Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53. - Wikidata - awgldk - TriplyDB

Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.

Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.

http://www.wikidata.org/entity/Q33853515

about

miR-30 regulates mitochondrial fission through targeting p53 and the dynamin-related protein-1 pathway Apoptosis in myocardial ischaemia and infarction Histone deacetylase inhibition reduces myocardial ischemia-reperfusion injury in mice Fabrication of mouse embryonic stem cell-derived layered cardiac cell sheets using a bioreactor culture system Puma joins the battery of BH3-only proteins that promote death and infarction during myocardial ischemia MitoNEET Protects HL-1 Cardiomyocytes from Oxidative Stress Mediated Apoptosis in an In Vitro Model of Hypoxia and Reoxygenation Transcription factor Foxo3a prevents apoptosis by regulating calcium through the apoptosis repressor with caspase recruitment domain p53 initiates apoptosis by transcriptionally targeting the antiapoptotic protein ARC Soluble adenylyl cyclase controls mitochondria-dependent apoptosis in coronary endothelial cells Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3.Mild hypoxia-induced cardiomyocyte hypertrophy via up-regulation of HIF-1α-mediated TRPC signalling p53 and TIGAR regulate cardiac myocyte energy homeostasis under hypoxic stress.Elucidation of how cancer cells avoid acidosis through comparative transcriptomic data analysis.Modulation of ceramide content and lack of apoptosis in the chronically hypoxic neonatal rat heart.Mitofusin 1 is negatively regulated by microRNA 140 in cardiomyocyte apoptosis.Inhibition of apoptosis by progesterone in cardiomyocytes.Hypoxia, angiotensin-II, and norepinephrine mediated apoptosis is stimulus specific in canine failed cardiomyocytes: a role for p38 MAPK, Fas-L and cyclin D1.Glucose reintroduction triggers the activation of Nrf2 during experimental ischemia reperfusion.Mesenchymal-endothelial transition contributes to cardiac neovascularization.Sabiporide improves cardiovascular function, decreases the inflammatory response and reduces mortality in acute metabolic acidosis in pigs.The Role of microRNAs in Mitochondria: Small Players Acting Wide.c-Jun N-terminal kinase (JNK-1) confers protection against brief but not extended ischemia during acute myocardial infarction.Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy.Cyclosporine A regulate oxidative stress-induced apoptosis in cardiomyocytes: mechanisms via ROS generation, iNOS and Hsp70 PUMA is critical for neonatal cardiomyocyte apoptosis induced by endoplasmic reticulum stress Bnip3 Binds and Activates p300: Possible Role in Cardiac Transcription and Myocyte Morphology.Inhibition of p53 function prevents renin-angiotensin system activation and stretch-mediated myocyte apoptosis BNIP3 promotes calcium and calpain-dependent cell death.DNase activation by hypoxia-acidosis parallels but is independent of programmed cell death Synergistic induction of miR-126 by hypoxia and HDAC inhibitors in cardiac myocytes.Cyclin-dependent kinase 2 signaling regulates myocardial ischemia/reperfusion injury.Vascular Regeneration in Ischemic Hindlimb by Adeno-Associated Virus Expressing Conditionally Silenced Vascular Endothelial Growth Factor Importance of bicarbonate transport for ischaemia-induced apoptosis of coronary endothelial cells.The role of Bcl-2 family member BNIP3 in cell death and disease: NIPping at the heels of cell death.Acidic preconditioning protects endothelial cells against apoptosis through p38- and Akt-dependent Bcl-xL overexpression.Sulindac confers high level ischemic protection to the heart through late preconditioning mechanisms.Acidic pre-conditioning suppresses apoptosis and increases expression of Bcl-xL in coronary endothelial cells under simulated ischaemia.The antiarrhythmic peptide rotigaptide (ZP123) increases gap junction intercellular communication in cardiac myocytes and HeLa cells expressing connexin 43.Effects of metabolic acidosis on intracellular pH responses in multiple cell types Enhanced cell survival and diminished apoptotic response to simulated ischemia-reperfusion in H9c2 cells by magnetic field preconditioning.

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P2860

Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.

http://www.wikidata.org/entity/Q33853515

description

1999 nî lūn-bûn

@nan

1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի օգոստոսին հրատարակված գիտական հոդված

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1999年の論文

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年學術文章

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name

Hypoxia-activated apoptosis of ...... ift and is independent of p53.

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Hypoxia-activated apoptosis of ...... ift and is independent of p53.

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Hypoxia-activated apoptosis of ...... ift and is independent of p53.

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Hypoxia-activated apoptosis of ...... ift and is independent of p53.

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Hypoxia-activated apoptosis of ...... ift and is independent of p53.

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Journal of Clinical Investigation

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Hypoxia-activated apoptosis of ...... ift and is independent of p53.

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B Sato

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D J Discher

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K A Webster

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N H Bishopric

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O Hernandez

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P2860

Apoptosis, oncosis, and necrosis. An overview of cell death

Adenovirus E1A inhibits cardiac myocyte-specific gene expression through its amino terminus

Tumor spectrum analysis in p53-mutant mice

Hypoxia-mediated selection of cells with diminished apoptotic potential in solid tumours

p53: puzzle and paradigm

Cell acidification in apoptosis: granulocyte colony-stimulating factor delays programmed cell death in neutrophils by up-regulating the vacuolar H(+)-ATPase.

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

Induction of the skeletal alpha-actin gene in alpha 1-adrenoceptor-mediated hypertrophy of rat cardiac myocytes

DNA fragmentation of human infarcted myocardial cells demonstrated by the nick end labeling method and DNA agarose gel electrophoresis.

Activation of human papillomavirus type 18 E6-E7 oncogene expression by transcription factor Sp1.

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3

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104

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10430605

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apoptotic process

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408414

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