Abundant kif21b is associated with accelerated progression in neurodegenerative diseases. - Wikidata - awgldk - TriplyDB

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

http://www.wikidata.org/entity/Q34392911

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Structural basis for misregulation of kinesin KIF21A autoinhibition by CFEOM1 disease mutations The Kinesin KIF21B Regulates Microtubule Dynamics and Is Essential for Neuronal Morphology, Synapse Function, and Learning and Memory Kinesin-4 KIF21B is a potent microtubule pausing factor.

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Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

http://www.wikidata.org/entity/Q34392911

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@ast

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@en

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@nl

type

label

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@ast

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@en

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@nl

prefLabel

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@ast

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@en

Abundant kif21b is associated with accelerated progression in neurodegenerative diseases.

@nl

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Acta neuropathologica communications

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Abundant kif21b is associated with accelerated progression in neurodegenerative diseases

@en

P2093

Annet F Wierenga-Wolf

http://www.w3.org/2001/XMLSchema#string

Ben A Oostra

http://www.w3.org/2001/XMLSchema#string

Elly M Hol

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Jon D Laman

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Marie-Jose Melief

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Marjan van Meurs

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Miriam E van Strien

http://www.w3.org/2001/XMLSchema#string

Rogier Q Hintzen

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P2860

Rating neurologic impairment in multiple sclerosis: An expanded disability status scale (EDSS)

The innate immune system in demyelinating disease

Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis

Novel dendritic kinesin sorting identified by different process targeting of two related kinesins: KIF21A and KIF21B

Neuropathological stageing of Alzheimer-related changes

Detection of cortical inflammatory lesions by double inversion recovery magnetic resonance imaging in patients with multiple sclerosis

A novel KIF21A mutation in a patient with congenital fibrosis of the extraocular muscles and Marcus Gunn jaw-winking phenomenon

The blood-brain barrier in cortical multiple sclerosis lesions

Genetic variation in the KIF1B locus influences susceptibility to multiple sclerosis

Kinesin superfamily motor proteins and intracellular transport

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10.1186/PREACCEPT-1855650047115971

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neurodegeneration

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