Mesenchymal-endothelial transition contributes to cardiac neovascularization. - Wikidata - awgldk - TriplyDB

Mesenchymal-endothelial transition contributes to cardiac neovascularization.

Mesenchymal-endothelial transition contributes to cardiac neovascularization.

http://www.wikidata.org/entity/Q34429225

about

Endothelial-Mesenchymal Transition in Regenerative Medicine Leukocytes: The Double-Edged Sword in Fibrosis Programming and reprogramming a human heart cell Cardiac Light-Sheet Fluorescent Microscopy for Multi-Scale and Rapid Imaging of Architecture and Function Hepatitis B virus-associated intrahepatic cholangiocarcinoma: a malignancy of distinctive characteristics between hepatocellular carcinoma and intrahepatic cholangiocarcinoma Developmental origin and lineage plasticity of endogenous cardiac stem cells Key nodes of a microRNA network associated with the integrated mesenchymal subtype of high-grade serous ovarian cancer Genetic tools for identifying and manipulating fibroblasts in the mouse Bmi1 Is a Key Epigenetic Barrier to Direct Cardiac Reprogramming.Chromatin-modifying agents convert fibroblasts to OCT4+ and VEGFR-2+ capillary tube-forming cells.Instructive Role of the Microenvironment in Preventing Renal Fibrosis SOX17 Regulates Conversion of Human Fibroblasts Into Endothelial Cells and Erythroblasts by Dedifferentiation Into CD34+ Progenitor Cells.Cardiac fibroblasts: from development to heart failure.Fibroblasts Rendered Antifibrotic, Antiapoptotic, and Angiogenic by Priming With Cardiosphere-Derived Extracellular Membrane Vesicles.Wnt10b Gain-of-Function Improves Cardiac Repair by Arteriole Formation and Attenuation of Fibrosis.cKit+ cardiac progenitors of neural crest origin.Rational transplant timing and dose of mesenchymal stromal cells in patients with acute myocardial infarction: a meta-analysis of randomized controlled trials.Phenotypically heterogeneous podoplanin-expressing cell populations are associated with the lymphatic vessel growth and fibrogenic responses in the acutely and chronically infarcted myocardium.Murine Models Demonstrate Distinct Vasculogenic and Cardiomyogenic cKit+ Lineages in the Heart Transcriptional control of cardiac fibroblast plasticity.A Repertoire of MicroRNAs Regulates Cancer Cell Starvation by Targeting Phospholipase D in a Feedback Loop That Operates Maximally in Cancer Cells Reciprocal Regulation of the Cardiac Epigenome by Chromatin Structural Proteins Hmgb and Ctcf: IMPLICATIONS FOR TRANSCRIPTIONAL REGULATION.Transcription factor TBX4 regulates myofibroblast accumulation and lung fibrosis.Endothelial cells are progenitors of cardiac pericytes and vascular smooth muscle cells.The long noncoding RNA Chaer defines an epigenetic checkpoint in cardiac hypertrophy.Interfacial Adipose Tissue in Systemic Sclerosis.Endothelial primary cilia inhibit atherosclerosis.Advanced imaging approaches for regenerative medicine: Emerging technologies for monitoring stem cell fate in vitro and in vivo.Concise Review: Pluripotent Stem Cell-Derived Cardiac Cells, A Promising Cell Source for Therapy of Heart Failure: Where Do We Stand?The third path of tubulointerstitial fibrosis: aberrant endothelial secretome.Preexisting endothelial cells mediate cardiac neovascularization after injury.Transdifferentiation Requires iNOS Activation: Role of RING1A S-Nitrosylation.Chemical transdifferentiation: closer to regenerative medicine.Stress-Induced Premature Senescence of Endothelial and Endothelial Progenitor Cells.Skeletal and cardiac muscle pericytes: Functions and therapeutic potential.Defining the Cardiac Fibroblast.Selenium Augments microRNA Directed Reprogramming of Fibroblasts to Cardiomyocytes via Nanog.Cellular plasticity in cardiovascular development and disease.The molecular basis of endothelial cell plasticity.Redefining the identity of cardiac fibroblasts.

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

http://www.wikidata.org/entity/Q34429225

description

2014 nî lūn-bûn

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2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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type

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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prefLabel

Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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Mesenchymal-endothelial transition contributes to cardiac neovascularization.

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Arjun Deb

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Enrico Stefani

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Eric Ubil

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Francesca Bargiacchi

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Jie Huang

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Jinzhu Duan

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Mauricio Rojas

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Seta Stanbouly

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Thomas M Vondriska

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Yan Lu

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P2860

Myofibroblasts and mechano-regulation of connective tissue remodelling

A guided tour into subcellular colocalization analysis in light microscopy

A robust and high-throughput Cre reporting and characterization system for the whole mouse brain

Suppression of induced pluripotent stem cell generation by the p53-p21 pathway

Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

In vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytes

Cardiac fibroblast: the renaissance cell.

Direct labeling and visualization of blood vessels with lipophilic carbocyanine dye DiI.

Small molecule RITA binds to p53, blocks p53-HDM-2 interaction and activates p53 function in tumors.

Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.

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585-590

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10.1038/NATURE13839

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7524

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514

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Manuel Rosa-Garrido

Indulekha C L Pillai

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266946816

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neovascularization

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4214889

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