Axonal protein synthesis and degradation are necessary for efficient growth cone regeneration. - Wikidata - awgldk - TriplyDB

Axonal protein synthesis and degradation are necessary for efficient growth cone regeneration.

Axonal protein synthesis and degradation are necessary for efficient growth cone regeneration.

http://www.wikidata.org/entity/Q34383999

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Characterization of the adaptor protein ARH expression in the brain and ARH molecular interactions Inhibition of Kinesin-5, a microtubule-based motor protein, as a strategy for enhancing regeneration of adult axons Efficient retrograde transport of pseudorabies virus within neurons requires local protein synthesis in axons Axonal protein synthesis and the regulation of local mitochondrial function Rheb and mTOR regulate neuronal polarity through Rap1B The neurology of mTOR Charcot-Marie-Tooth disease and intracellular traffic Remote control of gene function by local translation C. elegans as a genetic model to identify novel cellular and molecular mechanisms underlying nervous system regeneration The core apoptotic executioner proteins CED-3 and CED-4 promote initiation of neuronal regeneration in Caenorhabditis elegans Growth control mechanisms in neuronal regeneration The RNA binding and transport proteins staufen and fragile X mental retardation protein are expressed by rat primary afferent neurons and localize to peripheral and central axons Intra-axonal synthesis of eukaryotic translation initiation factors regulates local protein synthesis and axon growth in rat sympathetic neurons Caenorhabditis elegans neuronal regeneration is influenced by life stage, ephrin signaling, and synaptic branching.Extracellular stimuli specifically regulate localized levels of individual neuronal mRNAs.Axon regeneration requires a conserved MAP kinase pathway.Protein synthesis in distal axons is not required for growth cone responses to guidance cues Beta-subunits promote the expression of Ca(V)2.2 channels by reducing their proteasomal degradation.Axon regeneration requires coordinate activation of p38 and JNK MAPK pathways.Autophagy induction stabilizes microtubules and promotes axon regeneration after spinal cord injury.Dynamic remodelling of synapses can occur in the absence of the parent cell body Local translation in primary afferent fibers regulates nociception Bioinformatic and statistical analysis of the optic nerve head in a primate model of ocular hypertension.Distinct modes of neuritic growth in purkinje neurons at different developmental stages: axonal morphogenesis and cellular regulatory mechanisms Inhibition of the mammalian target of rapamycin signaling pathway suppresses dentate granule cell axon sprouting in a rodent model of temporal lobe epilepsy Pericontusion axon sprouting is spatially and temporally consistent with a growth-permissive environment after traumatic brain injury.Roles of channels and receptors in the growth cone during PNS axonal regeneration.Regulation of protein levels in subcellular domains through mRNA transport and localized translation.Neuroproteomics approaches to decipher neuronal regeneration and degeneration.Axonal transport proteomics reveals mobilization of translation machinery to the lesion site in injured sciatic nerve.Cytokines that promote nerve regeneration.Killer proteases and little strokes--how the things that do not kill you make you stronger.Syntaxin13 expression is regulated by mammalian target of rapamycin (mTOR) in injured neurons to promote axon regeneration mTORC1-independent reduction of retinal protein synthesis in type 1 diabetes.Mammalian target of rapamycin (mTOR) activation increases axonal growth capacity of injured peripheral nerves Functional complexity of the axonal growth cone: a proteomic analysis.Two distinct types of dying back axonal degeneration in vitro.The central dogma decentralized: new perspectives on RNA function and local translation in neurons.Clustering of the neural cell adhesion molecule (NCAM) at the neuronal cell surface induces caspase-8- and -3-dependent changes of the spectrin meshwork required for NCAM-mediated neurite outgrowth Sensing nerve injury at the axonal ER: activated Luman/CREB3 serves as a novel axonally synthesized retrograde regeneration signal.

P2860

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P2860

Axonal protein synthesis and degradation are necessary for efficient growth cone regeneration.

http://www.wikidata.org/entity/Q34383999

description

2005 nî lūn-bûn

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2005 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2005 թվականի հունվարին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Axonal protein synthesis and d ...... ient growth cone regeneration.

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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type

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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Axonal protein synthesis and d ...... ient growth cone regeneration.

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P356

JNEUROSCI.3073-04.2005

P1433

Journal of Neuroscience

P1476

Axonal protein synthesis and d ...... ient growth cone regeneration.

@en

P2093

Amanda M Codd

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Douglas S Campbell

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James W Fawcett

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Poonam Verma

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Ronald L Meyer

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Sabrina Chierzi

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P2860

eIF4 initiation factors: effectors of mRNA recruitment to ribosomes and regulators of translation

Translational control of ribosomal protein L4 mRNA is required for rapid neurite regeneration

Effects of neurotoxic and neuroprotective agents on peripheral nerve regeneration assayed by time-lapse imaging in vivo.

Protein-synthesizing machinery in the axon compartment.

ICP0 induces the accumulation of colocalizing conjugated ubiquitin.

Extracellular-signal-regulated kinase signalling in neurons.

Adaptation in the chemotactic guidance of nerve growth cones.

Netrin-1-mediated axon outgrowth requires deleted in colorectal cancer-dependent MAPK activation.

Age-related changes underlie switch in netrin-1 responsiveness as growth cones advance along visual pathway.

Microtubule polarity reversal accompanies regrowth of amputated neurites

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331-342

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scholarly article

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10.1523/JNEUROSCI.3073-04.2005

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2

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25

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15647476

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3687202

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