about
Transcriptional and functional profiling of human embryonic stem cell-derived cardiomyocytesDistinct roles of microRNA-1 and -499 in ventricular specification and functional maturation of human embryonic stem cell-derived cardiomyocytesIn vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytesBmi1 Is a Key Epigenetic Barrier to Direct Cardiac Reprogramming.Electrophysiological properties of human induced pluripotent stem cells.Direct reprogramming of human fibroblasts toward a cardiomyocyte-like stateConversion of human fibroblasts into functional cardiomyocytes by small molecules.Contractility of single cardiomyocytes differentiated from pluripotent stem cells depends on physiological shape and substrate stiffnessFacilitated maturation of Ca2+ handling properties of human embryonic stem cell-derived cardiomyocytes by calsequestrin expression.Direct reprogramming of fibroblasts into cardiomyocytes for cardiac regenerative medicine.A Singular Role of IK1 Promoting the Development of Cardiac Automaticity during Cardiomyocyte Differentiation by IK1 -Induced Activation of Pacemaker Current.Distinct cardiogenic preferences of two human embryonic stem cell (hESC) lines are imprinted in their proteomes in the pluripotent stateCrucial role of the sarcoplasmic reticulum in the developmental regulation of Ca2+ transients and contraction in cardiomyocytes derived from embryonic stem cells.Single cell qPCR reveals that additional HAND2 and microRNA-1 facilitate the early reprogramming progress of seven-factor-induced human myocytes.Protective effect of estrogen on intestinal ischemia-reperfusion injury in pubertal rats.Disease Model of GATA4 Mutation Reveals Transcription Factor Cooperativity in Human Cardiogenesis.Expandable Cardiovascular Progenitor Cells Reprogrammed from Fibroblasts.An inducible transgene expression system for regulated phenotypic modification of human embryonic stem cells.Repolarization Reserve and Action Potential Dynamics in Failing Myocytes.Reprogramming of mouse fibroblasts into cardiomyocyte-like cells in vitro.S-phase Synchronization Facilitates the Early Progression of Induced-Cardiomyocyte Reprogramming through Enhanced Cell-Cycle Exit.
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Ji-Dong Fu
@en
Ji-Dong Fu
@nl
type
label
Ji-Dong Fu
@en
Ji-Dong Fu
@nl
prefLabel
Ji-Dong Fu
@en
Ji-Dong Fu
@nl
P31
P496
0000-0003-2797-5193