Macrophages recruited via CCR2 produce insulin-like growth factor-1 to repair acute skeletal muscle injury.
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Extrinsic and intrinsic control of macrophage inflammatory responsesModulating exercise-induced hormesis: Does less equal more?Adipose tissue in obesity-related inflammation and insulin resistance: cells, cytokines, and chemokinesImmune modulation of stem cells and regenerationGrowth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasisAgeing prolongs inflammatory marker expression in regenerating rat skeletal muscles after injuryInflammation-a Critical Appreciation of the Role of Myeloid CellsIn Silico and In Vivo Experiments Reveal M-CSF Injections Accelerate Regeneration Following Muscle Laceration.The regulation of inflammatory mediators in acute kidney injury via exogenous mesenchymal stem cells.Concurrent Aerobic and Resistance Training Has Anti-Inflammatory Effects and Increases Both Plasma and Leukocyte Levels of IGF-1 in Late Middle-Aged Type 2 Diabetic Patients.IGF1 Shapes Macrophage Activation in Response to Immunometabolic Challenge.MCP/CCR2 signaling is essential for recruitment of mesenchymal progenitor cells during the early phase of fracture healing.Insulin resistance is associated with MCP1-mediated macrophage accumulation in skeletal muscle in mice and humansInflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR2.Sequenced response of extracellular matrix deadhesion and fibrotic regulators after muscle damage is involved in protection against future injury in human skeletal muscle.Beneficial effects of cod protein on inflammatory cell accumulation in rat skeletal muscle after injury are driven by its high levels of arginine, glycine, taurine and lysine.Deficient CX3CR1 signaling promotes recovery after mouse spinal cord injury by limiting the recruitment and activation of Ly6Clo/iNOS+ macrophages.Low-frequency electrical stimulation attenuates muscle atrophy in CKD--a potential treatment strategyAcute skeletal muscle injury: CCL2 expression by both monocytes and injured muscle is required for repair.Anti-inflammatory macrophages improve skeletal muscle recovery from ischemia-reperfusion.Messenger RNA sequencing and pathway analysis provide novel insights into the biological basis of chickens' feed efficiency.FOXP3+ T Cells Recruited to Sites of Sterile Skeletal Muscle Injury Regulate the Fate of Satellite Cells and Guide Effective Tissue Regeneration.Pancreatic islet expression of chemokine CCL2 suppresses autoimmune diabetes via tolerogenic CD11c+ CD11b+ dendritic cellsThe Development of Macrophage-Mediated Cell Therapy to Improve Skeletal Muscle Function after Injury.CD11b+Ly6G- myeloid cells mediate mechanical inflammatory pain hypersensitivity.CX3CR1 deficiency delays acute skeletal muscle injury repair by impairing macrophage functions.CX3CR1 deficiency promotes muscle repair and regeneration by enhancing macrophage ApoE productionObesity-driven inflammation and cancer risk: role of myeloid derived suppressor cells and alternately activated macrophages.Monocyte/Macrophage-derived IGF-1 Orchestrates Murine Skeletal Muscle Regeneration and Modulates Autocrine Polarization.Monocytes and macrophages in tissue repair: Implications for immunoregenerative biomaterial designExercise training and peripheral arterial disease.Cellular dynamics in the muscle satellite cell niche.Muscle-specific deletion of SOCS3 increases the early inflammatory response but does not affect regeneration after myotoxic injuryMacrophages and CSF-1: implications for development and beyondMatrix Metalloproteinases and Tissue Inhibitor of Metalloproteinases in Inflammation and Fibrosis of Skeletal MusclesPretreatment with Fish Oil-Based Lipid Emulsion Modulates Muscle Leukocyte Chemotaxis in Murine Model of Sublethal Lower Limb Ischemia.Accelerated regeneration of the skeletal muscle in RNF13-knockout mice is mediated by macrophage-secreted IL-4/IL-6.Altered macrophage phenotype transition impairs skeletal muscle regenerationMacrophages in injured skeletal muscle: a perpetuum mobile causing and limiting fibrosis, prompting or restricting resolution and regeneration.Mononuclear phagocyte system in kidney disease and repair.
P2860
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P2860
Macrophages recruited via CCR2 produce insulin-like growth factor-1 to repair acute skeletal muscle injury.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年学术文章
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2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
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2010年学术文章
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2010年學術文章
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name
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@ast
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@en
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@nl
type
label
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@ast
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@en
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@nl
prefLabel
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@ast
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@en
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@nl
P2093
P2860
P356
P1433
P1476
Macrophages recruited via CCR2 ...... acute skeletal muscle injury.
@en
P2093
Danping Huang
Israel F Charo
Noah Saederup
Richard M Ransohoff
P2860
P304
P356
10.1096/FJ.10-171579
P407
P577
2010-10-01T00:00:00Z