BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ. - Wikidata - awgldk - TriplyDB

BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ.

BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ.

http://www.wikidata.org/entity/Q34449830

about

The BRCA1/2 pathway prevents hematologic cancers in addition to breast and ovarian cancers Inhibition of homologous recombination by the PCNA-interacting protein PARI FAAP100 is essential for activation of the Fanconi anemia-associated DNA damage response pathway CtIP is required to initiate replication-dependent interstrand crosslink repair Fanconi anemia (FA) binding protein FAAP20 stabilizes FA complementation group A (FANCA) and participates in interstrand cross-link repair The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells PALB2 regulates recombinational repair through chromatin association and oligomerization Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome Molecular pathogenesis and clinical management of Fanconi anemia Susceptibility pathways in Fanconi's anemia and breast cancer How the fanconi anemia pathway guards the genome G4 resolvase 1 binds both DNA and RNA tetramolecular quadruplex with high affinity and is the major source of tetramolecular quadruplex G4-DNA and G4-RNA resolving activity in HeLa cell lysates Cigarette smoke induces genetic instability in airway epithelial cells by suppressing FANCD2 expression Getting Ready for the Dance: FANCJ Irons Out DNA Wrinkles Mechanisms of interstrand DNA crosslink repair and human disorders Interplay between Fanconi anemia and homologous recombination pathways in genome integrity Disease-causing missense mutations in human DNA helicase disorders Molecular functions and cellular roles of the ChlR1 (DDX11) helicase defective in the rare cohesinopathy Warsaw breakage syndrome Emerging critical roles of Fe-S clusters in DNA replication and repair.Insights into Fanconi Anaemia from the structure of human FANCE Molecular basis of BACH1/FANCJ recognition by TopBP1 in DNA replication checkpoint control Yeast Mph1 helicase dissociates Rad51-made D-loops: implications for crossover control in mitotic recombination.Chl1 DNA helicase regulates Scc2 deposition specifically during DNA-replication in Saccharomyces cerevisiae.Homologous recombination and human health: the roles of BRCA1, BRCA2, and associated proteins PALB2: the hub of a network of tumor suppressors involved in DNA damage responses Cellular and molecular consequences of defective Fanconi anemia proteins in replication-coupled DNA repair: mechanistic insights Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesis Characterization of the interactome of the human MutL homologues MLH1, PMS1, and PMS2 HES1 is a novel interactor of the Fanconi anemia core complex FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in mice Expanded roles of the Fanconi anemia pathway in preserving genomic stability The Fanconi anemia pathway and ubiquitin A never-ending story: the steadily growing family of the FA and FA-like genes ATR-dependent phosphorylation of FANCA on serine 1449 after DNA damage is important for FA pathway function FANCJ helicase uniquely senses oxidative base damage in either strand of duplex DNA and is stimulated by replication protein A to unwind the damaged DNA substrate in a strand-specific manner.FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein.The role of the Fanconi anemia network in the response to DNA replication stress.Fanconi anemia proteins stabilize replication forks.Remodeling of DNA replication structures by the branch point translocase FANCM Recruitment of fanconi anemia and breast cancer proteins to DNA damage sites is differentially governed by replication

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P2860

BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ.

http://www.wikidata.org/entity/Q34449830

description

2005 nî lūn-bûn

@nan

2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2005年の論文

@ja

2005年論文

@yue

2005年論文

@zh-hant

2005年論文

@zh-hk

2005年論文

@zh-mo

2005年論文

@zh-tw

2005年论文

@wuu

name

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@ast

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@en

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@nl

type

label

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@ast

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@en

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@nl

prefLabel

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@ast

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@en

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@nl

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P356

J.CCR.2005.08.004

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P1476

BACH1 is critical for homologo ...... oni anemia gene product FANCJ.

@en

P2093

Fan Zhang

http://www.w3.org/2001/XMLSchema#string

Junran Zhang

http://www.w3.org/2001/XMLSchema#string

Min Peng

http://www.w3.org/2001/XMLSchema#string

Paul R Andreassen

http://www.w3.org/2001/XMLSchema#string

Rachel Litman

http://www.w3.org/2001/XMLSchema#string

Sharon B Cantor

http://www.w3.org/2001/XMLSchema#string

Simon Powell

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Zhe Jin

http://www.w3.org/2001/XMLSchema#string

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255-265

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scholarly article

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10.1016/J.CCR.2005.08.004

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3

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8

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2005-09-01T00:00:00Z

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7608662

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16153896

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