Impaired action potential initiation in GABAergic interneurons causes hyperexcitable networks in an epileptic mouse model carrying a human Na(V)1.1 mutation
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The Revolution in Migraine Genetics: From Aching Channels Disorders to a Next-Generation MedicineMolecular pathophysiology and pharmacology of the voltage-sensing module of neuronal ion channelsHomeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potentialRare and common epilepsies converge on a shared gene regulatory network providing opportunities for novel antiepileptic drug discovery.The genetic relationship between epilepsy and hemiplegic migraineHuman Cerebrospinal Fluid Promotes Neuronal Viability and Activity of Hippocampal Neuronal Circuits In VitroUnaltered Network Activity and Interneuronal Firing During Spontaneous Cortical Dynamics In Vivo in a Mouse Model of Severe Myoclonic Epilepsy of Infancy.Model systems for studying cellular mechanisms of SCN1A-related epilepsy.Early-onset familial hemiplegic migraine due to a novel SCN1A mutation.Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.Precision physiology and rescue of brain ion channel disorders.Migraine genetics: current findings and future lines of research.Models for discovery of targeted therapy in genetic epileptic encephalopathies.Aberrant epilepsy-associated mutant Nav1.6 sodium channel activity can be targeted with cannabidiol.Watch Out, No Brakes! Impaired Inhibition Results in Hyperexcitable Networks.Interaction of electrically evoked activity with intrinsic dynamics of cultured cortical networks with and without functional fast GABAergic synaptic transmission.Inhibitory or excitatory? Optogenetic interrogation of the functional roles of GABAergic interneurons in epileptogenesis.Ion Channels in Genetic Epilepsy: From Genes and Mechanisms to Disease-Targeted Therapies.Neurons with Multiple Axons Have Functional Axon Initial Segments.INTERNEURONOPATHIES AND THEIR ROLE IN EARLY LIFE EPILEPSIES AND NEURODEVELOPMENTAL DISORDERS.Preclinical Animal Models for Dravet Syndrome: Seizure Phenotypes, Comorbidities and Drug Screening.The Perineuronal 'Safety' Net? Perineuronal Net Abnormalities in Neurological Disorders
P2860
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P2860
Impaired action potential initiation in GABAergic interneurons causes hyperexcitable networks in an epileptic mouse model carrying a human Na(V)1.1 mutation
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2014 nî lūn-bûn
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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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Impaired action potential init ...... ouse model carrying a human Na
@nl
Impaired action potential init ...... ying a human Na(V)1.1 mutation
@ast
Impaired action potential init ...... ying a human Na(V)1.1 mutation
@en
type
label
Impaired action potential init ...... ouse model carrying a human Na
@nl
Impaired action potential init ...... ying a human Na(V)1.1 mutation
@ast
Impaired action potential init ...... ying a human Na(V)1.1 mutation
@en
prefLabel
Impaired action potential init ...... ouse model carrying a human Na
@nl
Impaired action potential init ...... ying a human Na(V)1.1 mutation
@ast
Impaired action potential init ...... ying a human Na(V)1.1 mutation
@en
P2093
P2860
P50
P1476
Impaired action potential init ...... ying a human Na(V)1.1 mutation
@en
P2093
Andrew Escayg
Camille Liautard
Heinz Beck
Holger Lerche
Johannes Slotta
Marcel Dihné
Martin Pofahl
Stephan Theiss
Ulrike B S Hedrich
Yuanyuan Liu
P2860
P304
14874-14889
P356
10.1523/JNEUROSCI.0721-14.2014
P407
P577
2014-11-01T00:00:00Z