Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state - Wikidata - awgldk - TriplyDB

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

http://www.wikidata.org/entity/Q34390637

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Impact of recent innovations in the use of mass cytometry in support of drug development Chemicals as the Sole Transformers of Cell Fate Cellular Reprogramming Using Defined Factors and MicroRNAs Young at Heart: Pioneering Approaches to Model Nonischaemic Cardiomyopathy with Induced Pluripotent Stem Cells Micromanaging cardiac regeneration: Targeted delivery of microRNAs for cardiac repair and regeneration Reprogramming and transdifferentiation for cardiovascular development and regenerative medicine: where do we stand?Direct Reprogramming-The Future of Cardiac Regeneration?Programming and reprogramming a human heart cell MicroRNA-mediated regulation of differentiation and trans-differentiation in stem cells Investigating the transcriptional control of cardiovascular development The T-box gene family: emerging roles in development, stem cells and cancer Inhibition of TGFβ signaling increases direct conversion of fibroblasts to induced cardiomyocytes Induction of diverse cardiac cell types by reprogramming fibroblasts with cardiac transcription factors.Analysis of the dynamic co-expression network of heart regeneration in the zebrafish.Strategies and Challenges to Myocardial Replacement Therapy Cardiac fibrosis in myocardial infarction-from repair and remodeling to regeneration Direct reprogramming and biomaterials for controlling cell fate The roles of Mesp family proteins: functional diversity and redundancy in differentiation of pluripotent stem cells and mammalian mesodermal development Engineered Human Muscle Tissue from Skeletal Muscle Derived Stem Cells and Induced Pluripotent Stem Cell Derived Cardiac Cells A protein-tagging system for signal amplification in gene expression and fluorescence imaging.Directed Differentiation of Embryonic Stem Cells Into Cardiomyocytes by Bacterial Injection of Defined Transcription Factors Generation of Functional Human Cardiac Progenitor Cells by High-Efficiency Protein Transduction.Bmi1 Is a Key Epigenetic Barrier to Direct Cardiac Reprogramming.Core Transcription Factors, MicroRNAs, and Small Molecules Drive Transdifferentiation of Human Fibroblasts Towards The Cardiac Cell Lineage.Partial Reprogramming of Pluripotent Stem Cell-Derived Cardiomyocytes into Neurons.Reprogramming cell fates by small molecules.Reprogramming the cardiac field Direct reprogramming of fibroblasts into cardiomyocytes 'Above all, do no harm': safeguarding pluripotent stem cell therapy against iatrogenic tumorigenesis.Directed cardiomyogenesis of autologous human induced pluripotent stem cells recruited to infarcted myocardium with bioengineered antibodies.Cell replacement therapies: is it time to reprogram?MiR-133 promotes cardiac reprogramming by directly repressing Snai1 and silencing fibroblast signatures Human stem cells for modeling heart disease and for drug discovery.CellNet: network biology applied to stem cell engineering Carboxy terminus of GATA4 transcription factor is required for its cardiogenic activity and interaction with CDK4.Strategies for cardiac regeneration and repair.Integrating omics into the cardiac differentiation of human pluripotent stem cells.Reprogramming for cardiac regeneration.An emerging consensus on cardiac regeneration.The Drosophila Transcription Factors Tinman and Pannier Activate and Collaborate with Myocyte Enhancer Factor-2 to Promote Heart Cell Fate.

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Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

http://www.wikidata.org/entity/Q34390637

description

2013 nî lūn-bûn

@nan

2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2013年の論文

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2013年論文

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2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@ast

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@en

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@nl

type

label

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@ast

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@en

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@nl

prefLabel

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@ast

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@en

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@nl

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J.STEMCR.2013.07.005

P1433

Stem Cell Reports

P1476

Direct reprogramming of human fibroblasts toward a cardiomyocyte-like state

@en

P2093

Benoit G Bruneau

http://www.w3.org/2001/XMLSchema#string

C Ian Spencer

http://www.w3.org/2001/XMLSchema#string

Deepak Srivastava

http://www.w3.org/2001/XMLSchema#string

Lei Liu

http://www.w3.org/2001/XMLSchema#string

Li Qian

http://www.w3.org/2001/XMLSchema#string

Nicole R Stone

http://www.w3.org/2001/XMLSchema#string

Sheng Ding

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P2860

GATA4 mutations cause human congenital heart defects and reveal an interaction with TBX5

Mesp1 acts as a master regulator of multipotent cardiovascular progenitor specification

Direct conversion of fibroblasts to functional neurons by defined factors

Lentiviral vectors and protocols for creation of stable hESC lines for fluorescent tracking and drug resistance selection of cardiomyocytes

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

Embryonic stem cell lines derived from human blastocysts

Myocardin: a component of a molecular switch for smooth muscle differentiation

Activation of cardiac gene expression by myocardin, a transcriptional cofactor for serum response factor

FOG-2, a cofactor for GATA transcription factors, is essential for heart morphogenesis and development of coronary vessels from epicardium

Myocardin is required for cardiomyocyte survival and maintenance of heart function

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235-247

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scholarly article

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10.1016/J.STEMCR.2013.07.005

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3

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1

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Paul Delgado-Olguín

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2013-08-22T00:00:00Z

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259250065

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24319660

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3849259

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