lūn-bûn

@nan

2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@ast

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@en

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@nl

type

label

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@ast

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@en

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@nl

prefLabel

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@ast

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@en

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states.

@nl

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PNAS.1409797111

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Basal p21 controls population heterogeneity in cycling and quiescent cell cycle states

@en

P2093

Clifford L Wang

http://www.w3.org/2001/XMLSchema#string

K Wesley Overton

http://www.w3.org/2001/XMLSchema#string

Tobias Meyer

http://www.w3.org/2001/XMLSchema#string

William L Noderer

http://www.w3.org/2001/XMLSchema#string

P2860

The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

WAF1, a potential mediator of p53 tumor suppression

Stochastic gene expression in a single cell

Exploring the contextual sensitivity of factors that determine cell-to-cell variability in receptor-mediated apoptosis

The proliferation-quiescence decision is controlled by a bifurcation in CDK2 activity at mitotic exit.

Mitochondrial dysfunction accounts for the stochastic heterogeneity in telomere-dependent senescence.

Stochastic E2F activation and reconciliation of phenomenological cell-cycle models.

Origin of bistability underlying mammalian cell cycle entry

Proteasomal turnover of p21Cip1 does not require p21Cip1 ubiquitination.

Non-genetic cell-to-cell variability and the consequences for pharmacology

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E4386-93

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scholarly article

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10.1073/PNAS.1409797111

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41

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111

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Sabrina L Spencer

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2014-09-29T00:00:00Z

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266324382

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4205626

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