Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS. - Wikidata - awgldk - TriplyDB

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.

http://www.wikidata.org/entity/Q34447034

about

The rise of mitochondria in medicine "Oxygen Sensing" by Na,K-ATPase: These Miraculous Thiols Mitochondrial metabolic remodeling in response to genetic and environmental perturbations The Dual Function of Reactive Oxygen/Nitrogen Species in Bioenergetics and Cell Death: The Role of ATP Synthase Mitochondrial Regulation of the Muscle Microenvironment in Critical Limb Ischemia Counteracting Protein Kinase Activity in the Heart: The Multiple Roles of Protein Phosphatases Calcium signaling as a mediator of cell energy demand and a trigger to cell death T cell metabolism drives immunity Role of the MPTP in conditioning the heart - translatability and mechanism Mitochondria in the regulation of innate and adaptive immunity A roadmap for interpreting (13)C metabolite labeling patterns from cells Regulation of drug-metabolizing enzymes by local and systemic liver injuries Systems Medicine as an Emerging Tool for Cardiovascular Genetics Oncometabolites: Unconventional triggers of oncogenic signalling cascades Computational Models of Reactive Oxygen Species as Metabolic Byproducts and Signal-Transduction Modulators Mitochondrial Quality Control in Cardiac Diseases Investigating mitochondrial redox state using NADH and NADPH autofluorescence Ischaemic conditioning and targeting reperfusion injury: a 30 year voyage of discovery Ischemic A/D transition of mitochondrial complex I and its role in ROS generation ROS homeostasis and metabolism: a dangerous liason in cancer cells SIRT3 in Cardiac Physiology and Disease Being right on Q: shaping eukaryotic evolution GPR91: expanding the frontiers of Krebs cycle intermediates Understanding and preventing mitochondrial oxidative damage Metabolic reprogramming in macrophages and dendritic cells in innate immunity Contribution of metabolic reprogramming to macrophage plasticity and function Effects of Interferons and Viruses on Metabolism Reactive nitrogen species in cellular signaling Interplay between oxidant species and energy metabolism Mitochondrial ROS regulate thermogenic energy expenditure and sulfenylation of UCP1 Sex is a ubiquitous, ancient, and inherent attribute of eukaryotic life Mechanistic characterization of nitrite-mediated neuroprotection after experimental cardiac arrest Assessment of H2S in vivo using the newly developed mitochondria-targeted mass spectrometry probe MitoA Prophylactic Edaravone Prevents Transient Hypoxic-Ischemic Brain Injury: Implications for Perioperative Neuroprotection.Mitochondrial complex I deactivation is related to superoxide production in acute hypoxia.Epigenomic and transcriptomic approaches in the post-genomic era: path to novel targets for diagnosis and therapy of the ischaemic heart? Position Paper of the European Society of Cardiology Working Group on Cellular Biology of the Heart.Inhibition of sarcolemmal FAT/CD36 by sulfo-N-succinimidyl oleate rapidly corrects metabolism and restores function in the diabetic heart following hypoxia/reoxygenation European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Protective effect of aplysin on liver tissue and the gut microbiota in alcohol-fed rats.Focally perfused succinate potentiates brain metabolism in head injury patients

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P2860

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.

http://www.wikidata.org/entity/Q34447034

description

2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Ischaemic accumulation of succ ...... ury through mitochondrial ROS.

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Ischaemic accumulation of succ ...... ury through mitochondrial ROS.

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Ischaemic accumulation of succ ...... ury through mitochondrial ROS.

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Ischaemic accumulation of succ ...... ury through mitochondrial ROS.

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Ischaemic accumulation of succ ...... ury through mitochondrial ROS.

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Ischaemic accumulation of succ ...... ury through mitochondrial ROS.

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Ischaemic accumulation of succ ...... ury through mitochondrial ROS.

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Alan J Robinson

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Andrew M James

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Angela Logan

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Anna J Dare

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Chou-Hui Hu

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Edoardo Gaude

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Edward T Chouchani

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Ellen L Robb

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Filmon Eyassu

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Paul S Brookes

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P2860

Computational prediction of human metabolic pathways from the complete human genome

The mechanism of superoxide production by NADH:ubiquinone oxidoreductase (complex I) from bovine heart mitochondria

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Combined antiapoptotic and antioxidant approach to acute neuroprotection for stroke in hypertensive rats.

P2888

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431-435

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scholarly article

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10.1038/NATURE13909

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7527

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515

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Michael R. Duchen

Michael P. Murphy

Anthony C Smith

Michael Shattock

Richard C Hartley

Christian Frezza

Lorraine M Work

Kourosh Saeb-Parsy

Sean M Davidson

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2014-11-05T00:00:00Z

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267790296

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1017423660

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25383517

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reperfusion injury

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4255242

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